Estrogen receptor positivity in mammary tumors of Wnt-1 transgenic mice is influenced by collaborating oncogenic mutations.
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Selective estrogen receptor modulators inhibit growth and progression of premalignant lesions in a mouse model of ductal carcinoma in situ.Deconvoluting the obesity and breast cancer link: secretome, soil and seed interactionsMetabolic history impacts mammary tumor epithelial hierarchy and early drug response in mice.A mouse model for luminal epithelial like ER positive subtype of human breast cancer.Genetically engineered ERα-positive breast cancer mouse models.Wnt signaling can substitute for estrogen to induce division of ERalpha-positive cells in a mouse mammary tumor model.Somatic expression of PyMT or activated ErbB2 induces estrogen-independent mammary tumorigenesis.Expression of autotaxin and lysophosphatidic acid receptors increases mammary tumorigenesis, invasion, and metastases.Tip30 deletion in MMTV-Neu mice leads to enhanced EGFR signaling and development of estrogen receptor-positive and progesterone receptor-negative mammary tumors.Obesity, independent of p53 gene dosage, promotes mammary tumor progression and upregulates the p53 regulator microRNA-504.P53 genotype as a determinant of ER expression and tamoxifen response in the MMTV-Wnt-1 model of mammary carcinogenesisMouse models of estrogen receptor-positive breast cancerComparison of expression profiles of metastatic versus primary mammary tumors in MMTV-Wnt-1 and MMTV-Neu transgenic mice.Estrogen receptor-positive mammary tumorigenesis in TGFalpha transgenic mice progresses with progesterone receptor loss.Combination chemoprevention of HER2/neu-induced breast cancer using a cyclooxygenase-2 inhibitor and a retinoid X receptor-selective retinoidUnlocking the power of cross-species genomic analyses: identification of evolutionarily conserved breast cancer networks and validation of preclinical modelsEndocrine dysfunction in p27Kip1 deficient mice and susceptibility to Wnt-1 driven breast cancerMet induces diverse mammary carcinomas in mice and is associated with human basal breast cancer.MicroRNA-128 regulates the differentiation of rat bone mesenchymal stem cells into neuron-like cells by Wnt signaling.Wnt-1 is dominant over neu in specifying mammary tumor expression profiles.Hyperprolactinemia-inducing antipsychotics increase breast cancer risk by activating JAK-STAT5 in precancerous lesions.
P2860
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P2860
Estrogen receptor positivity in mammary tumors of Wnt-1 transgenic mice is influenced by collaborating oncogenic mutations.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Estrogen receptor positivity i ...... aborating oncogenic mutations.
@en
Estrogen receptor positivity i ...... aborating oncogenic mutations.
@nl
type
label
Estrogen receptor positivity i ...... aborating oncogenic mutations.
@en
Estrogen receptor positivity i ...... aborating oncogenic mutations.
@nl
prefLabel
Estrogen receptor positivity i ...... aborating oncogenic mutations.
@en
Estrogen receptor positivity i ...... aborating oncogenic mutations.
@nl
P2093
P2860
P356
P1433
P1476
Estrogen receptor positivity i ...... aborating oncogenic mutations.
@en
P2093
Gary C Chamness
Katrina Podsypanina
Rachel Schiff
Sarah Hatsell
Shixia Huang
Syed K Mohsin
Xiaomei Zhang
P2860
P2888
P304
P356
10.1038/SJ.ONC.1208597
P407
P577
2005-06-01T00:00:00Z