Endothelial nitric oxide synthase is a site of superoxide synthesis in endothelial cells treated with glyceryl trinitrate. - Wikidata - wikimedia - TriplyDB

Endothelial nitric oxide synthase is a site of superoxide synthesis in endothelial cells treated with glyceryl trinitrate.

Endothelial nitric oxide synthase is a site of superoxide synthesis in endothelial cells treated with glyceryl trinitrate.

http://www.wikidata.org/entity/Q42209398

about

Vascular dysfunction in retinopathy-an emerging role for arginase Endothelium-derived free radicals: for worse and for better.Endothelial progenitor dysfunction in the pathogenesis of diabetic retinopathy: treatment concept to correct diabetes-associated deficits.A mitochondrial superoxide theory for oxidative stress diseases and aging Melatonin inhibits nitrate tolerance in isolated coronary arteries.Long-term oral nitrate therapy is associated with adverse outcome in diabetic patients following elective percutaneous coronary intervention.Experimental diabetes causes breakdown of the blood-retina barrier by a mechanism involving tyrosine nitration and increases in expression of vascular endothelial growth factor and urokinase plasminogen activator receptor.Endothelial dysfunction and diabetes: effects on angiogenesis, vascular remodeling, and wound healing.Arginase II inhibition prevents nitrate tolerance.Therapeutic use of citrulline in cardiovascular disease.Deregulation of arginase induces bone complications in high-fat/high-sucrose diet diabetic mouse model The enigma of nitroglycerin bioactivation and nitrate tolerance: news, views and troubles The promise of cell-based therapies for diabetic complications: challenges and solutions Nitroglycerine-induced nitrate tolerance compromises propofol protection of the endothelial cells against TNF-α: the role of PKC-β2 and NADPH oxidase.The role of RhoA/Rho kinase pathway in endothelial dysfunction Diabetes-induced coronary vascular dysfunction involves increased arginase activity Peroxynitrite mediates diabetes-induced endothelial dysfunction: possible role of Rho kinase activation.Role of the general base Glu-268 in nitroglycerin bioactivation and superoxide formation by aldehyde dehydrogenase-2.Effect of gender on endothelium-dependent dilation to bradykinin in human adipose microvessels.Reply to Daiber et al.: “Doubt about an essential role for constitutive nitric oxide synthase in nitroglycerin-mediated vasodilation”.Fasudil ameliorates endothelial dysfunction in streptozotocin-induced diabetic rats: a possible role of Rho kinase.

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P2860

Endothelial nitric oxide synthase is a site of superoxide synthesis in endothelial cells treated with glyceryl trinitrate.

http://www.wikidata.org/entity/Q42209398

description

2000 nî lūn-bûn

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2000年の論文

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2000年論文

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2000年論文

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Endothelial nitric oxide synth ...... ated with glyceryl trinitrate.

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Endothelial nitric oxide synth ...... ated with glyceryl trinitrate.

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Endothelial nitric oxide synth ...... ated with glyceryl trinitrate.

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British Journal of Pharmacology

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Endothelial nitric oxide synth ...... ated with glyceryl trinitrate.

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P2093

A A Ogonowski

http://www.w3.org/2001/XMLSchema#string

L Jin

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R B Caldwell

http://www.w3.org/2001/XMLSchema#string

R W Caldwell

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W H Kaesemeyer

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P2860

Effect of supplemental oral L-arginine on exercise capacity in patients with stable angina pectoris

Effects of long-term nitroglycerin treatment on endothelial nitric oxide synthase (NOS III) gene expression, NOS III-mediated superoxide production, and vascular NO bioavailability

The reaction of no with superoxide

Evidence for enhanced vascular superoxide anion production in nitrate tolerance. A novel mechanism underlying tolerance and cross-tolerance.

Cell and tissue responses to oxidative damage.

Substrate binding and calmodulin binding to endothelial nitric oxide synthase coregulate its enzymatic activity.

The effect of various nitric oxide-donor agents on hydrogen peroxide-mediated toxicity: a direct correlation between nitric oxide formation and protection.

Superoxide anion is an endothelium-derived contracting factor.

Nitrate therapy for stable angina pectoris.

Nitric oxide (NO) donor molecules: effect of NO release rate on vascular smooth muscle cell proliferation in vitro.

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1019-1023

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10.1038/SJ.BJP.0703665

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5

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131

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11053225

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