Bmi-1 is a crucial regulator of prostate stem cell self-renewal and malignant transformation. - Wikidata - wikimedia - TriplyDB

Bmi-1 is a crucial regulator of prostate stem cell self-renewal and malignant transformation.

Bmi-1 is a crucial regulator of prostate stem cell self-renewal and malignant transformation.

http://www.wikidata.org/entity/Q42164562

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Role of Bmi-1 in regulation of ionizing irradiation-induced epithelial-mesenchymal transition and migration of breast cancer cells Regulation of DU145 prostate cancer cell growth by Scm-like with four mbt domains 2 Yin Yang 1 is associated with cancer stem cell transcription factors (SOX2, OCT4, BMI1) and clinical implication A Novel Aspect of Tumorigenesis-BMI1 Functions in Regulating DNA Damage Response Common stemness regulators of embryonic and cancer stem cells Androgen receptor and prostate cancer stem cells: biological mechanisms and clinical implications Emergence of zebrafish models in oncology for validating novel anticancer drug targets and nanomaterials Biomaterial strategies for stem cell maintenance during in vitro expansion Enhancing integrin function by VEGF/neuropilin signaling: implications for tumor biology A microRNA code for prostate cancer metastasis.MicroRNAs targeting prostate cancer stem cells Functional screen analysis reveals miR-26b and miR-128 as central regulators of pituitary somatomammotrophic tumor growth through activation of the PTEN-AKT pathway Wnt/β-catenin signalling in prostate cancer Phosphatase and tensin homolog deleted on chromosome 10 (PTEN) and PH domain and leucine-rich repeat phosphatase cross-talk (PHLPP) in cancer cells and in transforming growth factor β-activated stem cells Altered Expression of Genes in Signaling Pathways Regulating Proliferation of Hematopoietic Stem and Progenitor Cells in Mice with Subchronic Benzene Exposure Bmi1 regulates auditory hair cell survival by maintaining redox balance.VEGF/neuropilin-2 regulation of Bmi-1 and consequent repression of IGF-IR define a novel mechanism of aggressive prostate cancer Bmi-1 overexpression as an efficient prognostic marker in patients with nonsmall cell lung cancer Phosphorylation of Nanog is essential to regulate Bmi1 and promote tumorigenesis.Epithelial-to-mesenchymal transition in prostate cancer: paradigm or puzzle?VEGF targets the tumour cell.Bmi-1 regulates epithelial-to-mesenchymal transition to promote migration and invasion of breast cancer cells.Bmi1 expression in long-term germ stem cells.Epigenetic modifications by dietary phytochemicals: implications for personalized nutrition Analysis of marker-defined HNSCC subpopulations reveals a dynamic regulation of tumor initiating properties.Nonadhesive culture system as a model of rapid sphere formation with cancer stem cell properties.Inhibition of sphingosine kinase 2 downregulates the expression of c-Myc and Mcl-1 and induces apoptosis in multiple myeloma.Cancer genetics and epigenetics: two sides of the same coin?Estrogen receptor β activation impairs prostatic regeneration by inducing apoptosis in murine and human stem/progenitor enriched cell populations.Molecular pathways and targets in prostate cancer Bmi1 regulates self-renewal and epithelial to mesenchymal transition in breast cancer cells through Nanog.The role of epithelial plasticity in prostate cancer dissemination and treatment resistance Nanoparticle labeling identifies slow cycling human endometrial stromal cells β-catenin is required for prostate development and cooperates with Pten loss to drive invasive carcinoma.A Tale of Two Signals: AR and WNT in Development and Tumorigenesis of Prostate and Mammary Gland.Therapy-induced developmental reprogramming of prostate cancer cells and acquired therapy resistance.Bmi1 enhances tumorigenicity and cancer stem cell function in pancreatic adenocarcinoma.Surgical treatment for patients with Krukenberg tumor of stomach origin: clinical outcome and prognostic factors analysis.PLK1 inhibition down-regulates polycomb group protein BMI1 via modulation of the miR-200c/141 cluster.Coordinated regulation of polycomb group complexes through microRNAs in cancer.

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Bmi-1 is a crucial regulator of prostate stem cell self-renewal and malignant transformation.

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Bmi-1 is a crucial regulator o ...... and malignant transformation.

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Bmi-1 is a crucial regulator o ...... and malignant transformation.

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J.STEM.2010.11.013

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Hong Wu

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Owen N Witte

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Rita U Lukacs

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Sanaz Memarzadeh

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P2860

Role of Notch signaling in cell-fate determination of human mammary stem/progenitor cells

p63 is a prostate basal cell marker and is required for prostate development

Wnt signaling inhibitors regulate the transcriptional response to morphogenetic Shh-Gli signaling in the neural tube

Activation of beta-catenin-Tcf signaling in colon cancer by mutations in beta-catenin or APC

Microarray analysis identifies a death-from-cancer signature predicting therapy failure in patients with multiple types of cancer

Bmi-1 promotes neural stem cell self-renewal and neural development but not mouse growth and survival by repressing the p16Ink4a and p19Arf senescence pathways.

Intestinal polyposis in mice with a dominant stable mutation of the beta-catenin gene

Stem cells, cancer, and cancer stem cells

Bmi-1 is required for maintenance of adult self-renewing haematopoietic stem cells

Hedgehog signaling and Bmi-1 regulate self-renewal of normal and malignant human mammary stem cells

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