The decrease in the presynaptic calcium current is a major cause of short-term depression at a calyx-type synapse.
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Ca2+-dependent mechanisms of presynaptic control at central synapsesSynaptic vesicle pools and dynamicsElementary mechanisms producing facilitation of Cav2.1 (P/Q-type) channelsNeuroligin-2 deletion selectively decreases inhibitory synaptic transmission originating from fast-spiking but not from somatostatin-positive interneuronsCalmodulin regulation (calmodulation) of voltage-gated calcium channelsTowards a Unified Theory of Calmodulin Regulation (Calmodulation) of Voltage-Gated Calcium and Sodium ChannelsRyRCa2+ leak limits cardiac Ca2+ window current overcoming the tonic effect of calmodulinin miceA limited contribution of Ca2+ current facilitation to paired-pulse facilitation of transmitter release at the rat calyx of HeldSpontaneous Activity Defines Effective Convergence Ratios in an Inhibitory Circuit.Effects of Patterned Sound Deprivation on Short- and Long-Term Plasticity in the Rat Thalamocortical Auditory System In Vivo.Quantitative analysis of vesicle recycling at the calyx of Held synapse.Developmental changes in short-term plasticity at the rat calyx of Held synapse.Facilitation versus depression in cultured hippocampal neurons determined by targeting of Ca2+ channel Cavbeta4 versus Cavbeta2 subunits to synaptic terminalsShort-term plasticity and auditory processing in the ventral cochlear nucleus of normal and hearing-impaired animalsFormation and maturation of the calyx of Held.Endbulb synaptic depression within the range of presynaptic spontaneous firing and its impact on the firing reliability of cochlear nucleus bushy neurons.Diverse mechanisms underlie glycinergic feedback transmission onto rod bipolar cells in rat retina.Short-term synaptic depression and recovery at the mature mammalian endbulb of Held synapse in mice.Relative roles of different mechanisms of depression at the mouse endbulb of Held.Developmental shift to a mechanism of synaptic vesicle endocytosis requiring nanodomain Ca2+Systematic mapping of the state dependence of voltage- and Ca2+-dependent inactivation using simple open-channel measurements.Genetic, cellular, and functional evidence for Ca2+ inflow through Cav1.2 and Cav1.3 channels in murine spiral ganglion neurons.Calcium channels and synaptic transmission in familial hemiplegic migraine type 1 animal models.Calcineurin is universally involved in vesicle endocytosis at neuronal and nonneuronal secretory cellsRetinohypothalamic tract synapses in the rat suprachiasmatic nucleus demonstrate short-term synaptic plasticityOptical mapping of release properties in synapses.Contribution of calcium-dependent facilitation to synaptic plasticity revealed by migraine mutations in the P/Q-type calcium channel.Calcium imaging of auditory nerve fiber terminals in the cochlear nucleus.Exocytosis and endocytosis: modes, functions, and coupling mechanisms.Compound vesicle fusion increases quantal size and potentiates synaptic transmissionMaturation of calcium-dependent GABA, glycine, and glutamate release in the glycinergic MNTB-LSO pathwayPaired-pulse depression at photoreceptor synapses.A synaptic mechanism for retinal adaptation to luminance and contrastBrain-derived neurotrophic factor inhibits calcium channel activation, exocytosis, and endocytosis at a central nerve terminal.A three-pool model dissecting readily releasable pool replenishment at the calyx of heldMolecular determinants of CaV2.1 channel regulation by calcium-binding protein-1.Molecular determinants of modulation of CaV2.1 channels by visinin-like protein 2.Asynchronous Ca2+ current conducted by voltage-gated Ca2+ (CaV)-2.1 and CaV2.2 channels and its implications for asynchronous neurotransmitter release.Cholesterol regulates multiple forms of vesicle endocytosis at a mammalian central synapse.Glutamate transporter studies reveal the pruning of metabotropic glutamate receptors and absence of AMPA receptor desensitization at mature calyx of Held synapses.
P2860
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P2860
The decrease in the presynaptic calcium current is a major cause of short-term depression at a calyx-type synapse.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
The decrease in the presynapti ...... ssion at a calyx-type synapse.
@en
The decrease in the presynapti ...... ssion at a calyx-type synapse.
@nl
type
label
The decrease in the presynapti ...... ssion at a calyx-type synapse.
@en
The decrease in the presynapti ...... ssion at a calyx-type synapse.
@nl
prefLabel
The decrease in the presynapti ...... ssion at a calyx-type synapse.
@en
The decrease in the presynapti ...... ssion at a calyx-type synapse.
@nl
P1433
P1476
The decrease in the presynapti ...... ssion at a calyx-type synapse.
@en
P2093
Ling-Gang Wu
P304
P356
10.1016/J.NEURON.2005.03.024
P407
P50
P577
2005-05-01T00:00:00Z