Human immunodeficiency virus type 1 Nef incorporation into virions does not increase infectivity
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HIV Genome-Wide Protein Associations: a Review of 30 Years of ResearchThe identification of a small molecule compound that reduces HIV-1 Nef-mediated viral infectivity enhancementHIV-1 infection of T cells and macrophages are differentially modulated by virion-associated Hck: a Nef-dependent phenomenonSingle-domain antibody-SH3 fusions for efficient neutralization of HIV-1 Nef functionsMLV glycosylated-Gag is an infectivity factor that rescues Nef-deficient HIV-1.Recruitment of the nuclear form of uracil DNA glycosylase into virus particles participates in the full infectivity of HIV-1.Nef decreases HIV-1 sensitivity to neutralizing antibodies that target the membrane-proximal external region of TMgp41.Dendritic cell-mediated HIV-1 infection of T cells demonstrates a direct relationship to plasma viral RNA levelsCellular phenotype impacts human immunodeficiency virus type 1 viral protein R subcellular localizationStraightforward selection of broadly neutralizing single-domain antibodies targeting the conserved CD4 and coreceptor binding sites of HIV-1 gp120Comparative proteomic analysis of HIV-1 particles reveals a role for Ezrin and EHD4 in the Nef-dependent increase of virus infectivity.Mechanisms of HIV-1 Nef function and intracellular signaling.HIV Nef protein causes endothelial dysfunction in porcine pulmonary arteries and human pulmonary artery endothelial cells.HIV-1 Nef responsiveness is determined by Env variable regions involved in trimer association and correlates with neutralization sensitivityThe Antagonism of HIV-1 Nef to SERINC5 Particle Infectivity Restriction Involves the Counteraction of Virion-Associated Pools of the Restriction Factor.The Nef-like effect of murine leukemia virus glycosylated gag on HIV-1 infectivity is mediated by its cytoplasmic domain and depends on the AP-2 adaptor complex.The Nef-infectivity enigma: mechanisms of enhanced lentiviral infection.The activity of Nef on HIV-1 infectivitySuboptimal provirus expression explains apparent nonrandom cell coinfection with HIV-1.Spotlight on HIV-1 Nef: SERINC3 and SERINC5 Identified as Restriction Factors Antagonized by the Pathogenesis Factor.Induction of extremely low protein expression level by fusion of C-terminal region of Nef.
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P2860
Human immunodeficiency virus type 1 Nef incorporation into virions does not increase infectivity
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
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2008年论文
@zh-cn
name
Human immunodeficiency virus t ...... does not increase infectivity
@en
Human immunodeficiency virus t ...... does not increase infectivity
@nl
type
label
Human immunodeficiency virus t ...... does not increase infectivity
@en
Human immunodeficiency virus t ...... does not increase infectivity
@nl
prefLabel
Human immunodeficiency virus t ...... does not increase infectivity
@en
Human immunodeficiency virus t ...... does not increase infectivity
@nl
P2860
P356
P1433
P1476
Human immunodeficiency virus t ...... does not increase infectivity
@en
P2093
Nadine Laguette
Stéphane Basmaciogullari
P2860
P304
P356
10.1128/JVI.01633-08
P407
P577
2008-11-05T00:00:00Z