Skeletal malformations caused by overexpression of Cbfa1 or its dominant negative form in chondrocytes. - Wikidata - wikimedia - TriplyDB

Skeletal malformations caused by overexpression of Cbfa1 or its dominant negative form in chondrocytes.

Skeletal malformations caused by overexpression of Cbfa1 or its dominant negative form in chondrocytes.

http://www.wikidata.org/entity/Q42934690

about

Smad6 interacts with Runx2 and mediates Smad ubiquitin regulatory factor 1-induced Runx2 degradation Hey1 basic helix-loop-helix protein plays an important role in mediating BMP9-induced osteogenic differentiation of mesenchymal progenitor cells The life cycle of chondrocytes in the developing skeleton.Wnt signaling in cartilage development and diseases: lessons from animal studies A pathway to bone: signaling molecules and transcription factors involved in chondrocyte development and maturation Loss of the putative catalytic domain of HDAC4 leads to reduced thermal nociception and seizures while allowing normal bone development The transcription factor CCAAT-binding factor CBF/NF-Y regulates the proximal promoter activity in the human alpha 1(XI) collagen gene (COL11A1)Basic helix-loop-helix protein DEC1 promotes chondrocyte differentiation at the early and terminal stages Critical roles for collagenase-3 (Mmp13) in development of growth plate cartilage and in endochondral ossification Nkx3.2-mediated repression of Runx2 promotes chondrogenic differentiation Fibroblast growth factor signaling regulates Dach1 expression during skeletal development Distribution of the transcription factors Sox9, AP-2, and [delta]EF1 in adult murine articular and meniscal cartilage and growth plate Runx2 and Runx3 are essential for chondrocyte maturation, and Runx2 regulates limb growth through induction of Indian hedgehog Growth defect in Grg5 null mice is associated with reduced Ihh signaling in growth plates Dlx5 Is a cell autonomous regulator of chondrocyte hypertrophy in mice and functionally substitutes for Dlx6 during endochondral ossification Molecular development of fibular reduction in birds and its evolution from dinosaurs SnoN suppresses maturation of chondrocytes by mediating signal cross-talk between transforming growth factor-β and bone morphogenetic protein pathways.Dmp1-deficient mice display severe defects in cartilage formation responsible for a chondrodysplasia-like phenotype Nonthermal atmospheric pressure plasma enhances mouse limb bud survival, growth, and elongation Increased Runx2 expression associated with enhanced Wnt signaling in PDLLA internal fixation for fracture treatment.The gene for aromatase, a rate-limiting enzyme for local estrogen biosynthesis, is a downstream target gene of Runx2 in skeletal tissues.Toward regeneration of articular cartilage.MicroRNA-140 plays dual roles in both cartilage development and homeostasis.PTHrP induces autocrine/paracrine proliferation of bone tumor cells through inhibition of apoptosis Dysregulation of chondrogenesis in human cleidocranial dysplasia Haploinsufficiency of HDAC4 causes brachydactyly mental retardation syndrome, with brachydactyly type E, developmental delays, and behavioral problems.Mechanisms balancing skeletal matrix synthesis and degradation.Indian hedgehog signaling regulates transcription and expression of collagen type X via Runx2/Smads interactions.Zfp521 is a target gene and key effector of parathyroid hormone-related peptide signaling in growth plate chondrocytes.Runx2-I isoform contributes to fetal bone formation even in the absence of specific N-terminal amino acids.Overexpression of Galnt3 in chondrocytes resulted in dwarfism due to the increase of mucin-type O-glycans and reduction of glycosaminoglycans Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.Increased adipogenesis in cultured embryonic chondrocytes and in adult bone marrow of dominant negative Erg transgenic mice Regulatory mechanisms of osteoblast and osteoclast differentiation.Runx2, a multifunctional transcription factor in skeletal development.Osteogenic potential of osteoblasts from neonatal rats born to mothers treated with caffeine throughout pregnancy Wdr5 is required for chick skeletal development.Runx2 inhibits chondrocyte proliferation and hypertrophy through its expression in the perichondrium JMJD3 promotes chondrocyte proliferation and hypertrophy during endochondral bone formation in mice Control of osteoblast function and regulation of bone mass.

