Mice deficient in the CXCR2 ligand, CXCL1 (KC/GRO-alpha), exhibit increased susceptibility to dextran sodium sulfate (DSS)-induced colitis.
about
The Dual Role of Neutrophils in Inflammatory Bowel DiseasesLoss of TMF/ARA160 protein renders colonic mucus refractory to bacterial colonization and diminishes intestinal susceptibility to acute colitisCXCR2-positive neutrophils are essential for cuprizone-induced demyelination: relevance to multiple sclerosiscis-Urocanic acid attenuates acute dextran sodium sulphate-induced intestinal inflammation.Microbial ecology of the murine gut associated with the development of dextran sodium sulfate-induced colitis.The Myc 3' Wnt responsive element regulates neutrophil recruitment after acute colonic injury in mice.Dextran sodium sulfate (DSS) induces colitis in mice by forming nano-lipocomplexes with medium-chain-length fatty acids in the colonCXCL1 regulates pulmonary host defense to Klebsiella Infection via CXCL2, CXCL5, NF-kappaB, and MAPKs.Intestinal inflammation and mucosal barrier function.The complement anaphylatoxin C3a receptor (C3aR) contributes to the inflammatory response in dextran sulfate sodium (DSS)-induced colitis in mice.The pathogenicity of an enteric Citrobacter rodentium Infection is enhanced by deficiencies in the antioxidants selenium and vitamin E.Nondigestible oligosaccharides exert nonprebiotic effects on intestinal epithelial cells enhancing the immune response via activation of TLR4-NFκB.Tissue factor-dependent chemokine production aggravates experimental colitis.Matriptase protects against experimental colitis and promotes intestinal barrier recoveryIntrapulmonary administration of leukotriene B(4) augments neutrophil accumulation and responses in the lung to Klebsiella infection in CXCL1 knockout mice.The ion channel transient receptor potential melastatin-2 does not play a role in inflammatory mouse models of chronic obstructive pulmonary diseasesProgesterone protects endothelial cells after cerebrovascular occlusion by decreasing MCP-1- and CXCL1-mediated macrophage infiltration.Short- or long-term high-fat diet feeding plus acute ethanol binge synergistically induce acute liver injury in mice: an important role for CXCL1.The Nlrp3 inflammasome, IL-1β, and neutrophil recruitment are required for susceptibility to a nonhealing strain of Leishmania major in C57BL/6 mice.Patient-derived xenograft mouse models of pseudomyxoma peritonei recapitulate the human inflammatory tumor microenvironment.Granzyme M has a critical role in providing innate immune protection in ulcerative colitisp40phox expression regulates neutrophil recruitment and function during the resolution phase of intestinal inflammation.Chemokine CXCL1 mediated neutrophil recruitment: Role of glycosaminoglycan interactions.The angiogenic effect of probiotic Bacillus polyfermenticus on human intestinal microvascular endothelial cells is mediated by IL-8.International Union of Basic and Clinical Pharmacology. [corrected]. LXXXIX. Update on the extended family of chemokine receptors and introducing a new nomenclature for atypical chemokine receptors.The biology of chemokines and their receptors.Transcriptome profiling of mouse colonic eosinophils reveals a key role for eosinophils in the induction of s100a8 and s100a9 in mucosal healing.Interleukin-8, CXCL1, and MicroRNA miR-146a Responses to Probiotic Escherichia coli Nissle 1917 and Enteropathogenic E. coli in Human Intestinal Epithelial T84 and Monocytic THP-1 Cells after Apical or Basolateral Infection.Postbiotic Modulation of Retinoic Acid Imprinted Mucosal-like Dendritic Cells by Probiotic Lactobacillus reuteri 17938 In Vitro.Regulation of intestinal permeability: The role of proteasesBlood flow patterns spatially associated with platelet aggregates in murine colitisCXCL1, but not IL-6, significantly impacts intraocular inflammation during infection.Vascular Stem/Progenitor Cell Migration Induced by Smooth Muscle Cell-Derived Chemokine (C-C Motif) Ligand 2 and Chemokine (C-X-C motif) Ligand 1 Contributes to Neointima Formation.Differential In Vitro and In Vivo Toxicities of Antimicrobial Peptide Prodrugs for Potential Use in Cystic Fibrosis.Murine guanylate cyclase C regulates colonic injury and inflammation.CXCL1 contributes to host defense in polymicrobial sepsis via modulating T cell and neutrophil functions.Constitutive expression of MMP9 in intestinal epithelium worsens murine acute colitis and is associated with increased levels of proinflammatory cytokine Kc.Prebiotic oligosaccharides directly modulate proinflammatory cytokine production in monocytes via activation of TLR4.Inactivation of I kappaB-kinase-beta dependent genes in airway epithelium reduces tobacco smoke induced acute airway inflammation.Neuraminidase reprograms lung tissue and potentiates lipopolysaccharide-induced acute lung injury in mice.
