Blockade of TREM-2 exacerbates experimental autoimmune encephalomyelitis.
about
TREM2 Variants in Alzheimer's DiseaseIdentification of soluble TREM-2 in the cerebrospinal fluid and its association with multiple sclerosis and CNS inflammationThe triggering receptor expressed on myeloid cells (TREM) in inflammatory bowel disease pathogenesisMechanisms and potential therapeutic applications of microglial activation after brain injuryBrain innate immunity in the regulation of neuroinflammation: therapeutic strategies by modulating CD200-CD200R interaction involve the cannabinoid systemInflammation after Ischemic Stroke: The Role of Leukocytes and Glial CellsInefficient clearance of myelin debris by microglia impairs remyelinating processesPhagocytosis of microglia in the central nervous system diseasesThe linker for activation of B cells (LAB)/non-T cell activation linker (NTAL) regulates triggering receptor expressed on myeloid cells (TREM)-2 signaling and macrophage inflammatory responses independently of the linker for activation of T cells.Altered microglial response to Aβ plaques in APPPS1-21 mice heterozygous for TREM2.Role of the innate immune system in the pathogenesis of multiple sclerosis.The Pathophysiological Role of Microglia in Dynamic Surveillance, Phagocytosis and Structural Remodeling of the Developing CNS.DAP12 deficiency in liver allografts results in enhanced donor DC migration, augmented effector T cell responses and abrogation of transplant tolerance.Dual induction of TREM2 and tolerance-related transcript, Tmem176b, in amyloid transgenic mice: implications for vaccine-based therapies for Alzheimer's disease.Neurobiology of microglial action in CNS injuries: receptor-mediated signaling mechanisms and functional rolesThe immune response after hypoxia-ischemia in a mouse model of preterm brain injury.Microglia: unique and common features with other tissue macrophages.The role of immune cells, glia and neurons in white and gray matter pathology in multiple sclerosis.Upregulation of TREM2 ameliorates neuropathology and rescues spatial cognitive impairment in a transgenic mouse model of Alzheimer's diseaseIdentification of epigenetically altered genes in sporadic amyotrophic lateral sclerosis.Temporal gene profiling of the 5XFAD transgenic mouse model highlights the importance of microglial activation in Alzheimer's disease.Triggering receptor expressed on myeloid cells-1 (TREM-1): a new player in antiviral immunity?Differential modulation of TREM2 protein during postnatal brain development in mice.The evolving biology of microglia in Alzheimer's disease.Triggering receptor expressed on myeloid cells 2 (TREM2) deficiency attenuates phagocytic activities of microglia and exacerbates ischemic damage in experimental stroke.TREM2 Protein Expression Changes Correlate with Alzheimer's Disease Neurodegenerative Pathologies in Post-Mortem Temporal CorticesThe expanding roles of ITAM adapters FcRgamma and DAP12 in myeloid cells.Reduced BACE1 activity enhances clearance of myelin debris and regeneration of axons in the injured peripheral nervous system.microRNA-34a-Mediated Down-Regulation of the Microglial-Enriched Triggering Receptor and Phagocytosis-Sensor TREM2 in Age-Related Macular DegenerationTREM2 regulates microglial cell activation in response to demyelination in vivo.Microglia-targeted stem cell therapies for Alzheimer disease: A preclinical data review.The triggering receptor expressed on myeloid cells 2: "TREM-ming" the inflammatory component associated with Alzheimer's diseaseImmunomodulation by transplanted human embryonic stem cell-derived oligodendroglial progenitors in experimental autoimmune encephalomyelitis.TREM-2 promotes host resistance against Pseudomonas aeruginosa infection by suppressing corneal inflammation via a PI3K/Akt signaling pathway.Microglia and ischemic stroke: a double-edged sword.Cerebrospinal fluid soluble TREM2 is higher in Alzheimer disease and associated with mutation status.A rose by any other name? The potential consequences of microglial heterogeneity during CNS health and diseaseDebris clearance by microglia: an essential link between degeneration and regeneration.The importance of NAD in multiple sclerosisTREM and TREM-like receptors in inflammation and disease.
P2860
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P2860
Blockade of TREM-2 exacerbates experimental autoimmune encephalomyelitis.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
Blockade of TREM-2 exacerbates experimental autoimmune encephalomyelitis.
@en
Blockade of TREM-2 exacerbates experimental autoimmune encephalomyelitis.
@nl
type
label
Blockade of TREM-2 exacerbates experimental autoimmune encephalomyelitis.
@en
Blockade of TREM-2 exacerbates experimental autoimmune encephalomyelitis.
@nl
prefLabel
Blockade of TREM-2 exacerbates experimental autoimmune encephalomyelitis.
@en
Blockade of TREM-2 exacerbates experimental autoimmune encephalomyelitis.
@nl
P2093
P2860
P50
P356
P1476
Blockade of TREM-2 exacerbates experimental autoimmune encephalomyelitis.
@en
P2093
Cecilia Buonsanti
Margherita Mariani
Susan Gilfillan
P2860
P304
P356
10.1002/EJI.200636837
P407
P577
2007-05-01T00:00:00Z