Insulin stimulation of gene expression mediated by p21ras activation.
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P1343
1-Phosphatidylinositol 3-kinase activity is required for insulin-stimulated glucose transport but not for RAS activation in CHO cellsRegulation of Sos activity by intramolecular interactions.p21ras activation via hemopoietin receptors and c-kit requires tyrosine kinase activity but not tyrosine phosphorylation of p21ras GTPase-activating proteinA protein factor for ras p21-dependent activation of mitogen-activated protein (MAP) kinase through MAP kinase kinaseAssociation and colocalization of Eps15 with adaptor protein-2 and clathrinInvolvement of distinct G-proteins, Gpa2 and Ras, in glucose- and intracellular acidification-induced cAMP signalling in the yeast Saccharomyces cerevisiae.Platelet-derived growth factor receptor mediates activation of ras through different signaling pathways in different cell typesStimulation of gene induction and cell growth by the Ras effector RlfcAMP antagonizes p21ras-directed activation of extracellular signal-regulated kinase 2 and phosphorylation of mSos nucleotide exchange factorInhibition of rab5 GTPase activity stimulates membrane fusion in endocytosisGTPase-activating protein SH2-SH3 domains induce gene expression in a Ras-dependent fashion.Particle image correlation spectroscopy (PICS): retrieving nanometer-scale correlations from high-density single-molecule position dataData of rational process optimization for the production of a full IgG and its Fab fragment from hybridoma cellsDetermination of absolute amounts of GDP and GTP bound to Ras in mammalian cells: comparison of parental and Ras-overproducing NIH 3T3 fibroblastsSingle cell Ras-GTP analysis reveals altered Ras activity in a subpopulation of neurofibroma Schwann cells but not fibroblasts.Effect of bombesin receptor subtype-3 and its synthetic agonist on signaling, glucose transport and metabolism in myocytes from patients with obesity and type 2 diabetes.Ras signaling in the activation of glucose transport by insulin.Pertussis toxin-sensitive activation of p21ras by G protein-coupled receptor agonists in fibroblasts.The ras signaling pathway mimics insulin action on glucose transporter translocationThymic T cell anergy in autoimmune nonobese diabetic mice is mediated by deficient T cell receptor regulation of the pathway of p21ras activation.Association between GRB2/Sos and insulin receptor substrate 1 is not sufficient for activation of extracellular signal-regulated kinases by interleukin-4: implications for Ras activation by insulin.rac p21 is involved in insulin-induced membrane ruffling and rho p21 is involved in hepatocyte growth factor- and 12-O-tetradecanoylphorbol-13-acetate (TPA)-induced membrane ruffling in KB cells.The Saccharomyces cerevisiae SDC25 C-domain gene product overcomes the dominant inhibitory activity of Ha-Ras Asn-17.Microinjection of the SH2 domain of the 85-kilodalton subunit of phosphatidylinositol 3-kinase inhibits insulin-induced DNA synthesis and c-fos expressionInhibition of thyrotropin-stimulated DNA synthesis by microinjection of inhibitors of cellular Ras and cyclic AMP-dependent protein kinase.The posttranslational processing of ras p21 is critical for its stimulation of yeast adenylate cyclase.Reovirus oncolysis: the Ras/RalGEF/p38 pathway dictates host cell permissiveness to reovirus infectionCoagulation factor Xa stimulates platelet-derived growth factor release and mitogenesis in cultured vascular smooth muscle cells of rat.Circulating concentrations of C-Peptide and colorectal adenoma.Ras proteins are essential and selective for the action of insulin-like growth factor 1 late in the G1 phase of the cell cycle in BALB/c murine fibroblastsInterferon-alpha2b reduces phosphorylation and activity of MEK and ERK through a Ras/Raf-independent mechanism.Direct control of caveolin-1 expression by FOXO transcription factors.Rab5 induces Rac-independent lamellipodia formation and cell migration.FOXOs support the metabolic requirements of normal and tumor cells by promoting IDH1 expression.Ras-dependent regulation of c-Jun phosphorylation is mediated by the Ral guanine nucleotide exchange factor-Ral pathway.Growth factors can activate ATF2 via a two-step mechanism: phosphorylation of Thr71 through the Ras-MEK-ERK pathway and of Thr69 through RalGDS-Src-p38A dominant-negative mutant of mSOS1 inhibits insulin-induced Ras activation and reveals Ras-dependent and -independent insulin signaling pathways.Two dominant inhibitory mutants of p21ras interfere with insulin-induced gene expression.Role of protein kinase C in T-cell antigen receptor regulation of p21ras: evidence that two p21ras regulatory pathways coexist in T cellsEpidermal growth factor induces phosphorylation of extracellular signal-regulated kinase 2 via multiple pathways.
P2860
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P2860
Insulin stimulation of gene expression mediated by p21ras activation.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Insulin stimulation of gene expression mediated by p21ras activation.
@en
type
label
Insulin stimulation of gene expression mediated by p21ras activation.
@en
prefLabel
Insulin stimulation of gene expression mediated by p21ras activation.
@en
P2093
P2860
P1433
P1476
Insulin stimulation of gene expression mediated by p21ras activation
@en
P2093
Burgering BM
Maassen JA
McCormick F
van de Wetering ML
van der Eb AJ
P2860
P304
P356
10.1002/J.1460-2075.1991.TB08050.X
P407
P4510
P577
1991-05-01T00:00:00Z