In vitro synthesis of oncogenic human papillomaviruses requires episomal genomes for differentiation-dependent late expression.
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Role of the PDZ domain-binding motif of the oncoprotein E6 in the pathogenesis of human papillomavirus type 31Human papillomaviruses modulate expression of microRNA 203 upon epithelial differentiation to control levels of p63 proteinsThe cigarette smoke carcinogen benzo[a]pyrene enhances human papillomavirus synthesisGeneralized substitution of isoencoding codons shortens the duration of papillomavirus L1 protein expression in transiently gene-transfected keratinocytes due to cell differentiationNuclear accumulation of the papillomavirus E1 helicase blocks S-phase progression and triggers an ATM-dependent DNA damage responseHPV E6, E6AP and cervical cancer.Mechanism of genomic instability in cells infected with the high-risk human papillomaviruses.DNA replication of human papillomavirus type 31 is modulated by elements of the upstream regulatory region that lie 5' of the minimal originInduction of the human papillomavirus type 31 late promoter requires differentiation but not DNA amplificationViral DNA Replication Orientation and hnRNPs Regulate Transcription of the Human Papillomavirus 18 Late Promoter.Variant upstream regulatory region sequences differentially regulate human papillomavirus type 16 DNA replication throughout the viral life cycle.Viral E6-E7 transcription in the basal layer of organotypic cultures without apparent p21cip1 protein precedes immortalization of human papillomavirus type 16- and 18-transfected human keratinocytesTemporal usage of multiple promoters during the life cycle of human papillomavirus type 31b.Transactivation by the E2 protein of oncogenic human papillomavirus type 31 is not essential for early and late viral functions.Role of the E1--E4 protein in the differentiation-dependent life cycle of human papillomavirus type 31.Microarray analysis identifies interferon-inducible genes and Stat-1 as major transcriptional targets of human papillomavirus type 31.Differentiation-dependent chromatin rearrangement coincides with activation of human papillomavirus type 31 late gene expression.Cellular changes induced by low-risk human papillomavirus type 11 in keratinocytes that stably maintain viral episomesEarly polyadenylation signals of human papillomavirus type 31 negatively regulate capsid gene expression.In vitro progression of human papillomavirus 16 episome-associated cervical neoplasia displays fundamental similarities to integrant-associated carcinogenesis.HPV episome levels are potently decreased by pyrrole-imidazole polyamides.Human papillomavirus oncoproteins E6 and E7 independently abrogate the mitotic spindle checkpoint.Study of infectious virus production from HPV18/16 capsid chimeras.Recent advances in diagnosis and therapy of human papillomaviruses.Gene codon composition determines differentiation-dependent expression of a viral capsid gene in keratinocytes in vitro and in vivo.Genetic analysis of cis regulatory elements within the 5' region of the human papillomavirus type 31 upstream regulatory region during different stages of the viral life cyclePathogenesis of human papillomaviruses in differentiating epithelia.Genetic analysis of high-risk e6 in episomal maintenance of human papillomavirus genomes in primary human keratinocytes.Human papillomavirus type 16 E7 maintains elevated levels of the cdc25A tyrosine phosphatase during deregulation of cell cycle arrest.Saccharomyces cerevisiae is permissive for replication of bovine papillomavirus type 1.Long-term effect of interferon on keratinocytes that maintain human papillomavirus type 31.Food webs in the human body: linking ecological theory to viral dynamics.Persistence of human papillomavirus infection: keys to malignant progressionHuman papillomavirus type 31 E5 protein supports cell cycle progression and activates late viral functions upon epithelial differentiation.Human papillomavirus type 31 replication modes during the early phases of the viral life cycle depend on transcriptional and posttranscriptional regulation of E1 and E2 expression.Treatment of a human papillomavirus type 31b-positive cell line with benzo[a]pyrene increases viral titer through activation of the Erk1/2 signaling pathwayHuman papillomavirus genotype 31 does not express detectable microRNA levels during latent or productive virus replication.Replication and partitioning of papillomavirus genomes.The E7 open reading frame acts in cis and in trans to mediate differentiation-dependent activities in the human papillomavirus type 16 life cycle.Recent Insights into the Control of Human Papillomavirus (HPV) Genome Stability, Loss, and Degradation.
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In vitro synthesis of oncogenic human papillomaviruses requires episomal genomes for differentiation-dependent late expression.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on April 1996
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
In vitro synthesis of oncogeni ...... ion-dependent late expression.
@en
In vitro synthesis of oncogeni ...... ion-dependent late expression.
@nl
type
label
In vitro synthesis of oncogeni ...... ion-dependent late expression.
@en
In vitro synthesis of oncogeni ...... ion-dependent late expression.
@nl
prefLabel
In vitro synthesis of oncogeni ...... ion-dependent late expression.
@en
In vitro synthesis of oncogeni ...... ion-dependent late expression.
@nl
P2093
P2860
P356
P1476
In vitro synthesis of oncogeni ...... ion-dependent late expression.
@en
P2093
L A Laimins
M G Frattini
P2860
P304
P356
10.1073/PNAS.93.7.3062
P407
P577
1996-04-01T00:00:00Z