The role of the E1 and E2 proteins in the replication of human papillomavirus type 31b.
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Mechanisms of virus immune evasion lead to development from chronic inflammation to cancer formation associated with human papillomavirus infectionInteraction of NCOR/SMRT Repressor Complexes with Papillomavirus E8^E2C Proteins Inhibits Viral ReplicationNuclear accumulation of the papillomavirus E1 helicase blocks S-phase progression and triggers an ATM-dependent DNA damage responseDNA replication of human papillomavirus type 31 is modulated by elements of the upstream regulatory region that lie 5' of the minimal originInduction of the human papillomavirus type 31 late promoter requires differentiation but not DNA amplificationGenetic analysis of the human papillomavirus type 31 differentiation-dependent late promoter.Variant upstream regulatory region sequences differentially regulate human papillomavirus type 16 DNA replication throughout the viral life cycle.A C-terminal helicase domain of the human papillomavirus E1 protein binds E2 and the DNA polymerase alpha-primase p68 subunit.Transactivation by the E2 protein of oncogenic human papillomavirus type 31 is not essential for early and late viral functions.Role of the ATP-binding domain of the human papillomavirus type 11 E1 helicase in E2-dependent binding to the originDifferentiation-dependent chromatin rearrangement coincides with activation of human papillomavirus type 31 late gene expression.Early polyadenylation signals of human papillomavirus type 31 negatively regulate capsid gene expression.E1-mediated recruitment of a UAF1-USP deubiquitinase complex facilitates human papillomavirus DNA replicationBiology of human papillomaviruses.Nuclear export of human papillomavirus type 31 E1 is regulated by Cdk2 phosphorylation and required for viral genome maintenance.Pathogenesis of human papillomaviruses in differentiating epithelia.Long-term effect of interferon on keratinocytes that maintain human papillomavirus type 31.Human papillomavirus type 31 replication modes during the early phases of the viral life cycle depend on transcriptional and posttranscriptional regulation of E1 and E2 expression.Development of quantitative and high-throughput assays of polyomavirus and papillomavirus DNA replicationRecent Insights into the Control of Human Papillomavirus (HPV) Genome Stability, Loss, and Degradation.Human papillomavirus type 31 oncoproteins E6 and E7 are required for the maintenance of episomes during the viral life cycle in normal human keratinocytes.Inhibition of human papillomavirus DNA replication by an E1-derived p80/UAF1-binding peptide.Differential effects of the splice acceptor at nucleotide 3295 of human papillomavirus type 31 on stable and transient viral replication.Binding of the human papillomavirus E1 origin-recognition protein is regulated through complex formation with the E2 enhancer-binding protein.Recent advances in the search for antiviral agents against human papillomaviruses.Virology and molecular pathogenesis of HPV (human papillomavirus)-associated oropharyngeal squamous cell carcinomaHuman papillomavirus type 31b E1 and E2 transcript expression correlates with vegetative viral genome amplificationThe EVER proteins as a natural barrier against papillomaviruses: a new insight into the pathogenesis of human papillomavirus infections.In vitro synthesis of oncogenic human papillomaviruses requires episomal genomes for differentiation-dependent late expression.Human Beta-papillomavirus infection and keratinocyte carcinomas.Association of Human Papillomavirus 16 E2 with Rad50-Interacting Protein 1 Enhances Viral DNA Replication.Mapping of betapapillomavirus human papillomavirus 5 transcription and characterization of viral-genome replication function.DNA Oncogenic Virus-Induced Oxidative Stress, Genomic Damage, and Aberrant Epigenetic AlterationsThe carboxyl-terminal region of the human papillomavirus type 16 E1 protein determines E2 protein specificity during DNA replication.Yin yang 1 negatively regulates the differentiation-specific E1 promoter of human papillomavirus type 6The E8E2C protein, a negative regulator of viral transcription and replication, is required for extrachromosomal maintenance of human papillomavirus type 31 in keratinocytes.Differential requirements for conserved E2 binding sites in the life cycle of oncogenic human papillomavirus type 31The human papillomavirus type 16 E5 protein impairs TRAIL- and FasL-mediated apoptosis in HaCaT cells by different mechanismsHuman papillomavirus type 31b late gene expression is regulated through protein kinase C-mediated changes in RNA processing.Targeted mutagenesis of the human papillomavirus type 16 E2 transactivation domain reveals separable transcriptional activation and DNA replication functions.
P2860
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P2860
The role of the E1 and E2 proteins in the replication of human papillomavirus type 31b.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
The role of the E1 and E2 proteins in the replication of human papillomavirus type 31b.
@en
The role of the E1 and E2 proteins in the replication of human papillomavirus type 31b.
@nl
type
label
The role of the E1 and E2 proteins in the replication of human papillomavirus type 31b.
@en
The role of the E1 and E2 proteins in the replication of human papillomavirus type 31b.
@nl
prefLabel
The role of the E1 and E2 proteins in the replication of human papillomavirus type 31b.
@en
The role of the E1 and E2 proteins in the replication of human papillomavirus type 31b.
@nl
P356
P1433
P1476
The role of the E1 and E2 proteins in the replication of human papillomavirus type 31b.
@en
P2093
Frattini MG
Laimins LA
P304
P356
10.1006/VIRO.1994.1596
P407
P577
1994-11-01T00:00:00Z