Ethanol triggers neural crest apoptosis through the selective activation of a pertussis toxin-sensitive G protein and a phospholipase Cbeta-dependent Ca2+ transient.
about
Capturing structure and function in an embryonic heart with biophotonic tools.Connecting teratogen-induced congenital heart defects to neural crest cells and their effect on cardiac functionCalcium-mediated repression of β-catenin and its transcriptional signaling mediates neural crest cell death in an avian model of fetal alcohol syndromeFetal Alcohol Spectrum Disorder (FASD) Associated Neural Defects: Complex Mechanisms and Potential Therapeutic TargetsApoptosis of alcohol-exposed human placental cytotrophoblast cells is downstream of intracellular calcium signaling.Commonality in Down and fetal alcohol syndromes.Genomic factors that shape craniofacial outcome and neural crest vulnerability in FASD.High-throughput transcriptome sequencing identifies candidate genetic modifiers of vulnerability to fetal alcohol spectrum disorders.Rapid induction of apoptosis in gastrulating mouse embryos by ethanol and its prevention by HB-EGFEthanol suppresses PGC-1α expression by interfering with the cAMP-CREB pathway in neuronal cells.Epidermal growth factor-like growth factors prevent apoptosis of alcohol-exposed human placental cytotrophoblast cells.Transcriptome Profiling Identifies Ribosome Biogenesis as a Target of Alcohol Teratogenicity and Vulnerability during Early Embryogenesis.Neural crest development in fetal alcohol syndromeAgouti-related peptide and MC3/4 receptor agonists both inhibit excitatory hypothalamic ventromedial nucleus neuronsIntracellular calcium plays a critical role in the alcohol-mediated death of cerebellar granule neurons.An evolutionarily conserved mechanism of calcium-dependent neurotoxicity in a zebrafish model of fetal alcohol spectrum disordersElevated testosterone induces apoptosis in neuronal cells.CaMKII represses transcriptionally active β-catenin to mediate acute ethanol neurodegeneration and can phosphorylate β-catenin.CaMKII activation is a novel effector of alcohol's neurotoxicity in neural crest stem/progenitor cells.Genetic influence on dysmorphogenesis in embryos from different rat strains exposed to ethanol in vivo and in vitro.Structural constraints for alcohol-stimulated Ca2+ release in neural crest, and dual agonist/antagonist properties of n-octanol.Nifedipine Prevents Apoptosis of Alcohol-Exposed First-Trimester Trophoblast Cells.The avian embryo as a model for fetal alcohol spectrum disorder.Maternal Exposure to Ethanol During Pregnancy and Lactation Affects Glutamatergic System and Induces Oxidative Stress in Offspring Hippocampus.
P2860
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P2860
Ethanol triggers neural crest apoptosis through the selective activation of a pertussis toxin-sensitive G protein and a phospholipase Cbeta-dependent Ca2+ transient.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Ethanol triggers neural crest ...... beta-dependent Ca2+ transient.
@en
Ethanol triggers neural crest ...... beta-dependent Ca2+ transient.
@nl
type
label
Ethanol triggers neural crest ...... beta-dependent Ca2+ transient.
@en
Ethanol triggers neural crest ...... beta-dependent Ca2+ transient.
@nl
prefLabel
Ethanol triggers neural crest ...... beta-dependent Ca2+ transient.
@en
Ethanol triggers neural crest ...... beta-dependent Ca2+ transient.
@nl
P2093
P2860
P1476
Ethanol triggers neural crest ...... beta-dependent Ca2+ transient.
@en
P2093
Ana Garic-Stankovic
George R Flentke
Katherine A Debelak-Kragtorp
Marcos R Hernandez
Po Jen Chiang
Susan M Smith
P2860
P304
P356
10.1097/01.ALC.0000172460.05756.D9
P577
2005-07-01T00:00:00Z