Tyrosine residues in the granulocyte colony-stimulating factor (G-CSF) receptor mediate G-CSF-induced differentiation of murine myeloid leukemic (M1) cells.
about
Granulocyte colony-stimulating factor: molecular mechanisms of action during steady state and 'emergency' hematopoiesis.Control of myeloid differentiation and survival by Stats.Jak2-Stat5 interactions analyzed in yeast.Control of hematopoietic differentiation: lack of specificity in signaling by cytokine receptors.Novel point mutation in the extracellular domain of the granulocyte colony-stimulating factor (G-CSF) receptor in a case of severe congenital neutropenia hyporesponsive to G-CSF treatment.Absence of cytokine receptor-dependent specificity in red blood cell differentiation in vivoControl of myeloid-specific integrin alpha Mbeta 2 (CD11b/CD18) expression by cytokines is regulated by Stat3-dependent activation of PU.1.Sustained receptor activation and hyperproliferation in response to granulocyte colony-stimulating factor (G-CSF) in mice with a severe congenital neutropenia/acute myeloid leukemia-derived mutation in the G-CSF receptor gene.Multiple pathways contribute to the hyperproliferative responses from truncated granulocyte colony-stimulating factor receptors.Contribution of the membrane-distal tyrosine in intracellular signaling by the granulocyte colony-stimulating factor receptor.Epidermal growth factor receptor: association of extracellular domain negatively regulates intracellular kinase activation in the absence of ligand.Acceleration of granulocyte colony-stimulating factor-induced neutrophilic nuclear lobulation by overexpression of Lyn tyrosine kinase.Cooperation of p38 and extracellular signal-regulated kinase mitogen-activated protein kinase pathways during granulocyte colony-stimulating factor-induced hemopoietic cell proliferation.The proximal tyrosines of the cytoplasmic domain of the beta chain of the type I interferon receptor are essential for signal transducer and activator of transcription (Stat) 2 activation. Evidence that two Stat2 sites are required to reach a threshDifferential ability of SOCS proteins to regulate IL-6 and CSF-1 induced macrophage differentiation.Multiple Signals Mediate Proliferation, Differentiation, and Survival from the Granulocyte Colony-stimulating Factor Receptor in Myeloid 32D Cells
P2860
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P2860
Tyrosine residues in the granulocyte colony-stimulating factor (G-CSF) receptor mediate G-CSF-induced differentiation of murine myeloid leukemic (M1) cells.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Tyrosine residues in the granu ...... e myeloid leukemic (M1) cells.
@en
type
label
Tyrosine residues in the granu ...... e myeloid leukemic (M1) cells.
@en
prefLabel
Tyrosine residues in the granu ...... e myeloid leukemic (M1) cells.
@en
P2093
P2860
P356
P1476
Tyrosine residues in the granu ...... ne myeloid leukemic (M1) cells
@en
P2093
P2860
P304
26947-26953
P356
10.1074/JBC.271.43.26947
P407
P577
1996-10-01T00:00:00Z