P2860
Q28080144-7D112D60-9B35-4885-B4CB-A714616F5ED4Q28586189-C4E1C260-D1B8-431D-866A-33EADA9E2FBEQ30493331-A70F7C72-0987-4127-82F0-AECE1A54E798Q33742124-6A52C326-C931-4947-A92A-53E2EA3F5BAAQ33842489-5F9550D3-EA0B-4267-9F1F-00A3FB8DE94DQ33923735-E473A4C7-7A80-4663-ABB0-5304F269F553Q34199709-6474EAA2-D19D-4BA7-8B40-77CEC2F8421FQ34280062-679A1E5B-D664-408E-B2C4-2F11B0F8DC83Q34438743-BF2B9B3A-1497-4161-BD8D-87B53825E1DBQ34701117-FDA81056-EB4C-4666-943B-3FEC6B4E5D35Q34740127-2FA83058-EA56-4AFA-81A4-D4C9AEAEF20EQ34987852-B1C02BE6-37D4-4B6D-93F8-DA2FE0D28D84Q35287011-8E180077-0984-4D25-8705-6FF2942A0959Q35785774-B0E02282-3CEC-4C96-BC54-B837DDD94203Q35851033-A101E968-5C3A-40A3-BC98-EB2894B99118Q35951096-D3620390-AAEA-4246-8C89-7E0C63C74DFAQ36101721-96BC604F-B470-450E-9B57-E1C3CA8795EBQ36108884-89156035-204B-40AC-81A0-B51EBFD5FF51Q36788745-77B9F81A-6344-48EF-80B1-A8C52A2E18F9Q36797297-570F83E2-C3A6-4F11-AB73-F81E97AB6CC3Q37151517-29F9DE78-1245-42DA-9F8B-CBF409EA8CF2Q37192211-A7542183-C7D0-4419-BB0D-ACDC8B88FABDQ37254227-63BB8936-9F30-439A-9337-20706C2956FCQ37421164-F77DDD72-64AC-47E6-A92F-A06132F9E6CDQ37432097-4F2220E0-510E-402A-B637-9F0A291FA6AAQ37998913-B4861F2C-6884-41AE-AD05-9207CD401CFDQ38645859-8999686B-A28B-40BE-8BA0-9311D0F2F0B3Q38764707-C9FD25B6-0326-404D-9A97-C3BE8B770678Q39055343-858492E1-F329-4FBE-B060-BB00626239BBQ39239228-8591023D-6E24-420E-B37A-EF236C865A14Q40462136-D3ABA38F-EB65-46B9-87C2-C485EAC0DFDEQ40473896-D04E1666-615B-4A96-A519-81A052991C09Q41109128-A88332D6-13C8-43B3-B8C3-6E29664656B9Q41289926-D4C9D80A-A27B-408B-BCD6-AC297C8FB634Q41982611-47C91595-5AE5-4754-8ED2-105F48BE3837Q42199779-94562956-8F8C-43F1-B0AE-0D0FCA6C34D0Q42435344-74EE1FF5-1149-4E52-9243-BB398FF93DA2Q42454418-2F76FA8D-8F26-47D9-B3BE-8DD834E0BF68Q42713759-6F74426C-1A9C-4382-8C7A-78DF9BAC36BCQ42869378-EA2DB766-20B9-46D8-AE17-978F803F6E73
P2860
Mice deficient in the CXCR2 ligand, CXCL1 (KC/GRO-alpha), exhibit increased susceptibility to dextran sodium sulfate (DSS)-induced colitis.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Mice deficient in the CXCR2 li ...... sulfate (DSS)-induced colitis.
@en
Mice deficient in the CXCR2 li ...... sulfate (DSS)-induced colitis.
@nl
type
label
Mice deficient in the CXCR2 li ...... sulfate (DSS)-induced colitis.
@en
Mice deficient in the CXCR2 li ...... sulfate (DSS)-induced colitis.
@nl
prefLabel
Mice deficient in the CXCR2 li ...... sulfate (DSS)-induced colitis.
@en
Mice deficient in the CXCR2 li ...... sulfate (DSS)-induced colitis.
@nl
P2093
P2860
P356
P1433
P1476
Mice deficient in the CXCR2 li ...... sulfate (DSS)-induced colitis.
@en
P2093
Aiping Zhao
Karen M Kopydlowski
Karen Thomas
Louis J Detolla
M Joshua Cody
Masayuki Fukata
Sergio A Lira
Stefanie N Vogel
Terez Shea-Donohue
P2860
P304
P356
10.1177/1753425908088724
P577
2008-04-01T00:00:00Z