The stress-activated protein kinase Hog1 mediates S phase delay in response to osmostress.
about
Control of Ubp3 ubiquitin protease activity by the Hog1 SAPK modulates transcription upon osmostress.Design, synthesis, and characterization of a highly effective Hog1 inhibitor: a powerful tool for analyzing MAP kinase signaling in yeastFrom START to FINISH: the influence of osmotic stress on the cell cycleCompaction properties of an intrinsically disordered protein: Sic1 and its kinase-inhibitor domain.The Neurospora crassa OS MAPK pathway-activated transcription factor ASL-1 contributes to circadian rhythms in pathway responsive clock-controlled genesImpaired manganese metabolism causes mitotic misregulation.The p57 CDKi integrates stress signals into cell-cycle progression to promote cell survival upon stress.Control of high osmolarity signalling in the yeast Saccharomyces cerevisiae.Multilayered control of gene expression by stress-activated protein kinases.How Saccharomyces cerevisiae copes with toxic metals and metalloids.The p38 and Hog1 SAPKs control cell cycle progression in response to environmental stresses.Response to hyperosmotic stressCurrent understanding of HOG-MAPK pathway in Aspergillus fumigatus.Osmostress-induced gene expression--a model to understand how stress-activated protein kinases (SAPKs) regulate transcriptionExploiting the yeast stress-activated signaling network to inform on stress biology and disease signaling.Recovery from stress - a cell cycle perspective.Microbial response to environmental stresses: from fundamental mechanisms to practical applications.Hog1 targets Whi5 and Msa1 transcription factors to downregulate cyclin expression upon stress.The Hog1 MAP Kinase Promotes the Recovery from Cell Cycle Arrest Induced by Hydrogen Peroxide in Candida albicans.Intramolecular interactions stabilizing compact conformations of the intrinsically disordered kinase-inhibitor domain of Sic1: a molecular dynamics investigation.Stress Adaptation.Multiple signaling kinases target Mrc1 to prevent genomic instability triggered by transcription-replication conflicts.ChIP-seq reveals a role for CrzA in the Aspergillus fumigatus high-osmolarity glycerol response (HOG) signalling pathway.The defects in cell wall integrity and G2-M transition of the ∆htl1 mutant are interconnected.Transcriptional timing and noise of yeast cell cycle regulators-a single cell and single molecule approach.
P2860
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P2860
The stress-activated protein kinase Hog1 mediates S phase delay in response to osmostress.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
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2009年论文
@zh-cn
name
The stress-activated protein k ...... lay in response to osmostress.
@en
The stress-activated protein k ...... lay in response to osmostress.
@nl
type
label
The stress-activated protein k ...... lay in response to osmostress.
@en
The stress-activated protein k ...... lay in response to osmostress.
@nl
prefLabel
The stress-activated protein k ...... lay in response to osmostress.
@en
The stress-activated protein k ...... lay in response to osmostress.
@nl
P2860
P50
P356
P1476
The stress-activated protein k ...... lay in response to osmostress.
@en
P2093
Gilad Yaakov
Josep Clotet
P2860
P304
P356
10.1091/MBC.E09-02-0129
P577
2009-05-28T00:00:00Z