P2860

Q24296392-F9F461E6-8CC2-41E4-BF1F-0EFB44B63D81 Q24655929-9418771D-73C9-41C7-9E53-4C6F85BAB80D Q24806072-4F772F1C-7ABD-46A0-9F2E-EC7EA19760E3 Q26774008-72063E41-8E87-4C37-B7D4-2F2FFB864B6C Q26995910-549C4246-E93F-4499-B471-09969568F328 Q27345064-30C44D85-26B4-4860-867A-33BD31D3DCEA Q28178924-8C47C90F-F94A-45EA-BAC9-DD622B4D9D2C Q28207957-59AB6771-11D1-44A7-96A9-E7AC499D4637 Q28505336-71E14A6B-49C7-40A5-B7FC-9FBFB6A16832 Q28506917-3236BE9C-EE28-490F-A5AD-A5296452E0FF Q28507465-4C5E2C6C-7331-48FD-A2E4-142D7E7F8E70 Q28511841-74AF5971-338B-4152-96FB-89F5E8B49A2F Q28586013-5C3E0734-4238-48E4-8FF6-85216AEB464D Q28587801-D6508A89-CB60-4A8C-A748-BFC2DD5F18C9 Q28588130-5DCACF3E-F0A0-4CB4-8C1E-C4ACBD93F224 Q28597226-29B2B387-A763-41F8-89AA-D1754FC4E237 Q30450699-71C82570-05F4-4D55-BDA3-20906EBFB191 Q33209762-D60E4DA4-831F-4316-9961-526DD91EAE5C Q33462524-CFDC320E-BFF4-4992-A3D8-A43A9B4607C4 Q33723759-83A774DA-CEBB-4DAB-AD33-21FE5673476A Q33826530-895C88DF-E59E-4894-A649-481CD60A69D8 Q33872729-582B66B2-443A-4204-BB16-2844AE9A396C Q33885698-DDC6F69B-8A47-408E-BF7C-BECC510A9C72 Q33916140-3CAF1B88-FCE3-4AF5-B800-74B891E84267 Q34015442-F86CCBA3-9A1C-4C98-BEEA-1AED452EBEC7 Q34050919-37BB8271-BA7E-4BF6-B37E-4F8F468DBA0A Q34129759-18D42432-ED44-4B49-84C8-FA71C5442AC1 Q34138974-295CA252-7B74-4C8D-8599-4DFBD3A12129 Q34217226-5C1D3143-2BD7-48E2-B63A-7FA8E66D2FE7 Q34227055-A743447F-FAE4-47AA-8B4C-753307987E0D Q34249099-0EEA9B8E-0F7E-426E-8F9A-FB2A47F578DC Q34333682-0DE008F1-498E-454E-90A4-EDAD74806AF4 Q34479060-409A3A22-19C2-4B4F-A176-5340B9F35919 Q34728971-8A6A474B-C8C0-4FD2-BBE2-24104F386F57 Q34811834-7A5520D5-3500-4BBF-BC6D-D79E5733EE64 Q35072810-3E525BA8-FB09-45B6-8D5D-D176122454FD Q35083564-051C0B45-9EBA-4EA1-B624-59E0447BCB9C Q35106164-3E04DC10-6B50-483F-BF32-2622F3CF21DB Q35127287-1C483C51-E4C0-4487-8F42-C02E4477F1D5 Q35128237-43A03918-A8B6-4BB2-8855-F387DF301E32

P2860

Skeletal malformations caused by overexpression of Cbfa1 or its dominant negative form in chondrocytes.

http://www.wikidata.org/entity/Q42934690

description

2001 nî lūn-bûn

@nan

2001年の論文

@ja

2001年論文

@yue

2001年論文

@zh-hant

2001年論文

@zh-hk

2001年論文

@zh-mo

2001年論文

@zh-tw

2001年论文

@wuu

2001年论文

@zh

2001年论文

@zh-cn

name

Skeletal malformations caused ...... negative form in chondrocytes.

@en

Skeletal malformations caused ...... negative form in chondrocytes.

@nl

type

label

Skeletal malformations caused ...... negative form in chondrocytes.

@en

Skeletal malformations caused ...... negative form in chondrocytes.

@nl

prefLabel

Skeletal malformations caused ...... negative form in chondrocytes.

@en

Skeletal malformations caused ...... negative form in chondrocytes.

