Specific inhibition of the mitochondrial permeability transition prevents lethal reperfusion injury.
about
Translocator protein (18 kDa): a promising therapeutic target and diagnostic tool for cardiovascular diseasesRescue of Heart Failure by Mitochondrial RecoveryMyocardial reperfusion injury: looking beyond primary PCIMechanisms of sudden cardiac death: oxidants and metabolismExperimental treatments for mitochondrial dysfunction in sepsis: A narrative reviewAttenuation of skeletal muscle and renal injury to the lower limb following ischemia-reperfusion using mPTP inhibitor NIM-811Endoplasmic reticulum stress in diabetic hearts abolishes erythropoietin-induced myocardial protection by impairment of phospho-glycogen synthase kinase-3beta-mediated suppression of mitochondrial permeability transitionIsoflurane preconditioning elicits competent endogenous mechanisms of protection from oxidative stress in cardiomyocytes derived from human embryonic stem cellsTranslocation of glycogen synthase kinase-3β (GSK-3β), a trigger of permeability transition, is kinase activity-dependent and mediated by interaction with voltage-dependent anion channel 2 (VDAC2).The targeting of cyclophilin D by RNAi as a novel cardioprotective therapy: evidence from two-photon imaging.Cardioprotective effects of cyclosporine A in an in vivo model of myocardial ischemia and reperfusion.Nonanticoagulant heparin reduces myocyte Na+ and Ca2+ loading during simulated ischemia and decreases reperfusion injury.Cyclosporine preserves mitochondrial morphology after myocardial ischemia/reperfusion independent of calcineurin inhibition.Cyclosporine A at reperfusion reduces infarct size in pigsMechanism for resveratrol-induced cardioprotection against reperfusion injury involves glycogen synthase kinase 3beta and mitochondrial permeability transition pore.The cardioprotective effect of necrostatin requires the cyclophilin-D component of the mitochondrial permeability transition poreMitochondrial morphology and cardiovascular diseaseCardiac mitochondria and arrhythmias.Quantifying acute myocardial injury using ratiometric fluorometry.In vivo fluorometric assessment of cyclosporine on mitochondrial function during myocardial ischemia and reperfusionRu360, a specific mitochondrial calcium uptake inhibitor, improves cardiac post-ischaemic functional recovery in rats in vivo.The role of mitochondria in protection of the heart by preconditioning.Mitochondrial kinase signalling pathways in myocardial protection from ischaemia/reperfusion-induced necrosis.Functional and pharmacological characteristics of permeability transition in isolated human heart mitochondria.Respiratory uncoupling by increased H(+) or K(+) flux is beneficial for heart mitochondrial turnover of reactive oxygen species but not for permeability transitionThe cardioprotective efficacy of TVP1022 in a rat model of ischaemia/reperfusion.Combination of cyclosporine and erythropoietin improves brain infarct size and neurological function in rats after ischemic stroke.Bundled postconditioning therapies improve hemodynamics and neurologic recovery after 17 min of untreated cardiac arrest.Urocortin prevents mitochondrial permeability transition in response to reperfusion injury indirectly by reducing oxidative stressMitochondrial cyclophilin-D as a potential therapeutic target for post-myocardial infarction heart failureA novel role for mitochondrial sphingosine-1-phosphate produced by sphingosine kinase-2 in PTP-mediated cell survival during cardioprotection.Cyclosporin A and cardioprotection: from investigative tool to therapeutic agent.Emerging beneficial roles of sirtuins in heart failure.The Mitochondrial Unfolded Protein Response Protects against Anoxia in Caenorhabditis elegansCyclosporin variably and inconsistently reduces infarct size in experimental models of reperfused myocardial infarction: a systematic review and meta-analysis.Cyclophilin D: knocking on death's door.Physiological and pathological roles of the mitochondrial permeability transition pore in the heart.Identification of ER-000444793, a Cyclophilin D-independent inhibitor of mitochondrial permeability transition, using a high-throughput screen in cryopreserved mitochondria.Astragaloside IV inhibits oxidative stress-induced mitochondrial permeability transition pore opening by inactivating GSK-3β via nitric oxide in H9c2 cardiac cells.Mitochondria as a drug target in ischemic heart disease and cardiomyopathy
P2860
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P2860
Specific inhibition of the mitochondrial permeability transition prevents lethal reperfusion injury.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Specific inhibition of the mit ...... nts lethal reperfusion injury.
@en
Specific inhibition of the mit ...... nts lethal reperfusion injury.
@nl
type
label
Specific inhibition of the mit ...... nts lethal reperfusion injury.
@en
Specific inhibition of the mit ...... nts lethal reperfusion injury.
@nl
prefLabel
Specific inhibition of the mit ...... nts lethal reperfusion injury.
@en
Specific inhibition of the mit ...... nts lethal reperfusion injury.
@nl
P2093
P1476
Specific inhibition of the mit ...... nts lethal reperfusion injury.
@en
P2093
Danina Muntean
Dominique Robert
Joseph Loufouat
Lara Chalabreysse
Laurent Argaud
Michel Ovize
Odile Gateau-Roesch
P304
P356
10.1016/J.YJMCC.2004.12.001
P577
2005-01-26T00:00:00Z