Mechanical ventilation uncouples synthesis and assembly of elastin and increases apoptosis in lungs of newborn mice. Prelude to defective alveolar septation during lung development?
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Early injury of the neonatal lung contributes to premature lung aging: a hypothesisPathogenesis of bronchopulmonary dysplasia: when inflammation meets organ developmentUnderstanding the Impact of Infection, Inflammation, and Their Persistence in the Pathogenesis of Bronchopulmonary DysplasiaThe Extracellular Matrix in Bronchopulmonary Dysplasia: Target and SourceAnimal Models, Learning Lessons to Prevent and Treat Neonatal Chronic Lung DiseaseLoss of Lysyl Oxidase-like 3 Attenuates Embryonic Lung Development in MiceProlonged mechanical ventilation with air induces apoptosis and causes failure of alveolar septation and angiogenesis in lungs of newborn mice.Postnatal inflammation in the pathogenesis of bronchopulmonary dysplasiaLysyl oxidase activity is dysregulated during impaired alveolarization of mouse and human lungs.Chronic lung disease in the preterm infant. Lessons learned from animal modelsbeta6 Integrin subunit deficiency alleviates lung injury in a mouse model of bronchopulmonary dysplasia.Highly conserved transcriptional responses to mechanical ventilation of the lung.Inflammation in fetal sheep from intra-amniotic injection of Ureaplasma parvum.Lung interstitial cells during alveolarization.Animal models of bronchopulmonary dysplasia. The term mouse modelsMechanical ventilation injury and repair in extremely and very preterm lungs.Inhibition of transforming growth factor β worsens elastin degradation in a murine model of Kawasaki diseaseIL-10 inhibits inflammatory cytokines released by fetal mouse lung fibroblasts exposed to mechanical stretch.Bronchopulmonary dysplasia early changes leading to long-term consequences.A subset of epithelial cells with CCSP promoter activity participates in alveolar development.Lung matrix and vascular remodeling in mechanically ventilated elastin haploinsufficient newborn mice.Inhibiting lung elastase activity enables lung growth in mechanically ventilated newborn mice.Aberrant signaling pathways of the lung mesenchyme and their contributions to the pathogenesis of bronchopulmonary dysplasiaGenetic, host, and environmental interactions in a 19 year old with severe chronic obstructive lung disease; observations regarding the pathophysiology of airflow obstructionNeonatal mice genetically modified to express the elastase inhibitor elafin are protected against the adverse effects of mechanical ventilation on lung growthVisualization of neonatal lung injury associated with mechanical ventilation using x-ray dark-field radiography.Role of matrix metalloprotease-9 in hyperoxic injury in developing lung.Absence of TNF-α enhances inflammatory response in the newborn lung undergoing mechanical ventilation.Ventilator-induced lung injury. Similarity and differences between children and adults.Positive end-expiratory pressure and tidal volume during initial ventilation of preterm lambs.The calcitriol analogue EB1089 impairs alveolarization and induces localized regions of increased fibroblast density in neonatal rat lung.Deregulation of the lysyl hydroxylase matrix cross-linking system in experimental and clinical bronchopulmonary dysplasia.Recent advances in late lung development and the pathogenesis of bronchopulmonary dysplasia.Recent advances in the mechanisms of lung alveolarization and the pathogenesis of bronchopulmonary dysplasia.Bronchopulmonary dysplasia - an overview about pathophysiologic concepts.Bridging Lung Development with Chronic Obstructive Pulmonary Disease. Relevance of Developmental Pathways in Chronic Obstructive Pulmonary Disease Pathogenesis.miR-29b supplementation decreases expression of matrix proteins and improves alveolarization in mice exposed to maternal inflammation and neonatal hyperoxia.Conditional deletion of epithelial IKKβ impairs alveolar formation through apoptosis and decreased VEGF expression during early mouse lung morphogenesis.Looking ahead: where to next for animal models of bronchopulmonary dysplasia?Initial Suppression of Transforming Growth Factor-β Signaling and Loss of TGFBI Causes Early Alveolar Structural Defects Resulting in Bronchopulmonary Dysplasia.
P2860
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P2860
Mechanical ventilation uncouples synthesis and assembly of elastin and increases apoptosis in lungs of newborn mice. Prelude to defective alveolar septation during lung development?
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
Mechanical ventilation uncoupl ...... ation during lung development?
@en
Mechanical ventilation uncoupl ...... ation during lung development?
@nl
type
label
Mechanical ventilation uncoupl ...... ation during lung development?
@en
Mechanical ventilation uncoupl ...... ation during lung development?
@nl
prefLabel
Mechanical ventilation uncoupl ...... ation during lung development?
@en
Mechanical ventilation uncoupl ...... ation during lung development?
@nl
P2093
P2860
P1476
Mechanical ventilation uncoupl ...... ation during lung development?
@en
P2093
Anjali Dixit
Berit E Jacobson
Cristina M Alvira
Eric S Shinwell
Lucia M Mokres
Marlene Rabinovitch
Richard D Bland
Robert Ertsey
P2860
P356
10.1152/AJPLUNG.00362.2007
P577
2007-10-12T00:00:00Z