@nl

P1433

Q42934690-794F8D73-CF84-4EF7-9ADC-095F1C5DC7BD

P1476

Q42934690-2FDFD259-AAC6-4EB0-84EC-F89DF4FC2048

P2093

Q42934690-0D139538-E025-41A7-8C0E-B81E6CE296DE

Q42934690-0E511D53-7902-4067-8FF3-9B5748FADC70

Q42934690-5223633B-FB40-40DA-B07C-B0053310C47F

Q42934690-55CD9CB4-226A-47F8-AC6F-FD9E49B80FBC

Q42934690-6FB317C0-2565-4C03-A65B-3E6E8413BB69

Q42934690-791D26F9-8D60-4FB9-99E5-720DF379C306

Q42934690-7D934017-B7E0-461D-B923-FD6A1846EB9F

Q42934690-84A82020-DD3E-4BA5-BDCC-EABE3318B8E6

Q42934690-8BDCB20C-6333-4419-BE58-A4FF338C2A27

Q42934690-A268AF5D-9017-4201-A02C-CFBB5B323F04

P2860

Q42934690-0142ACCD-0E1A-4F5B-AC77-2652795C3372

Q42934690-020E5C02-1CC6-40F2-A5A1-7B66D4BAC7FC

Q42934690-03DE3259-7220-4DEF-8BB3-9F742E0EDB98

Q42934690-0AA632DE-B331-4F32-A9FF-FBF6942B7E75

Q42934690-0F96EB6A-A512-4020-BD13-0A628813CEE1

Q42934690-1355CD38-F8FB-481E-A919-3A12ECF32B1C

Q42934690-18BEE671-A5D5-4AA4-B3D9-35306ADB2BBA

Q42934690-226B7727-2420-4FCD-BDC9-FC0C6B97BC5E

Q42934690-27392F8D-6081-4F5E-AB44-54EB1F895123

Q42934690-424CE3C4-9BE2-4A1B-AE00-DA348700403B

P304

Q42934690-E706069E-431E-489C-A84E-389CEF618052

P31

Q42934690-2AF05EFC-CB6E-4E1B-9B60-97F243FF5AA8

P356

Q42934690-A0F3AE7A-FBA1-472C-9B1A-744009403D94

P407

Q42934690-0DD8C033-0BBE-4F67-8E54-DA4CD45F249C

P433

Q42934690-31553601-B79F-4E9C-BE19-4FF0810220B9

P478

Q42934690-522838D7-6EE6-42F0-A569-D6D5A22CF30E

P577

Q42934690-A328BFBB-E962-42E3-8EAB-046B029E92BA

P5875

Q42934690-564B81D7-C401-4698-A55E-785BC4343C18

P698

Q42934690-50FC05B4-77EC-40C8-9F44-4EB252919530

P921

Q42934690-E6ADAFDB-10CB-49E0-A422-28CDC4847EB2

P932

Q42934690-385A7CE5-6966-45AE-971B-546B51F5296B

P356

P1433

Journal of Cell Biology

P1476

Skeletal malformations caused ...... negative form in chondrocytes.

@en

P2093

Enomoto H

http://www.w3.org/2001/XMLSchema#string

Enomoto-Iwamoto M

http://www.w3.org/2001/XMLSchema#string

Iwamoto M

http://www.w3.org/2001/XMLSchema#string

Kanatani N

http://www.w3.org/2001/XMLSchema#string

Komori T

http://www.w3.org/2001/XMLSchema#string

Kurisu K

http://www.w3.org/2001/XMLSchema#string

Liu Y

http://www.w3.org/2001/XMLSchema#string

Nakata K

http://www.w3.org/2001/XMLSchema#string

Ohmori T

http://www.w3.org/2001/XMLSchema#string

Takada K

http://www.w3.org/2001/XMLSchema#string

P2860

Bone morphogenetic protein signaling is required for maintenance of differentiated phenotype, control of proliferation, and hypertrophy in chondrocytes

Indian hedgehog signaling regulates proliferation and differentiation of chondrocytes and is essential for bone formation

Cbfa1, a candidate gene for cleidocranial dysplasia syndrome, is essential for osteoblast differentiation and bone development

Osf2/Cbfa1: a transcriptional activator of osteoblast differentiation

Limb alterations in brachypodism mice due to mutations in a new member of the TGF beta-superfamily

Regulation of rate of cartilage differentiation by Indian hedgehog and PTH-related protein

Maturational disturbance of chondrocytes in Cbfa1-deficient mice

Regulation of chondrocyte differentiation by Cbfa1

Modulated expression of type X collagen in Meckel's cartilage with different developmental fates

Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts

P304

87-100

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1083/JCB.153.1.87

http://www.w3.org/2001/XMLSchema#string

P407

P433

1

http://www.w3.org/2001/XMLSchema#string

P478

153

http://www.w3.org/2001/XMLSchema#string

P577

2001-04-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

277432117

http://www.w3.org/2001/XMLSchema#string

P698

11285276

http://www.w3.org/2001/XMLSchema#string

P921

P932

2185519

http://www.w3.org/2001/XMLSchema#string