RORA, a large common fragile site gene, is involved in cellular stress response. - Wikidata - wikimedia - TriplyDB

RORA, a large common fragile site gene, is involved in cellular stress response.

RORA, a large common fragile site gene, is involved in cellular stress response.

http://www.wikidata.org/entity/Q46931410

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The FHIT gene product: tumor suppressor and genome "caretaker"ROR nuclear receptors: structures, related diseases, and drug discovery Tumor Suppressor Genes within Common Fragile Sites Are Active Players in the DNA Damage Response The retinoid-related orphan receptor alpha (RORA) gene and fear-related psychopathology.Convergence of linkage, gene expression and association data demonstrates the influence of the RAR-related orphan receptor alpha (RORA) gene on neovascular AMD: a systems biology based approach Prediction of potential cancer-risk regions based on transcriptome data: towards a comprehensive view Genetic variation of clock genes and cancer risk: a field synopsis and meta-analysis Retinoic acid-related orphan receptor-α is induced in the setting of DNA damage and promotes pulmonary emphysema.RORα binds to E2F1 to inhibit cell proliferation and regulate mammary gland branching morphogenesis Ligand regulation of retinoic acid receptor-related orphan receptors: implications for development of novel therapeutics Regulation of p53 stability and apoptosis by a ROR agonist RORα suppresses breast tumor invasion by inducing SEMA3F expression Action of RORs and their ligands in (patho)physiology.Prenatal diagnosis of a de novo interstitial deletion of 11q (11q22.3 → q23.3) associated with abnormal ultrasound findings by array comparative genomic hybridization.REV-ERB and ROR nuclear receptors as drug targets.Traumatic stress, oxidative stress and post-traumatic stress disorder: neurodegeneration and the accelerated-aging hypothesis.Prospective study of common variants in the retinoic acid receptor-related orphan receptor α gene and risk of neovascular age-related macular degeneration.Are common fragile sites merely structural domains or highly organized "functional" units susceptible to oncogenic stress?Genetic or epigenetic silencing of low density lipoprotein receptor-related protein 1B expression in oral squamous cell carcinoma.Genetics of age-related macular degeneration: current concepts, future directions.Retinoid-related Orphan Receptors (RORs): Roles in Cellular Differentiation and Development.Genomic instability in induced stem cells Microarray characterization of gene expression changes in blood during acute ethanol exposure The retinoid-related orphan receptor RORα promotes keratinocyte differentiation via FOXN1 Common chromosome fragile sites in human and murine epithelial cells and FHIT/FRA3B loss-induced global genome instability Retinoid-related orphan receptors (RORs): critical roles in development, immunity, circadian rhythm, and cellular metabolism The distribution of immunomodulatory cells in the lungs of patients with idiopathic pulmonary fibrosis Orphan nuclear receptors in breast cancer pathogenesis and therapeutic response.RORα, a potential tumor suppressor and therapeutic target of breast cancer.Retinoic Acid-Related Orphan Receptors (RORs): Regulatory Functions in Immunity, Development, Circadian Rhythm, and Metabolism.RORα switches transcriptional mode of ERRγ that results in transcriptional repression of CYP2E1 under ethanol-exposure.Genome-wide catalogue of chromosomal aberrations in barrett's esophagus and esophageal adenocarcinoma: a high-density single nucleotide polymorphism array analysis.RORα and RORγ expression inversely correlates with human melanoma progression.EPRS is a critical regulator of cell proliferation and estrogen signaling in ER+ breast cancer.Nuclear receptor retinoid-related orphan receptor alpha promotes apoptosis but is reduced in human gastric cancer.The interplay between retinoic acid receptor-related orphan receptors and human diseases.Molecular characterization of common fragile sites as a strategy to discover cancer susceptibility genes.The common fragile site FRA16D gene product WWOX: roles in tumor suppression and genomic stability.Very large common fragile site genes and their potential role in cancer development.WWOX, large common fragile site genes, and cancer.

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RORA, a large common fragile site gene, is involved in cellular stress response.

http://www.wikidata.org/entity/Q46931410

description

2006 nî lūn-bûn

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2006年の論文

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年學術文章

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2006年學術文章

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name

RORA, a large common fragile site gene, is involved in cellular stress response.

@en

RORA, a large common fragile site gene, is involved in cellular stress response.

@nl

type

label

RORA, a large common fragile site gene, is involved in cellular stress response.

@en

RORA, a large common fragile site gene, is involved in cellular stress response.

@nl

prefLabel

RORA, a large common fragile site gene, is involved in cellular stress response.

@en

RORA, a large common fragile site gene, is involved in cellular stress response.

@nl

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RORA, a large common fragile site gene, is involved in cellular stress response.

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Kuhn R

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McAvoy S

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Smith DI

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Zhu Y

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P2860

Exogenous expression of a dominant negative RORalpha1 vector in muscle cells impairs differentiation: RORalpha1 directly interacts with p300 and myoD

ROR gamma: the third member of ROR/RZR orphan receptor subfamily that is highly expressed in skeletal muscle

Isoform-specific amino-terminal domains dictate DNA-binding properties of ROR alpha, a novel family of orphan hormone nuclear receptors

RORgamma t, a novel isoform of an orphan receptor, negatively regulates Fas ligand expression and IL-2 production in T cells

WWOX, the common chromosomal fragile site, FRA16D, cancer gene

The common fragile site FRA16D and its associated gene WWOX are highly conserved in the mouse at Fra8E1

DNA polymerase alpha inhibition by aphidicolin induces gaps and breaks at common fragile sites in human chromosomes

ATR Regulates Fragile Site Stability

Role of the nuclear hormone receptor ROR gamma in transcriptional regulation, thymocyte survival, and lymphoid organogenesis.

RORalpha1 and RORalpha4 suppress TNF-alpha-induced VCAM-1 and ICAM-1 expression in human endothelial cells.

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