T cell self-reactivity forms a cytokine milieu for spontaneous development of IL-17+ Th cells that cause autoimmune arthritis. - Wikidata - wikimedia - TriplyDB

T cell self-reactivity forms a cytokine milieu for spontaneous development of IL-17+ Th cells that cause autoimmune arthritis.

T cell self-reactivity forms a cytokine milieu for spontaneous development of IL-17+ Th cells that cause autoimmune arthritis.

http://www.wikidata.org/entity/Q42767257

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Central role of the gut epithelial barrier in the pathogenesis of chronic intestinal inflammation: lessons learned from animal models and human genetics IL-17RC: a partner in IL-17 signaling and beyond The T-cell pool is anergized in patients with multiple sclerosis in remission APC-derived cytokines and T cell polarization in autoimmune inflammation Enigma of IL-17 and Th17 Cells in Rheumatoid Arthritis and in Autoimmune Animal Models of Arthritis The Role of the Transcriptional Regulation of Stromal Cells in Chronic Inflammation Review: is there a window of opportunity for treatment of systemic juvenile idiopathic arthritis?Thymus, innate immunity and autoimmune arthritis: interplay of gene and environment The thymus and rheumatology: should we care?Sex bias in experimental immune-mediated, drug-induced liver injury in BALB/c mice: suggested roles for Tregs, estrogen, and IL-6 Evidence for PI-3K-dependent migration of Th17-polarized cells in response to CCR2 and CCR6 agonists Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility.Disrupted TH17/Treg balance in patients with chronic low back pain.Local BAFF gene silencing suppresses Th17-cell generation and ameliorates autoimmune arthritis Investigation of the relationship between the onset of arthritis and uveitis in genetically predisposed SKG mice Modulation of IL-17 and Foxp3 expression in the prevention of autoimmune arthritis in mice Imatinib mesylate, a new kid on the block for the treatment of anti-neutrophil cytoplasmic autoantibodies-associated vasculitis?Erythropoietin: a potent inducer of peripheral immuno/inflammatory modulation in autoimmune EAE.Tracing functional antigen-specific CCR6 Th17 cells after vaccination.Chronic arthritis leads to disturbances in the bone collagen network.IL-17-producing T cells can augment autoantibody-induced arthritis Functional Analysis of the rs774872314, rs116171003, rs200231898 and rs201107751 Polymorphisms in the Human RORγT Gene Promoter Region.Activated IL-23/IL-17 pathway closely correlates with increased Foxp3 expression in livers of chronic hepatitis B patients.A crucial role for tumor necrosis factor receptor 1 in synovial lining cells and the reticuloendothelial system in mediating experimental arthritis Transgenic models for cytokine-induced neurological disease.Cutting edge: CTLA-4--B7 interaction suppresses Th17 cell differentiation Mislocalization of SLP-76 leads to aberrant inflammatory cytokine and autoantibody production Rag2-deficient IL-1 Receptor Antagonist-deficient Mice Are a Novel Colitis Model in Which Innate Lymphoid Cell-derived IL-17 Is Involved in the Pathogenesis.Increased frequencies of Th22 cells as well as Th17 cells in the peripheral blood of patients with ankylosing spondylitis and rheumatoid arthritis The adaptor protein CIKS/ACT1 is necessary for collagen-induced arthritis, and it contributes to the production of collagen-specific antibody Corneal transplantation and immune privilege.CD4+ T cells sensitized by vascular smooth muscle induce vasculitis, and interferon gamma is critical for the initiation of vascular pathology.Detection of T cell responses to a ubiquitous cellular protein in autoimmune disease Bone formation and resorption are both increased in experimental autoimmune arthritis.Th17 cells can provide B cell help in autoantibody induced arthritis.TNFα promotes Th17 cell differentiation through IL-6 and IL-1β produced by monocytes in rheumatoid arthritis.Two different regulatory T cell populations that promote corneal allograft survival.Cutting edge: TCR revision affects predominantly Foxp3 cells and skews them toward the Th17 lineage.Interleukin 6 in autoimmune and inflammatory diseases: a personal memoir Immune aging and autoimmunity.

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P2860

T cell self-reactivity forms a cytokine milieu for spontaneous development of IL-17+ Th cells that cause autoimmune arthritis.

http://www.wikidata.org/entity/Q42767257

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2007 nî lūn-bûn

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T cell self-reactivity forms a ...... at cause autoimmune arthritis.

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Journal of Experimental Medicine

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Hiroyuki Yoshitomi

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Motomu Hashimoto

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Noriko Sakaguchi

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Satoshi Tanaka

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Shimon Sakaguchi

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Takashi Nomura

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P2860

ZAP-70: a 70 kd protein-tyrosine kinase that associates with the TCR zeta chain

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

Immunologic self-tolerance maintained by activated T cells expressing IL-2 receptor alpha-chains (CD25). Breakdown of a single mechanism of self-tolerance causes various autoimmune diseases

Transforming growth factor-beta induces development of the T(H)17 lineage

A missense single-nucleotide polymorphism in a gene encoding a protein tyrosine phosphatase (PTPN22) is associated with rheumatoid arthritis.

Autoimmune-associated lymphoid tyrosine phosphatase is a gain-of-function variant

Interleukin-17 family members and inflammation

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17

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41-47

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10.1084/JEM.20062259

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1

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204

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Yoichiro Iwakura

Tomoyuki Yamaguchi

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2007-01-16T00:00:00Z

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6574560

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17227914

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2118414

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