Glutamate receptors in the rat medial prefrontal cortex regulate set-shifting ability.
about
Positive allosteric modulation of metabotropic glutamate 5 (mGlu5) receptors reverses N-Methyl-D-aspartate antagonist-induced alteration of neuronal firing in prefrontal cortexNeural structures underlying set-shifting: roles of medial prefrontal cortex and anterior cingulate cortexLimbic circuitry of the midline thalamusActivation of metabotropic glutamate 2/3 receptors reverses the effects of NMDA receptor hypofunction on prefrontal cortex unit activity in awake ratsModulation of the Endocannabinoids N-Arachidonoylethanolamine (AEA) and 2-Arachidonoylglycerol (2-AG) on Executive Functions in Humans1-aminocyclopropanecarboxylic acid (ACPC) produces procognitive but not antipsychotic-like effects in rats.Using the MATRICS to guide development of a preclinical cognitive test battery for research in schizophrenia.Methamphetamine self-administration produces attentional set-shifting deficits and alters prefrontal cortical neurophysiology in rats.Adolescent alcohol exposure reduces behavioral flexibility, promotes disinhibition, and increases resistance to extinction of ethanol self-administration in adulthood.The prelimbic cortex and subthalamic nucleus contribute to cue-guided behavioral switching.Antioxidant Treatment with N-acetyl Cysteine Prevents the Development of Cognitive and Social Behavioral Deficits that Result from Perinatal Ketamine TreatmentAMPA/kainate, NMDA, and dopamine D1 receptor function in the nucleus accumbens core: a context-limited role in the encoding and consolidation of instrumental memory.The mouse attentional-set-shifting task: a method for assaying successful cognitive aging?Contralateral disconnection of the rat prelimbic cortex and dorsomedial striatum impairs cue-guided behavioral switching.Disruption of performance in the five-choice serial reaction time task induced by administration of N-methyl-D-aspartate receptor antagonists: relevance to cognitive dysfunction in schizophreniaSerum glutamine, set-shifting ability and anorexia nervosa.Divergent plasticity of prefrontal cortex networks.Interaction of N-methyl-D-aspartate and group 5 metabotropic glutamate receptors on behavioral flexibility using a novel operant set-shift paradigm.Predictably irrational: assaying cognitive inflexibility in mouse models of schizophrenia.N-methyl-D-aspartate receptor-mediated glutamate transmission in nucleus accumbens plays a more important role than that in dorsal striatum in cognitive flexibility.Affective and cognitive mechanisms of risky decision making.Loss of GluN2A-containing NMDA receptors impairs extra-dimensional set-shiftingRule learning and reward contingency are associated with dissociable patterns of dopamine activation in the rat prefrontal cortex, nucleus accumbens, and dorsal striatum.Analysis and design of behavioral experiments to characterize population learning.Performance on a strategy set shifting task in rats following adult or adolescent cocaine exposure.Local N-methyl-d-aspartate receptor antagonism in the prefrontal cortex attenuates spatial cognitive deficits induced by gonadectomy in adult male rats.Aberrant learning and memory in addiction.Impaired attention and synaptic senescence of the prefrontal cortex involves redox regulation of NMDA receptors.Conditional loss of GluN2B in cortex and hippocampus impairs attentional set formation.Activation of type 5 metabotropic glutamate receptors attenuates deficits in cognitive flexibility induced by NMDA receptor blockade.The Novel, Nicotinic Alpha7 Receptor Partial Agonist, BMS-933043, Improves Cognition and Sensory Processing in Preclinical Models of Schizophrenia.Neural correlates of rules and conflict in medial prefrontal cortex during decision and feedback epochs.Distinct prefrontal cortical regions negatively regulate evoked activity in nucleus accumbens subregions.Targeting prefrontal cortical dopamine D1 and N-methyl-D-aspartate receptor interactions in schizophrenia treatment.Behavioral and neurophysiological analyses of dynamic learning processes.Behavioural pharmacology: 40+ years of progress, with a focus on glutamate receptors and cognition.Performance on a strategy set shifting task during adolescence in a genetic model of attention deficit/hyperactivity disorder: methylphenidate vs. atomoxetine treatmentsEarly developmental elevations of brain kynurenic acid impair cognitive flexibility in adults: reversal with galantamineAcute elevations of brain kynurenic acid impair cognitive flexibility: normalization by the alpha7 positive modulator galantamine.Adolescent behavior and dopamine availability are uniquely sensitive to dietary omega-3 fatty acid deficiency.
P2860
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P2860
Glutamate receptors in the rat medial prefrontal cortex regulate set-shifting ability.
description
2003 nî lūn-bûn
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2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
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2003年學術文章
@zh-hant
name
Glutamate receptors in the rat medial prefrontal cortex regulate set-shifting ability.
@en
Glutamate receptors in the rat medial prefrontal cortex regulate set-shifting ability.
@nl
type
label
Glutamate receptors in the rat medial prefrontal cortex regulate set-shifting ability.
@en
Glutamate receptors in the rat medial prefrontal cortex regulate set-shifting ability.
@nl
prefLabel
Glutamate receptors in the rat medial prefrontal cortex regulate set-shifting ability.
@en
Glutamate receptors in the rat medial prefrontal cortex regulate set-shifting ability.
@nl
P1476
Glutamate receptors in the rat medial prefrontal cortex regulate set-shifting ability.
@en
P2093
Karyn Groth
Mark R Stefani
P304
P356
10.1037/0735-7044.117.4.728
P577
2003-08-01T00:00:00Z