Rapid and spontaneous loss of phthiocerol dimycocerosate (PDIM) from Mycobacterium tuberculosis grown in vitro: implications for virulence studies.
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Phosphorylation of the Peptidoglycan Synthase PonA1 Governs the Rate of Polar Elongation in MycobacteriaCrystal Structure of Mycobacterium tuberculosis Polyketide Synthase 11 (PKS11) Reveals Intermediates in the Synthesis of Methyl-branched AlkylpyronesMycobacterium tuberculosis complex lipid virulence factors preserved in the 17,000-year-old skeleton of an extinct bison, Bison antiquusSurface hydrolysis of sphingomyelin by the outer membrane protein Rv0888 supports replication of Mycobacterium tuberculosis in macrophagesDrug resistant clinical isolates of Mycobacterium tuberculosis from different genotypes exhibit differential host responses in THP-1 cellsDiversity of Mycobacterium tuberculosis across Evolutionary ScalesHigh Persister Mutants in Mycobacterium tuberculosisA Hidden Markov Model for identifying essential and growth-defect regions in bacterial genomes from transposon insertion sequencing data.The impact of mouse passaging of Mycobacterium tuberculosis strains prior to virulence testing in the mouse and guinea pig aerosol models.Strains of the East Asian (W/Beijing) lineage of Mycobacterium tuberculosis are DosS/DosT-DosR two-component regulatory system natural mutants.Origins of a 350-kilobase genomic duplication in Mycobacterium tuberculosis and its impact on virulenceRelevant assay to study the adhesion of Plasmodium falciparum-infected erythrocytes to the placental epithelium.Variation among genome sequences of H37Rv strains of Mycobacterium tuberculosis from multiple laboratories.Gluconeogenic carbon flow of tricarboxylic acid cycle intermediates is critical for Mycobacterium tuberculosis to establish and maintain infectionMycobacterium tuberculosis lineage influences innate immune response and virulence and is associated with distinct cell envelope lipid profilesMassive gene duplication event among clinical isolates of the Mycobacterium tuberculosis W/Beijing family.Does M. tuberculosis genomic diversity explain disease diversity?SigG does not control gene expression in response to DNA damage in Mycobacterium tuberculosis H37Rv.Mycobacterium tuberculosis strains lacking surface lipid phthiocerol dimycocerosate are susceptible to killing by an early innate host response.Inactivation of tesA reduces cell wall lipid production and increases drug susceptibility in mycobacteria.Spontaneous phthiocerol dimycocerosate-deficient variants of Mycobacterium tuberculosis are susceptible to gamma interferon-mediated immunity.Essential Role of the ESX-5 Secretion System in Outer Membrane Permeability of Pathogenic MycobacteriaChlamydia trachomatis In Vivo to In Vitro Transition Reveals Mechanisms of Phase Variation and Down-Regulation of Virulence FactorsComparative genomics between human and animal associated subspecies of the Mycobacterium avium complex: a basis for pathogenicity.Both phthiocerol dimycocerosates and phenolic glycolipids are required for virulence of Mycobacterium marinum.Sulfolipid-1 biosynthesis restricts Mycobacterium tuberculosis growth in human macrophagesMycobacterium tuberculosis lacking all mycolic acid cyclopropanation is viable but highly attenuated and hyperinflammatory in miceUpregulation of the phthiocerol dimycocerosate biosynthetic pathway by rifampin-resistant, rpoB mutant Mycobacterium tuberculosisDisruption of the serine/threonine protein kinase H affects phthiocerol dimycocerosates synthesis in Mycobacterium tuberculosis.Antituberculosis thiophenes define a requirement for Pks13 in mycolic acid biosynthesisLoss of Lipid Virulence Factors Reduces the Efficacy of the BCG Vaccine.A Nonsense Mutation in Mycobacterium marinum That Is Suppressible by a Novel MechanismAssociation between spoligotype-VNTR types and virulence of Mycobacterium bovis in cattle.A hydrolase of trehalose dimycolate induces nutrient influx and stress sensitivity to balance intracellular growth of Mycobacterium tuberculosis.The Mycobacterium tuberculosis cytochromes P450: physiology, biochemistry & molecular intervention.Molecular insights into bacteroid development during Rhizobium-legume symbiosis.Virulence factors of the Mycobacterium tuberculosis complexBiosynthesis of mycobacterial lipids by polyketide synthases and beyond.Purification of Helicobacter suis Strains From Biphasic Cultures by Single Colony Isolation: Influence on Strain Characteristics.MTBVAC from discovery to clinical trials in tuberculosis-endemic countries.
P2860
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P2860
Rapid and spontaneous loss of phthiocerol dimycocerosate (PDIM) from Mycobacterium tuberculosis grown in vitro: implications for virulence studies.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Rapid and spontaneous loss of ...... cations for virulence studies.
@en
type
label
Rapid and spontaneous loss of ...... cations for virulence studies.
@en
prefLabel
Rapid and spontaneous loss of ...... cations for virulence studies.
@en
P2860
P356
P1433
P1476
Rapid and spontaneous loss of ...... cations for virulence studies.
@en
P2093
Michael B Reed
Pilar Domenech
P2860
P304
P356
10.1099/MIC.0.029199-0
P577
2009-08-06T00:00:00Z