Heat shock protein 90 inhibitors attenuate LPS-induced endothelial hyperpermeability. - Wikidata - wikimedia - TriplyDB

Heat shock protein 90 inhibitors attenuate LPS-induced endothelial hyperpermeability.

Heat shock protein 90 inhibitors attenuate LPS-induced endothelial hyperpermeability.

http://www.wikidata.org/entity/Q46775424

about

Novel regulators of endothelial barrier function LPS induces pp60c-src-mediated tyrosine phosphorylation of Hsp90 in lung vascular endothelial cells and mouse lung.Impedance analysis of GPCR-mediated changes in endothelial barrier function: overview and fundamental considerations for stable and reproducible measurements Mechanisms of nitric oxide synthase uncoupling in endotoxin-induced acute lung injury: role of asymmetric dimethylarginine.Comparative analysis of novel and conventional Hsp90 inhibitors on HIF activity and angiogenic potential in clear cell renal cell carcinoma: implications for clinical evaluation Pharmacological induction of the 70-kDa heat shock protein protects against brain injury Differential and opposing effects of imatinib on LPS- and ventilator-induced lung injury.LPS-induced delayed preconditioning is mediated by Hsp90 and involves the heat shock response in mouse kidney.PKC-dependent phosphorylation of eNOS at T495 regulates eNOS coupling and endothelial barrier function in response to G+ -toxins.Cytoskeletal mechanisms regulating vascular endothelial barrier function in response to acute lung injury Harvesting, identification and barrier function of human lung microvascular endothelial cells p53 protects against LPS-induced lung endothelial barrier dysfunction.Terazosin activates Pgk1 and Hsp90 to promote stress resistance c-Abl mediated tyrosine phosphorylation of paxillin regulates LPS-induced endothelial dysfunction and lung injury Regulators of endothelial and epithelial barrier integrity and function in acute lung injury.Resident Endothelial Cells and Endothelial Progenitor Cells Restore Endothelial Barrier Function After Inflammatory Lung Injury.Glutathione supplementation attenuates lipopolysaccharide-induced mitochondrial dysfunction and apoptosis in a mouse model of acute lung injury.Changes in endothelial connexin 43 expression inversely correlate with microvessel permeability and VE-cadherin expression in endotoxin-challenged lungs Histone deacetylase inhibitors prevent pulmonary endothelial hyperpermeability and acute lung injury by regulating heat shock protein 90 function.Heat shock protein 90 inhibitors protect and restore pulmonary endothelial barrier function.Hsp90 inhibition suppresses NF-κB transcriptional activation via Sirt-2 in human lung microvascular endothelial cells.Tripterine prevents endothelial barrier dysfunction by inhibiting endogenous peroxynitrite formation.Regulation of endothelial barrier function by TGF-β type I receptor ALK5: potential role of contractile mechanisms and heat shock protein 90.Heat shock protein 90 inhibitors prevent LPS-induced endothelial barrier dysfunction by disrupting RhoA signaling.Anti-inflammatory properties and pharmacological induction of Hsp70 after brain injury.Identification of RNA-binding Proteins in Macrophages by Interactome Capture Endothelial cell signaling and ventilator-induced lung injury: molecular mechanisms, genomic analyses, and therapeutic targets.Bone Morphogenetic Protein-Modulator BMPER Regulates Endothelial Barrier Function.Actin dynamics in the regulation of endothelial barrier functions and neutrophil recruitment during endotoxemia and sepsis.Hsp90 regulates NADPH oxidase activity and is necessary for superoxide but not hydrogen peroxide production.High glucose enhances LPS-stimulated human PMVEC hyperpermeability via the NO pathway.Metabolite identification in fecal microbiota transplantation mouse livers and combined proteomics with chronic unpredictive mild stress mouse livers.Activation of the sweet taste receptor, T1R3, by the artificial sweetener sucralose regulates the pulmonary endothelium.Catalpol restores LPS-elicited rat microcirculation disorder by regulation of a network of signaling involving inhibition of TLR-4 and SRC.Hsp70 Suppresses Mitochondrial Reactive Oxygen Species and Preserves Pulmonary Microvascular Barrier Integrity Following Exposure to Bacterial Toxins.

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P2860

Heat shock protein 90 inhibitors attenuate LPS-induced endothelial hyperpermeability.

http://www.wikidata.org/entity/Q46775424

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

@zh

2008年學術文章

@zh-hant

name

Heat shock protein 90 inhibitors attenuate LPS-induced endothelial hyperpermeability.

@en

Heat shock protein 90 inhibitors attenuate LPS-induced endothelial hyperpermeability.

@nl

type

label

Heat shock protein 90 inhibitors attenuate LPS-induced endothelial hyperpermeability.

@en

Heat shock protein 90 inhibitors attenuate LPS-induced endothelial hyperpermeability.

@nl

prefLabel

Heat shock protein 90 inhibitors attenuate LPS-induced endothelial hyperpermeability.

@en

Heat shock protein 90 inhibitors attenuate LPS-induced endothelial hyperpermeability.

@nl

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AJPLUNG.00350.2007

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American Journal of Physiology - Lung Cellular and Molecular Physiology

P1476

Heat shock protein 90 inhibitors attenuate LPS-induced endothelial hyperpermeability.

@en

P2093

Anuran Chatterjee

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Connie Snead

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Galina Antonova

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Gunay Yetik-Anacak

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Jingmin Zeng

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John D Catravas

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P2860

Heat shock protein 90 as an endogenous protein enhancer of inducible nitric-oxide synthase

Inhibition of heat shock protein HSP90-pp60v-src heteroprotein complex formation by benzoquinone ansamycins: essential role for stress proteins in oncogenic transformation.

Disruption of hsp90 function results in degradation of the death domain kinase, receptor-interacting protein (RIP), and blockage of tumor necrosis factor-induced nuclear factor-kappaB activation

Src kinase regulation by phosphorylation and dephosphorylation

VE-cadherin: adhesion at arm's length

Characterization of tyrosine phosphorylation of paxillin in vitro by focal adhesion kinase

Focal adhesions, contractility, and signaling

Protein nitration in rat lungs during hyperoxia exposure: a possible role of myeloperoxidase

The epidemiology of sepsis in the United States from 1979 through 2000

pp125FAK-dependent tyrosine phosphorylation of paxillin creates a high-affinity binding site for Crk

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L755-63

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