about
Fate mapping of IL-17-producing T cells in inflammatory responsesThe aryl hydrocarbon receptor links TH17-cell-mediated autoimmunity to environmental toxinsActivation of the aryl hydrocarbon receptor dampens the severity of inflammatory skin conditions.Detection of T cell responses to a ubiquitous cellular protein in autoimmune diseaseAn IL-9 fate reporter demonstrates the induction of an innate IL-9 response in lung inflammationRegulation and function of innate and adaptive interleukin-17-producing cellsPreferential recruitment of CCR6-expressing Th17 cells to inflamed joints via CCL20 in rheumatoid arthritis and its animal model.Treatment of advanced tumors with agonistic anti-GITR mAb and its effects on tumor-infiltrating Foxp3+CD25+CD4+ regulatory T cells.Plasticity of Th17 cells in Peyer's patches is responsible for the induction of T cell-dependent IgA responsesNatural agonists for aryl hydrocarbon receptor in culture medium are essential for optimal differentiation of Th17 T cells.Differential influences of the aryl hydrocarbon receptor on Th17 mediated responses in vitro and in vivo.Development, regulation and functional capacities of Th17 cells.External influences on the immune system via activation of the aryl hydrocarbon receptor.Control of immune responses by antigen-specific regulatory T cells expressing the folate receptor.Clinical and Histopathological Amelioration of Experimental Autoimmune Encephalomyelitis by AAV Vectors Expressing a Soluble Interleukin-23 Receptor.Complement drives Th17 cell differentiation and triggers autoimmune arthritis.Guidance of regulatory T cell development by Satb1-dependent super-enhancer establishment.T cell self-reactivity forms a cytokine milieu for spontaneous development of IL-17+ Th cells that cause autoimmune arthritis.Graded attenuation of TCR signaling elicits distinct autoimmune diseases by altering thymic T cell selection and regulatory T cell function.Interleukin-17-producing gammadelta T cells selectively expand in response to pathogen products and environmental signals.Dysbiosis Contributes to Arthritis Development via Activation of Autoreactive T Cells in the Intestine.Lipid raft dynamics linked to sperm competency for fertilization in mice.Addendum: Guidance of regulatory T cell development by Satb1-dependent super-enhancer establishment.Modulation of Th17 development and function by activation of the aryl hydrocarbon receptor--the role of endogenous ligands.Erratum: Guidance of regulatory T cell development by Satb1-dependent super-enhancer establishment.Foxp3-dependent and -independent molecules specific for CD25+CD4+ natural regulatory T cells revealed by DNA microarray analysis.Autoimmune Th17 Cells Induced Synovial Stromal and Innate Lymphoid Cell Secretion of the Cytokine GM-CSF to Initiate and Augment Autoimmune Arthritis.Satb1 regulates the effector program of encephalitogenic tissue Th17 cells in chronic inflammationIdentification of a genomic enhancer that enforces proper apoptosis induction in thymic negative selection.Arthritis and pneumonitis produced by the same T cell clones from mice with spontaneous autoimmune arthritisConversion of antigen-specific effector/memory T cells into Foxp3-expressing Treg cells by inhibition of CDK8/19Correction to: The pathogenicity of Th17 cells in autoimmune diseasesThe pathogenicity of Th17 cells in autoimmune diseasesImpaired T cell receptor signaling and development of T cell-mediated autoimmune arthritisSynovial Tissue Inflammation Mediated by Autoimmune T CellsTreatment of advanced tumors with agonistic anti-GITR mAb and its effects on tumor-infiltrating Foxp3CD25CD4 regulatory T cells
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Keiji Hirota
@ast
Keiji Hirota
@en
Keiji Hirota
@es
Keiji Hirota
@nl
type
label
Keiji Hirota
@ast
Keiji Hirota
@en
Keiji Hirota
@es
Keiji Hirota
@nl
prefLabel
Keiji Hirota
@ast
Keiji Hirota
@en
Keiji Hirota
@es
Keiji Hirota
@nl
P106
P108
P31
P496
0000-0002-4737-0661