Mutations in the pore region modify epithelial sodium channel gating by shear stress.
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Epithelial Na(+) channel regulation by cytoplasmic and extracellular factorsENaC regulation by proteases and shear stressHow does the urothelium affect bladder function in health and disease? ICI-RS 2011Urinary bladder, cystitis and nerve/urothelial interactions.Urothelial signaling.Cysteine palmitoylation of the γ subunit has a dominant role in modulating activity of the epithelial sodium channelThe actin cytoskeleton and small G protein RhoA are not involved in flow-dependent activation of ENaC.Expression and Analysis of Flow-regulated Ion Channels in Xenopus Oocytes.Neural control of the lower urinary tract: peripheral and spinal mechanismsExtracellular allosteric regulatory subdomain within the gamma subunit of the epithelial Na+ channel.Role of epithelial sodium channels and their regulators in hypertension.Cys palmitoylation of the beta subunit modulates gating of the epithelial sodium channelSecond transmembrane domain modulates epithelial sodium channel gating in response to shear stressPoint mutations in the transmembrane region of the clic1 ion channel selectively modify its biophysical propertiesMechanisms of disease: involvement of the urothelium in bladder dysfunction.Extracellular finger domain modulates the response of the epithelial sodium channel to shear stressGain-of-function variant of the human epithelial sodium channel.Regulation of cation transport in the distal nephron by mechanical forces.The epithelial sodium channel mediates the directionality of galvanotaxis in human keratinocytes.Activation of the Caenorhabditis elegans Degenerin Channel by Shear Stress Requires the MEC-10 Subunit.A new trick for an old dogma: ENaC proteins as mechanotransducers in vascular smooth muscle.Gamma subunit second transmembrane domain contributes to epithelial sodium channel gating and amiloride block.Regulation of Potassium HomeostasisThe epithelial Na+ channel is inhibited by a peptide derived from proteolytic processing of its alpha subunit.Novel determinants of epithelial sodium channel gating within extracellular thumb domains.βENaC acts as a mechanosensor in renal vascular smooth muscle cells that contributes to renal myogenic blood flow regulation, protection from renal injury and hypertension.Lack of a role of membrane-protein interactions in flow-dependent activation of ENaC.Revealing a subclinical salt-losing phenotype in heterozygous carriers of the novel S562P mutation in the alpha subunit of the epithelial sodium channel.Sensor Mechanism and Afferent Signal Transduction of the Urinary Bladder: Special Focus on transient receptor potential Ion ChannelsENaC in Cholinergic Brush Cells
P2860
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P2860
Mutations in the pore region modify epithelial sodium channel gating by shear stress.
description
2004 nî lūn-bûn
@nan
2004年の論文
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2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
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name
Mutations in the pore region modify epithelial sodium channel gating by shear stress.
@en
Mutations in the pore region modify epithelial sodium channel gating by shear stress.
@nl
type
label
Mutations in the pore region modify epithelial sodium channel gating by shear stress.
@en
Mutations in the pore region modify epithelial sodium channel gating by shear stress.
@nl
prefLabel
Mutations in the pore region modify epithelial sodium channel gating by shear stress.
@en
Mutations in the pore region modify epithelial sodium channel gating by shear stress.
@nl
P2860
P356
P1476
Mutations in the pore region modify epithelial sodium channel gating by shear stress.
@en
P2093
Marcelo D Carattino
Thomas R Kleyman
P2860
P304
P356
10.1074/JBC.M413123200
P407
P50
P577
2004-11-29T00:00:00Z