Hyperfibrinolysis is common in out-of-hospital cardiac arrest: results from a prospective observational thromboelastometry study.
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Discrepant fibrinolytic response in plasma and whole blood during experimental endotoxemia in healthy volunteersDiverse coagulopathies in a rabbit model with different abdominal injuries.Tranexamic acid for treatment and prophylaxis of bleeding and hyperfibrinolysis.Acute myocardial infarction is associated with endothelial glycocalyx and cell damage and a parallel increase in circulating catecholamines.Effect of mild hypothermia on the coagulation-fibrinolysis system and physiological anticoagulants after cardiopulmonary resuscitation in a porcine modelHyperfibrinolysis, physiologic fibrinolysis, and fibrinolysis shutdown: the spectrum of postinjury fibrinolysis and relevance to antifibrinolytic therapyAssociation between biomarkers of endothelial injury and hypocoagulability in patients with severe sepsis: a prospective study.Fibrinolysis shutdown phenotype masks changes in rodent coagulation in tissue injury versus hemorrhagic shock.Thromboelastometric analysis of the risk factors for return of spontaneous circulation in adult patients with out-of-hospital cardiac arrestShock-induced systemic hyperfibrinolysis is attenuated by plasma-first resuscitation.Overwhelming tPA release, not PAI-1 degradation, is responsible for hyperfibrinolysis in severely injured trauma patients.Acute Fibrinolysis Shutdown after Injury Occurs Frequently and Increases Mortality: A Multicenter Evaluation of 2,540 Severely Injured Patients.Viscoelastic measurements of platelet function, not fibrinogen function, predicts sensitivity to tissue-type plasminogen activator in trauma patients.Endothelial Dysfunction in Resuscitated Cardiac Arrest (ENDO-RCA): safety and efficacy of low-dose prostacyclin administration and blood pressure target in addition to standard therapy, as compared to standard therapy alone, in post-cardiac arrest sShock induced endotheliopathy (SHINE) in acute critical illness - a unifying pathophysiologic mechanism.Restoring hemostasis: fibrinogen concentrate versus cryoprecipitate.Viscoelastic Tissue Plasminogen Activator Challenge Predicts Massive Transfusion in 15 Minutes.[Uncritical use of tranexamic acid in trauma patients : Do no further harm!]Coagulofibrinolytic Changes in Patients with Post-cardiac Arrest Syndrome.Rationale for the selective administration of tranexamic acid to inhibit fibrinolysis in the severely injured patient.Tissue injury suppresses fibrinolysis after hemorrhagic shock in nonhuman primates (rhesus macaque).Increased Fibrinolysis as a Specific Marker of Poor Outcome After Cardiac Arrest
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P2860
Hyperfibrinolysis is common in out-of-hospital cardiac arrest: results from a prospective observational thromboelastometry study.
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name
Hyperfibrinolysis is common in ...... onal thromboelastometry study.
@en
Hyperfibrinolysis is common in ...... onal thromboelastometry study.
@nl
type
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Hyperfibrinolysis is common in ...... onal thromboelastometry study.
@en
Hyperfibrinolysis is common in ...... onal thromboelastometry study.
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prefLabel
Hyperfibrinolysis is common in ...... onal thromboelastometry study.
@en
Hyperfibrinolysis is common in ...... onal thromboelastometry study.
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P2093
P1433
P1476
Hyperfibrinolysis is common in ...... onal thromboelastometry study.
@en
P2093
C J Schlimp
J Cadamuro
P304
P356
10.1016/J.RESUSCITATION.2012.08.318
P577
2012-08-23T00:00:00Z