Herpes simplex virus 1 DNA is in unstable nucleosomes throughout the lytic infection cycle, and the instability of the nucleosomes is independent of DNA replication.
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The differential mobilization of histones H3.1 and H3.3 by herpes simplex virus 1 relates histone dynamics to the assembly of viral chromatinInhibition of LSD1 reduces herpesvirus infection, shedding, and recurrence by promoting epigenetic suppression of viral genomesGenome wide nucleosome mapping for HSV-1 shows nucleosomes are deposited at preferred positions during lytic infection.Dynamic modulation of HSV chromatin drives initiation of infection and provides targets for epigenetic therapies.Herpesviral ICP0 Protein Promotes Two Waves of Heterochromatin Removal on an Early Viral Promoter during Lytic Infection.Chromatin Modulation of Herpesvirus Lytic Gene Expression: Managing Nucleosome Density and Heterochromatic Histone Modifications.A novel selective LSD1/KDM1A inhibitor epigenetically blocks herpes simplex virus lytic replication and reactivation from latency.Visualizing the replicating HSV-1 virus using STED super-resolution microscopy.The dynamics of HCF-1 modulation of herpes simplex virus chromatin during initiation of infection.Role of polycomb proteins in regulating HSV-1 latency.Canonical and variant forms of histone H3 are deposited onto the human cytomegalovirus genome during lytic and latent infectionsEpigenetic repression of herpes simplex virus infection by the nucleosome remodeler CHD3Brain transcriptomes of harbor seals demonstrate gene expression patterns of animals undergoing a metabolic disease and a viral infection.HSV-1 ICP22: hijacking host nuclear functions to enhance viral infection.Chromatin dynamics during lytic infection with herpes simplex virus 1.Selective recruitment of host factors by HSV-1 replication centers.Combinatorial effects of the glucocorticoid receptor and Krüppel-like transcription factor 15 on bovine herpesvirus 1 transcription and productive infection.Promoting Simultaneous Onset of Viral Gene Expression Among Cells Infected with Herpes Simplex Virus-1.Spatiotemporal dynamics of HSV genome nuclear entry and compaction state transitions using bioorthogonal chemistry and super-resolution microscopy.Timing Is Everything.
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P2860
Herpes simplex virus 1 DNA is in unstable nucleosomes throughout the lytic infection cycle, and the instability of the nucleosomes is independent of DNA replication.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Herpes simplex virus 1 DNA is ...... ndependent of DNA replication.
@en
Herpes simplex virus 1 DNA is ...... ndependent of DNA replication.
@nl
type
label
Herpes simplex virus 1 DNA is ...... ndependent of DNA replication.
@en
Herpes simplex virus 1 DNA is ...... ndependent of DNA replication.
@nl
prefLabel
Herpes simplex virus 1 DNA is ...... ndependent of DNA replication.
@en
Herpes simplex virus 1 DNA is ...... ndependent of DNA replication.
@nl
P2860
P356
P1433
P1476
Herpes simplex virus 1 DNA is ...... ndependent of DNA replication.
@en
P2093
Jonathan J Lacasse
Luis M Schang
P2860
P304
11287-11300
P356
10.1128/JVI.01468-12
P407
P577
2012-08-08T00:00:00Z