Cytotoxicity of advanced glycation endproducts is mediated by oxidative stress.
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Advanced glycation endproducts interfere with adhesion and neurite outgrowthThe hydrolyzable gallotannin, penta-O-galloyl-β-D-glucopyranoside, inhibits the formation of advanced glycation endproducts by protecting protein structure.Neurotoxicity of advanced glycation end-products for cultured cortical neurons.Chitinase and Fizz family members are a generalized feature of nematode infection with selective upregulation of Ym1 and Fizz1 by antigen-presenting cellsNeuroprotective effects of active ingredients isolated from Pegasus laternarius on cultured cerebral neurons.Induction of neuronal death by microglial AGE-albumin: implications for Alzheimer's disease.Abnormal thiamine-dependent processes in Alzheimer's Disease. Lessons from diabetesNeuronal activation in the central nervous system of rats in the initial stage of chronic kidney disease-modulatory effects of losartan and moxonidine.Azithromycin synergistically enhances anti-proliferative activity of vincristine in cervical and gastric cancer cells.N-acetylcysteine in the prevention of radiocontrast-induced nephropathy: clinical trials and end points.The Possible Mechanism of Advanced Glycation End Products (AGEs) for Alzheimer's DiseaseGlyceraldehyde caused Alzheimer's disease-like alterations in diagnostic marker levels in SH-SY5Y human neuroblastoma cellsGlutathione deficiency in type 2 diabetes impairs cytokine responses and control of intracellular bacteria.Oxidative-nitrosative stress and poly(ADP-ribose) polymerase (PARP) activation in experimental diabetic neuropathy: the relation is revisited.Advanced glycation and lipoxidation end products--amplifiers of inflammation: the role of food.Glibenclamide impairs responses of neutrophils against Burkholderia pseudomallei by reduction of intracellular glutathione.Advanced glycation endproducts and their pathogenic roles in neurological disorders.Protein folding, misfolding, aggregation and their implications in human diseases: discovering therapeutic ways to amyloid-associated diseases.Reducing methylglyoxal as a therapeutic target for diabetic heart disease.Advanced glycation end-product (AGE) induces apoptosis in human retinal ARPE-19 cells via promoting mitochondrial dysfunction and activating the Fas-FasL signaling.AGEs Promote Oxidative Stress and Induce Apoptosis in Retinal Pigmented Epithelium Cells RAGE-dependently.Rosiglitazone protects neuroblastoma cells against advanced glycation end products-induced injury.The antioxidant 3H-1,2-dithiole-3-thione potentiates advanced glycation end-product-induced oxidative stress in SH-SY5Y cells.Prion protein complexed to N2a cellular RNAs through its N-terminal domain forms aggregates and is toxic to murine neuroblastoma cellsAntimycobacterial and synergistic effects of 18β-glycyrrhetinic acid or glycyrrhetinic acid-30-piperazine in combination with isoniazid, rifampicin or streptomycin against Mycobacterium bovis.Identification of a flavonoid isolated from plum (Prunus domestica) as a potent inhibitor of Hepatitis C virus entry.Genotoxicity of advanced glycation end products: involvement of oxidative stress and of angiotensin II type 1 receptors.Collagen type I prevents glyoxal-induced apoptosis in osteoblastic cells cultured on titanium alloy.Advanced glycation endproducts induce changes in glucose consumption, lactate production, and ATP levels in SH-SY5Y neuroblastoma cells by a redox-sensitive mechanism.Anti-inflammatory antioxidants attenuate the expression of inducible nitric oxide synthase mediated by advanced glycation endproducts in murine microglia.Protein modification by dicarbonyl molecular species in neurodegenerative diseases.Erythropoietin attenuates advanced glycation endproducts-induced toxicity of Schwann cells in vitro.A plant-derived dehydrorotenoid: a new inhibitor of hepatitis C virus entry.Is There Inflammatory Synergy in Type II Diabetes Mellitus and Alzheimer's Disease?The ubiquitous conserved glycopeptidase Gcp prevents accumulation of toxic glycated proteins.Advanced glycation end products inhibit the expression of collagens type I and III by human gingival fibroblasts.Involvement of a gut-retina axis in protection against dietary glycemia-induced age-related macular degeneration.The RAGE receptor and its ligands are highly expressed in astrocytes in a grade-dependant manner in the striatum and subependymal layer in Huntington's disease.Receptor for advanced glycation end products (RAGE)-mediated cytotoxicity of 3-hydroxypyridinium derivatives.The Receptor for Advanced Glycation Endproducts is a mediator of toxicity by IAPP and other proteotoxic aggregates: Establishing and Exploiting Common Ground for Novel Amyloidosis Therapies.
P2860
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P2860
Cytotoxicity of advanced glycation endproducts is mediated by oxidative stress.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Cytotoxicity of advanced glycation endproducts is mediated by oxidative stress.
@en
type
label
Cytotoxicity of advanced glycation endproducts is mediated by oxidative stress.
@en
prefLabel
Cytotoxicity of advanced glycation endproducts is mediated by oxidative stress.
@en
P2093
P356
P1476
Cytotoxicity of advanced glycation endproducts is mediated by oxidative stress.
@en
P2093
A M Cunningham
P Riederer
R Schinzel
P2888
P304
P356
10.1007/S007020050108
P577
1998-01-01T00:00:00Z
P6179
1023016084