Diminished production of anti-inflammatory mediators during neutrophil apoptosis and macrophage phagocytosis in chronic granulomatous disease (CGD).
about
Chronic Granulomatous Disease; fundamental stages in our understanding of CGDPPARγ and the Innate Immune System Mediate the Resolution of InflammationGenetic disorders coupled to ROS deficiencyMechanism-Based Strategies for the Management of Autoimmunity and Immune Dysregulation in Primary ImmunodeficienciesNeutrophils regulate tissue Neutrophilia in inflammation via the oxidant-modified lipid lysophosphatidylserineGenome-wide association study identifies nox3 as a critical gene for susceptibility to noise-induced hearing loss.NOX2-dependent regulation of inflammationGenetic dissection of the ity3 locus identifies a role for ncf2 co-expression modules and suggests selp as a candidate gene underlying the ity3.2 locus.Tryptophan/kynurenine metabolism in human leukocytes is independent of superoxide and is fully maintained in chronic granulomatous disease.Relationships between the phagocytic ability of milk macrophages and polymorphonuclear cells and somatic cell counts in uninfected cowsPPARγ activation normalizes resolution of acute sterile inflammation in murine chronic granulomatous disease.Gene therapy of chronic granulomatous disease: the engraftment dilemmaSimultaneous Host-Pathogen Transcriptome Analysis during Granulibacter bethesdensis Infection of Neutrophils from Healthy Subjects and Patients with Chronic Granulomatous DiseaseChronic granulomatous disease: why an inflammatory disease?Chronic granulomatous disease: a review of the infectious and inflammatory complications.Pioglitazone restores phagocyte mitochondrial oxidants and bactericidal capacity in chronic granulomatous disease.Impaired phagocytosis of apoptotic cells by macrophages in chronic granulomatous disease is reversed by IFN-γ in a nitric oxide-dependent mannerRescuing of deficient killing and phagocytic activities of macrophages derived from non-obese diabetic mice by treatment with geldanamycin or heat shock: potential clinical implications.Adaptive immune defects against glycoantigens in chronic granulomatous disease via dysregulated nitric oxide productionTGF-beta: the perpetrator of immune suppression by regulatory T cells and suicidal T cells.NOX2 As a Target for Drug Development: Indications, Possible Complications, and Progress.Regulation of innate immunity by NADPH oxidase.Impaired efferocytosis in human chronic granulomatous disease is reversed by pioglitazone treatmentMacrophages programmed by apoptotic cells inhibit epithelial-mesenchymal transition in lung alveolar epithelial cells via PGE2, PGD2, and HGFPPARγ activation following apoptotic cell instillation promotes resolution of lung inflammation and fibrosis via regulation of efferocytosis and proresolving cytokines.Derivation and functional analysis of patient-specific induced pluripotent stem cells as an in vitro model of chronic granulomatous diseaseCutaneous and other lupus-like symptoms in carriers of X-linked chronic granulomatous disease: incidence and autoimmune serology.Impact of reactive oxygen species (ROS) on the control of parasite loads and inflammation in Leishmania amazonensis infectionG-CSF activation of AKT is not sufficient to prolong neutrophil survivalLiver X receptor and STAT1 cooperate downstream of Gas6/Mer to induce anti-inflammatory arginase 2 expression in macrophages.Reactive oxygen species regulate neutrophil recruitment and survival in pneumococcal pneumoniaImpaired apoptotic cell clearance in CGD due to altered macrophage programming is reversed by phosphatidylserine-dependent production of IL-4Neutrophils from patients with SAPHO syndrome show no signs of aberrant NADPH oxidase-dependent production of intracellular reactive oxygen species.Persistence of the bacterial pathogen Granulibacter bethesdensis in chronic granulomatous disease monocytes and macrophages lacking a functional NADPH oxidase.Nonresolving inflammation in gp91phox-/- mice, a model of human chronic granulomatous disease, has lower adenosine and cyclic adenosine 5'-monophosphateWound healing essentials: let there be oxygenChronic granulomatous disease as a risk factor for autoimmune disease.Crohn's as an immune deficiency: from apparent paradox to evolving paradigm.miR-15a/16 regulates macrophage phagocytosis after bacterial infection.Association Between Discoid Lupus Erythematosus and Chronic Granulomatous Disease--Report of Two Cases and Review of the Literature.
P2860
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P2860
Diminished production of anti-inflammatory mediators during neutrophil apoptosis and macrophage phagocytosis in chronic granulomatous disease (CGD).
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Diminished production of anti- ...... c granulomatous disease (CGD).
@en
Diminished production of anti- ...... chronic granulomatous disease
@nl
type
label
Diminished production of anti- ...... c granulomatous disease (CGD).
@en
Diminished production of anti- ...... chronic granulomatous disease
@nl
prefLabel
Diminished production of anti- ...... c granulomatous disease (CGD).
@en
Diminished production of anti- ...... chronic granulomatous disease
@nl
P2093
P2860
P356
P1476
Diminished production of anti- ...... c granulomatous disease (CGD).
@en
P2093
David Goldblatt
Joanna Buddle
Joanne R Brown
Louise Morton
P2860
P304
P356
10.1189/JLB.1202599
P577
2003-05-01T00:00:00Z