Synaptic plasticity defect following visual deprivation in Alzheimer's disease model transgenic mice.
about
At the interface of sensory and motor dysfunctions and Alzheimer's disease.Inconsistencies and controversies surrounding the amyloid hypothesis of Alzheimer's diseaseReducing hippocampal extracellular matrix reverses early memory deficits in a mouse model of Alzheimer's disease.BACE1 is necessary for experience-dependent homeostatic synaptic plasticity in visual cortexRemovable cranial windows for long-term imaging in awake miceBlocking PirB up-regulates spines and functional synapses to unlock visual cortical plasticity and facilitate recovery from amblyopia.Presynaptic Aβ40 prevents synapse addition in the adult Drosophila neuromuscular junction.Aβ alters the connectivity of olfactory neurons in the absence of amyloid plaques in vivoTau-amyloid interactions in the rTgTauEC model of early Alzheimer's disease suggest amyloid-induced disruption of axonal projections and exacerbated axonal pathologyADAMTS expression and function in central nervous system injury and disorders.Human LilrB2 is a β-amyloid receptor and its murine homolog PirB regulates synaptic plasticity in an Alzheimer's model.PirB regulates a structural substrate for cortical plasticity.The Binding Receptors of Aβ: an Alternative Therapeutic Target for Alzheimer's Disease.Mechanisms of homeostatic plasticity in the excitatory synapse.Microglia-Synapse Pathways: Promising Therapeutic Strategy for Alzheimer's Disease.Amyloid-β Oligomers May Impair SNARE-Mediated Exocytosis by Direct Binding to Syntaxin 1a.Neuroscience. Promiscuous Alzheimer's amyloid: yet another partner.Activity-Dependent Dysfunction in Visual and Olfactory Sensory Systems in Mouse Models of Down Syndrome.Early Activation of Experience-Independent Dendritic Spine Turnover in a Mouse Model of Alzheimer's Disease.Reconsideration of Amyloid Hypothesis and Tau Hypothesis in Alzheimer's Disease.Evidence of demyelination in mild cognitive impairment and dementia using a direct and specific magnetic resonance imaging measure of myelin content.Cellular Receptors of Amyloid β Oligomers (AβOs) in Alzheimer's Disease
P2860
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P2860
Synaptic plasticity defect following visual deprivation in Alzheimer's disease model transgenic mice.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Synaptic plasticity defect fol ...... disease model transgenic mice.
@en
type
label
Synaptic plasticity defect fol ...... disease model transgenic mice.
@en
prefLabel
Synaptic plasticity defect fol ...... disease model transgenic mice.
@en
P2093
P2860
P1476
Synaptic plasticity defect fol ...... disease model transgenic mice.
@en
P2093
Carla J Shatz
Demetris K Roumis
Glenn J Goldey
Mark L Andermann
Mark W Albers
Matthew P Frosch
R Clay Reid
P2860
P304
P356
10.1523/JNEUROSCI.5369-11.2012
P407
P577
2012-06-01T00:00:00Z