Transformation by ras modifies AP1 composition and activity.
about
Critical role of c-Jun overexpression in liver metastasis of human breast cancer xenograft modelKaposi's sarcoma-associated herpesvirus induction of AP-1 and interleukin 6 during primary infection mediated by multiple mitogen-activated protein kinase pathways.Multiple mitogen-activated protein kinase signaling pathways connect the cot oncoprotein to the c-jun promoter and to cellular transformationJunB potentiates function of BRCA1 activation domain 1 (AD1) through a coiled-coil-mediated interactionWnt signalling in lung development and diseasesA KRAS-directed transcriptional silencing pathway that mediates the CpG island methylator phenotypeA murine ATFa-associated factor with transcriptional repressing activityDistinct roles of Jun : Fos and Jun : ATF dimers in oncogenesisThe H3K27me3 demethylase JMJD3 contributes to the activation of the INK4A-ARF locus in response to oncogene- and stress-induced senescenceNeoplastic transformation of rat thyroid cells requires the junB and fra-1 gene induction which is dependent on the HMGI-C gene productp75-Ras-GRF1 is a c-Jun/AP-1 target protein: its up regulation results in increased Ras activity and is necessary for c-Jun-induced nonadherent growth of Rat1a cellsFra-1 promotes growth and survival in RAS-transformed thyroid cells by controlling cyclin A transcriptionLigand-specific sequential regulation of transcription factors for differentiation of MCF-7 cellsRegulation of a multigenic invasion programme by the transcription factor, AP-1: re-expression of a down-regulated gene, TSC-36, inhibits invasion.An association of a simultaneous nuclear and cytoplasmic localization of Fra-1 with breast malignancy.FRA-1 protein overexpression is a feature of hyperplastic and neoplastic breast disordersDeoxycholate induces COX-2 expression via Erk1/2-, p38-MAPK and AP-1-dependent mechanisms in esophageal cancer cells.A network of mitogen-activated protein kinases links G protein-coupled receptors to the c-jun promoter: a role for c-Jun NH2-terminal kinase, p38s, and extracellular signal-regulated kinase 5.Fra-1 induces morphological transformation and increases in vitro invasiveness and motility of epithelioid adenocarcinoma cells.Emerging roles of ATF2 and the dynamic AP1 network in cancer.KRAS and YAP1 converge to regulate EMT and tumor survivalInduction of postmitotic neuroretina cell proliferation by distinct Ras downstream signaling pathways.An extracellular signal-regulated kinase 1- and 2-dependent program of chromatin trafficking of c-Fos and Fra-1 is required for cyclin D1 expression during cell cycle reentry.Regulation of interleukin-12 gene expression and its anti-tumor activities by prostaglandin E2 derived from mammary carcinomasThe mammalian Jun proteins: redundancy and specificity.Transactivation of Fra-1 and consequent activation of AP-1 occur extracellular signal-regulated kinase dependently.E-cadherin transcriptional downregulation by promoter methylation but not mutation is related to epithelial-to-mesenchymal transition in breast cancer cell lines.Signal transduction mediated by the Ras/Raf/MEK/ERK pathway from cytokine receptors to transcription factors: potential targeting for therapeutic intervention.The elements of human cyclin D1 promoter and regulation involved.Oxidative stress promotes myofibroblast differentiation and tumour spreading.Reverse engineering a hierarchical regulatory network downstream of oncogenic KRAS.Invasion of normal human fibroblasts induced by v-Fos is independent of proliferation, immortalization, and the tumor suppressors p16INK4a and p53.The regulatory mechanism of the LY6K gene expression in human breast cancer cellsJunB breakdown in mid-/late G2 is required for down-regulation of cyclin A2 levels and proper mitosisThe PKCθ pathway participates in the aberrant accumulation of Fra-1 protein in invasive ER-negative breast cancer cells.A genetic variation of the p38β promoter region is correlated with an increased risk of sporadic colorectal cancerSynergistic activation of human involucrin gene expression by Fra-1 and p300--evidence for the presence of a multiprotein complex.The Fra-1-miR-134-SDS22 feedback loop amplifies ERK/JNK signaling and reduces chemosensitivity in ovarian cancer cells.Extracellular signal-regulated kinase signaling pathway regulates breast cancer cell migration by maintaining slug expression.Epigenetic regulation of miR-21 in colorectal cancer: ITGB4 as a novel miR-21 target and a three-gene network (miR-21-ITGΒ4-PDCD4) as predictor of metastatic tumor potential.
P2860
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P2860
Transformation by ras modifies AP1 composition and activity.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Transformation by ras modifies AP1 composition and activity.
@en
Transformation by ras modifies AP1 composition and activity.
@nl
type
label
Transformation by ras modifies AP1 composition and activity.
@en
Transformation by ras modifies AP1 composition and activity.
@nl
prefLabel
Transformation by ras modifies AP1 composition and activity.
@en
Transformation by ras modifies AP1 composition and activity.
@nl
P2093
P356
P1433
P1476
Transformation by ras modifies AP1 composition and activity.
@en
P2093
P2888
P304
P356
10.1038/SJ.ONC.1200900
P407
P577
1997-02-01T00:00:00Z
P5875
P6179
1003123705