HIV-1 Tat and morphine have interactive effects on oligodendrocyte survival and morphology. - Wikidata - wikimedia - TriplyDB

HIV-1 Tat and morphine have interactive effects on oligodendrocyte survival and morphology.

HIV-1 Tat and morphine have interactive effects on oligodendrocyte survival and morphology.

http://www.wikidata.org/entity/Q41763649

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Oligodendrocyte Injury and Pathogenesis of HIV-1-Associated Neurocognitive Disorders Interaction between Tat and Drugs of Abuse during HIV-1 Infection and Central Nervous System Disease Roles and functions of HIV-1 Tat protein in the CNS: an overview A growth factor attenuates HIV-1 Tat and morphine induced damage to human neurons: implication in HIV/AIDS-drug abuse cases Morphine enhances HIV-1SF162-mediated neuron death and delays recovery of injured neurites The retrovirus/superantigen hypothesis of multiple sclerosis.HIV-1 alters neural and glial progenitor cell dynamics in the central nervous system: coordinated response to opiates during maturation.Quantitation of parvalbumin+ neurons and human immunodeficiency virus type 1 (HIV-1) regulatory gene expression in the HIV-1 transgenic rat: effects of vitamin A deficiency and morphine The pathogenesis of murine coronavirus infection of the central nervous system.Interactive comorbidity between opioid drug abuse and HIV-1 Tat: chronic exposure augments spine loss and sublethal dendritic pathology in striatal neurons beta-Chemokine production by neural and glial progenitor cells is enhanced by HIV-1 Tat: effects on microglial migration.Interactions of HIV and drugs of abuse: the importance of glia, neural progenitors, and host genetic factors.Effects of chronic HIV-1 Tat exposure in the CNS: heightened vulnerability of males versus females to changes in cell numbers, synaptic integrity, and behavior Oligodendrocyte responses to buprenorphine uncover novel and opposing roles of μ-opioid- and nociceptin/orphanin FQ receptors in cell development: implications for drug addiction treatment during pregnancy Morphine potentiates neurodegenerative effects of HIV-1 Tat through actions at μ-opioid receptor-expressing glia.Chronic SIV and morphine treatment increases heat shock protein 5 expression at the synapse.Rodent models for HIV-associated neurocognitive disorders.Morphine and gp120 toxic interactions in striatal neurons are dependent on HIV-1 strain.Oligodendrocytes Are Targets of HIV-1 Tat: NMDA and AMPA Receptor-Mediated Effects on Survival and Development.Interactive Effects of Morphine on HIV Infection: Role in HIV-Associated Neurocognitive Disorder.Opiate drug use and the pathophysiology of neuroAIDS.Chronic HIV-1 Tat and HIV reduce Rbfox3/NeuN: evidence for sex-related effects The chemokine receptor CXCR2 and coronavirus-induced neurologic disease.Synaptic dysfunction in the hippocampus accompanies learning and memory deficits in human immunodeficiency virus type-1 Tat transgenic mice.Effects of Morphine on Behavioral Task Performance in SIV-Infected Rhesus Macaques.Genetic knockouts suggest a critical role for HIV co-receptors in models of HIV gp120-induced brain injury 5α-reduced progestogens ameliorate mood-related behavioral pathology, neurotoxicity, and microgliosis associated with exposure to HIV-1 Tat.HIV-1 Tat Inhibits Autotaxin Lysophospholipase D Activity and Modulates Oligodendrocyte Differentiation HIV-1 Tat disrupts blood-brain barrier integrity and increases phagocytic perivascular macrophages and microglia in the dorsal striatum of transgenic mice.Human immunodeficiency virus protein Tat induces oligodendrocyte injury by enhancing outward K+ current conducted by KV1.3.A failure to normalize biochemical and metabolic insults during morphine withdrawal disrupts synaptic repair in mice transgenic for HIV-gp120.Central HIV-1 Tat exposure elevates anxiety and fear conditioned responses of male mice concurrent with altered mu-opioid receptor-mediated G-protein activation and β-arrestin 2 activity in the forebrain.CCR5 mediates HIV-1 Tat-induced neuroinflammation and influences morphine tolerance, dependence, and reward.[Retrovirus superantigen hypothesis of multiple sclerosis].Transgenic mice expressing HIV-1 envelope protein gp120 in the brain as an animal model in neuroAIDS research.Doxycycline-inducible and astrocyte-specific HIV-1 Tat transgenic mice (iTat) as an HIV/neuroAIDS model.

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P2860

HIV-1 Tat and morphine have interactive effects on oligodendrocyte survival and morphology.

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Annadora J Bruce-Keller

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Avindra Nath

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Kurt F Hauser

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Pamela E Knapp

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Shiping Zou

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Shreya K Buch

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Valeriya V Adjan

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Yun Kyung Hahn

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P2860

Human immunodeficiency virus type 1 gp120 induces apoptosis in human primary neurons through redox-regulated activation of neutral sphingomyelinase

HIV-1 associated dementia: symptoms and causes

Caspase-3 activity is reduced after spinal cord injury in mice lacking dynorphin: differential effects on glia and neurons.

Apoptotic death of striatal neurons induced by human immunodeficiency virus-1 Tat and gp120: Differential involvement of caspase-3 and endonuclease G

The neuropathogenesis of AIDS

Morphine stimulates HIV replication in primary cultures of human Kupffer cells

Synergistic neurotoxicity of opioids and human immunodeficiency virus-1 Tat protein in striatal neurons in vitro

Opioid system diversity in developing neurons, astroglia, and oligodendroglia in the subventricular zone and striatum: impact on gliogenesis in vivo

Opioid addiction and pregnancy: perinatal exposure to buprenorphine affects myelination in the developing brain

Active caspase-3 expression during postnatal development of rat cerebellum is not systematically or consistently associated with apoptosis

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10.1002/GLIA.20746

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2

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57

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