Inhibition of NF-kappaB sensitizes human pancreatic carcinoma cells to apoptosis induced by etoposide (VP16) or doxorubicin. - Wikidata - wikimedia - TriplyDB

Inhibition of NF-kappaB sensitizes human pancreatic carcinoma cells to apoptosis induced by etoposide (VP16) or doxorubicin.

Inhibition of NF-kappaB sensitizes human pancreatic carcinoma cells to apoptosis induced by etoposide (VP16) or doxorubicin.

http://www.wikidata.org/entity/Q42503199

about

Preadministration of High-Dose Salicylates, Suppressors of NF- B Activation, May Increase the Chemosensitivity of Many Cancers: An Example of Proapoptotic Signal Modulation Therapy ZM336372 induces apoptosis associated with phosphorylation of GSK-3beta in pancreatic adenocarcinoma cell lines Gemcitabine-based chemogene therapy for pancreatic cancer using Ad-dCK::UMK GDEPT and TS/RR siRNA strategies Apoptosis: targets in pancreatic cancer New insights into the role of nuclear factor-kB in cell growth regulation Leucine-zipper protein, LDOC1, inhibits NF-kappaB activation and sensitizes pancreatic cancer cells to apoptosis Augmentation of therapeutic responses in melanoma by inhibition of IRAK-1,-4 Signals from within: the DNA-damage-induced NF-kappaB response Nitrosylcobalamin potentiates the anti-neoplastic effects of chemotherapeutic agents via suppression of survival signaling Late activation of stress kinases (SAPK/JNK) by genotoxins requires the DNA repair proteins DNA-PKcs and CSB Primers on molecular pathways. The glycogen synthase kinase-3beta Glycogen synthase kinase-3 inhibition disrupts nuclear factor-kappaB activity in pancreatic cancer, but fails to sensitize to gemcitabine chemotherapy The role of FADD in pancreatic cancer cell proliferation and drug resistance S100P: a novel therapeutic target for cancer.Real-time monitoring of nuclear factor kappaB activity in cultured cells and in animal models.α-Mangostin: a dietary antioxidant derived from the pericarp of Garcinia mangostana L. inhibits pancreatic tumor growth in xenograft mouse model.Fluid Shear Stress Sensitizes Cancer Cells to Receptor-Mediated Apoptosis via Trimeric Death Receptors.Irreversible EGFR inhibitor EKB-569 targets low-LET γ-radiation-triggered rel orchestration and potentiates cell death in squamous cell carcinoma.Liguzinediol improved the heart function and inhibited myocardial cell apoptosis in rats with heart failure Nuclear factor-kappaB p65/relA silencing induces apoptosis and increases gemcitabine effectiveness in a subset of pancreatic cancer cells Induction of cancer cell death by isoflavone: the role of multiple signaling pathways.Effect of cromolyn on S100P interactions with RAGE and pancreatic cancer growth and invasion in mouse models.APE1/Ref-1 role in redox signaling: translational applications of targeting the redox function of the DNA repair/redox protein APE1/Ref-1.Clusterin silencing sensitizes pancreatic cancer MIA-PaCa-2 cells to gmcitabine via regulation of NF-kB/Bcl-2 signaling.Sensitization of tumor cells to cancer therapy by molecularly targeted inhibition of the inhibitor of nuclear factor κB kinase.Plumbagin, a plant derived natural agent inhibits the growth of pancreatic cancer cells in in vitro and in vivo via targeting EGFR, Stat3 and NF-κB signaling pathways.AKT inhibition is associated with chemosensitisation in the pancreatic cancer cell line MIA-PaCa-2 Targeting signal transduction in pancreatic cancer treatment.Apoptotic pathways in pancreatic ductal adenocarcinoma Cancer Stem Cells, EMT, and Developmental Pathway Activation in Pancreatic Tumors Epigenetic regulation of SOX9 by the NF-κB signaling pathway in pancreatic cancer stem cells.MyD88 expression by CNS-resident cells is pivotal for eliciting protective immunity in brain abscesses.Recent prognostic factors in diffuse large B-cell lymphoma indicate NF-kappaB pathway as a target for new therapeutic strategies.Interaction of stellate cells with pancreatic carcinoma cells S100P-derived RAGE antagonistic peptide reduces tumor growth and metastasis.Lysyl oxidase propeptide sensitizes pancreatic and breast cancer cells to doxorubicin-induced apoptosis.Inhibition of NUCKS Facilitates Corneal Recovery Following Alkali Burn Natural and synthetic α,β-unsaturated carbonyls for NF-κB inhibition.Mechanisms of resistance to chemotherapeutic and anti-angiogenic drugs as novel targets for pancreatic cancer therapy.Potentiation of tumour apoptosis by human growth hormone via glutathione production and decreased NF-kappaB activity.

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Inhibition of NF-kappaB sensitizes human pancreatic carcinoma cells to apoptosis induced by etoposide (VP16) or doxorubicin.

http://www.wikidata.org/entity/Q42503199

description

2001 nî lūn-bûn

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2001年の論文

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年学术文章

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2001年學術文章

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2001年學術文章

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2001年學術文章

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name

Inhibition of NF-kappaB sensit ...... oposide (VP16) or doxorubicin.

@en

Inhibition of NF-kappaB sensit ...... apoptosis induced by etoposide

@nl

type

label

Inhibition of NF-kappaB sensit ...... oposide (VP16) or doxorubicin.

@en

Inhibition of NF-kappaB sensit ...... apoptosis induced by etoposide

@nl

prefLabel

Inhibition of NF-kappaB sensit ...... oposide (VP16) or doxorubicin.

@en

Inhibition of NF-kappaB sensit ...... apoptosis induced by etoposide

@nl

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Inhibition of NF-kappaB sensit ...... oposide (VP16) or doxorubicin.

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H Kalthoff

http://www.w3.org/2001/XMLSchema#string

H Schäfer

http://www.w3.org/2001/XMLSchema#string

J Vorndamm

http://www.w3.org/2001/XMLSchema#string

M Breitenbroich

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U R Fölsch

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W E Schmidt

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P2860

The Bcl-3 oncoprotein acts as a bridging factor between NF-kappaB/Rel and nuclear co-regulators

NF-kappaB induces expression of the Bcl-2 homologue A1/Bfl-1 to preferentially suppress chemotherapy-induced apoptosis

Sulfasalazine: a potent and specific inhibitor of nuclear factor kappa B

TNF- and cancer therapy-induced apoptosis: potentiation by inhibition of NF-kappaB

Dissection of TNF receptor 1 effector functions: JNK activation is not linked to apoptosis while NF-kappaB activation prevents cell death

Control of apoptosis by Rel/NF-kappaB transcription factors.

Transcriptional activation by NF-kappaB requires multiple coactivators

The promoter of human p22/PACAP response gene 1 (PRG1) contains functional binding sites for the p53 tumor suppressor and for NFkappaB.

Transcriptional repression by wild-type p53 utilizes histone deacetylases, mediated by interaction with mSin3a.

The immunosuppressive fungal metabolite gliotoxin specifically inhibits transcription factor NF-kappaB.

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10.1038/SJ.ONC.1204168

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