The basal-like breast carcinoma phenotype is regulated by SLUG gene expression.
about
NEDD9 is a positive regulator of epithelial-mesenchymal transition and promotes invasion in aggressive breast cancerSLUG: a new target of lymphoid enhancer factor-1 in human osteoblastsThe epithelial-mesenchymal transition (EMT) phenomenonEpigenetic control of the basal-like gene expression profile via Interleukin-6 in breast cancer cellsYin Yang 1 is associated with cancer stem cell transcription factors (SOX2, OCT4, BMI1) and clinical implicationSLUG: Critical regulator of epithelial cell identity in breast development and cancerActivation of the canonical Wnt/β-catenin pathway in ATF3-induced mammary tumorsCirculating tumor cell clusters-associated gene plakoglobin and breast cancer survival.Slug controls stem/progenitor cell growth dynamics during mammary gland morphogenesisEpithelial to mesenchymal transition and breast cancer.SLUG/SNAI2 and tumor necrosis factor generate breast cells with CD44+/CD24- phenotype.Epithelial-stromal interaction 1 (EPSTI1) substitutes for peritumoral fibroblasts in the tumor microenvironmentPrognostic value of different amounts of cancer stem cells in different molecular subtypes of breast cancerDifferences in the transcriptional response to fulvestrant and estrogen deprivation in ER-positive breast cancerNew promising drug targets in cancer- and metastasis-initiating cells.Key signalling nodes in mammary gland development and cancer. The Snail1-Twist1 conspiracy in malignant breast cancer progression.Snail family regulation and epithelial mesenchymal transitions in breast cancer progressionMusashi1 regulates breast tumor cell proliferation and is a prognostic indicator of poor survival.TNFalpha up-regulates SLUG via the NF-kappaB/HIF1alpha axis, which imparts breast cancer cells with a stem cell-like phenotype.Novel therapies against aggressive and recurrent epithelial cancers by molecular targeting tumor- and metastasis-initiating cells and their progenies.New advances on critical implications of tumor- and metastasis-initiating cells in cancer progression, treatment resistance and disease recurrenceCD146 expression in human breast cancer cell lines induces phenotypic and functional changes observed in Epithelial to Mesenchymal TransitionCD133 is a useful surrogate marker for predicting chemosensitivity to neoadjuvant chemotherapy in breast cancer.MicroRNA-200 family modulation in distinct breast cancer phenotypes.Phenotypic heterogeneity of breast cancer stem cells.Minireview: Basal-like breast cancer: from molecular profiles to targeted therapiesGenetic predisposition directs breast cancer phenotype by dictating progenitor cell fate.In vitro analysis of breast cancer cell line tumourspheres and primary human breast epithelia mammospheres demonstrates inter- and intrasphere heterogeneity.Epithelial-mesenchymal transition and cancer stem cells: a dangerously dynamic duo in breast cancer progression.Prostate-Derived ETS Factor Regulates Epithelial-to-Mesenchymal Transition through Both SLUG-Dependent and Independent MechanismsActivation of KRAS promotes the mesenchymal features of basal-type breast cancer.Beta-catenin/HuR post-transcriptional machinery governs cancer stem cell features in response to hypoxiaPhloroglucinol suppresses metastatic ability of breast cancer cells by inhibition of epithelial-mesenchymal cell transition.FOXF2 deficiency promotes epithelial-mesenchymal transition and metastasis of basal-like breast cancerCell fate takes a slug in BRCA1-associated breast cancerG9a interacts with Snail and is critical for Snail-mediated E-cadherin repression in human breast cancerHigh motility of triple-negative breast cancer cells is due to repression of plakoglobin gene by metastasis modulator protein SLUG.Suppression of invasion and metastasis of triple-negative breast cancer lines by pharmacological or genetic inhibition of slug activity.Hypoxia-induced carbonic anhydrase IX facilitates lactate flux in human breast cancer cells by non-catalytic function.A new role of SNAI2 in postlactational involution of the mammary gland links it to luminal breast cancer development.
P2860
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P2860
The basal-like breast carcinoma phenotype is regulated by SLUG gene expression.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
The basal-like breast carcinoma phenotype is regulated by SLUG gene expression.
@en
The basal-like breast carcinoma phenotype is regulated by SLUG gene expression.
@nl
type
label
The basal-like breast carcinoma phenotype is regulated by SLUG gene expression.
@en
The basal-like breast carcinoma phenotype is regulated by SLUG gene expression.
@nl
prefLabel
The basal-like breast carcinoma phenotype is regulated by SLUG gene expression.
@en
The basal-like breast carcinoma phenotype is regulated by SLUG gene expression.
@nl
P2093
P50
P356
P1476
The basal-like breast carcinoma phenotype is regulated by SLUG gene expression
@en
P2093
M Taffurelli
P Paterini
S Tavolari
T Guarnieri
P356
10.1002/PATH.2254
P577
2008-01-01T00:00:00Z