Hypoxic reduction in cellular glutathione levels requires mitochondrial reactive oxygen species.
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Biochemical and morphological changes in endothelial cells in response to hypoxic interstitial edema.Effects of TLR agonists on the hypoxia-regulated transcription factor HIF-1alpha and dendritic cell maturation under normoxic conditionsHypoxia and fetal heart development.Panobinostat reduces hypoxia-induced cisplatin resistance of non-small cell lung carcinoma cells via HIF-1α destabilizationMitochondrial dysfunction resulting from loss of cytochrome c impairs cellular oxygen sensing and hypoxic HIF-alpha activationConsequences of neonatal resuscitation with supplemental oxygen.Ischemia-reperfusion and cardioprotection: a delicate balance between reactive oxygen species generation and redox homeostasis.Flutamide protects against trauma-hemorrhage-induced liver injury via attenuation of the inflammatory response, oxidative stress, and apopotosis.Reactive oxygen species and cellular oxygen sensingResistance to chemotherapy in cancer: a complex and integrated cellular response.Hypoxia-induced and A2A adenosine receptor-independent T-cell suppression is short lived and easily reversible1H NMR detects different metabolic profiles in glioblastoma stem-like cells.Protective Effects of Peroxisome Proliferator-Activated Receptor-α Agonist, Wy14643, on Hypoxia/Reoxygenation Injury in Primary Rat HepatocytesDifferent mtDNA mutations modify tumor progression in dependence of the degree of respiratory complex I impairment.Fenretinide cytotoxicity is independent of both constitutive and pharmacologically modulated glutathione levels in pediatric acute lymphoblastic leukemia cells cultured at hypoxia.ICB3E induces iNOS expression by ROS-dependent JNK and ERK activation for apoptosis of leukemic cells.Induction of HIF-2alpha is dependent on mitochondrial O2 consumption in an O2-sensitive adrenomedullary chromaffin cell line.Critical role of mitochondrial glutathione in the survival of hepatocytes during hypoxia.Menadione triggers cell death through ROS-dependent mechanisms involving PARP activation without requiring apoptosisA Rationally Designed Reversible 'Turn-Off' Sensor for GlutathioneAge-related defects in erythrocyte 2,3-diphosphoglycerate metabolism in dementia.Pathophysiological hypoxia affects the redox state and IL-2 signalling of human CD4+ T cells and concomitantly impairs survival and proliferation.The expression of redox proteins in phyllodes tumor.Quantitative assessment of specific carbonic anhydrase inhibitors effect on hypoxic cells using electrical impedance assays.HIF-1α triggers long-lasting glutamate excitotoxicity via system xc- in cerebral ischaemia-reperfusion.Red blood cell metabolism under prolonged anaerobic storage.HIF-1α inhibition by diethylstilbestrol and its polyacetal conjugate in hypoxic prostate tumour cells: insights from NMR metabolomics.Hypoxic stabilization of mRNA is HIF-independent but requires mtROS
P2860
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P2860
Hypoxic reduction in cellular glutathione levels requires mitochondrial reactive oxygen species.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Hypoxic reduction in cellular ...... drial reactive oxygen species.
@en
Hypoxic reduction in cellular ...... drial reactive oxygen species.
@nl
type
label
Hypoxic reduction in cellular ...... drial reactive oxygen species.
@en
Hypoxic reduction in cellular ...... drial reactive oxygen species.
@nl
prefLabel
Hypoxic reduction in cellular ...... drial reactive oxygen species.
@en
Hypoxic reduction in cellular ...... drial reactive oxygen species.
@nl
P2093
P2860
P1476
Hypoxic reduction in cellular ...... drial reactive oxygen species.
@en
P2093
Brian Keith
Kyle D Mansfield
M Celeste Simon
P2860
P304
P356
10.1152/JAPPLPHYSIOL.00449.2004
P407
P577
2004-06-04T00:00:00Z