Smad3 null mice display more rapid wound closure and reduced scar formation after a stab wound to the cerebral cortex.
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Astrocytes as a source for extracellular matrix molecules and cytokinesBlood-brain barrier dysfunction, TGFβ signaling, and astrocyte dysfunction in epilepsyRole of the lesion scar in the response to damage and repair of the central nervous systemInhibiting scar formation in vitro and in vivo by adenovirus-mediated mutant Smad4: a preliminary report.Molecular mechanisms of astrogliosis: new approaches with mouse genetics.TGFβ signaling in the brain increases with aging and signals to astrocytes and innate immune cells in the weeks after stroke.Chondroitin-4-sulfation negatively regulates axonal guidance and growthCandesartan, an angiotensin II AT₁-receptor blocker and PPAR-γ agonist, reduces lesion volume and improves motor and memory function after traumatic brain injury in mice.Brain injury-associated biomarkers of TGF-beta1, S100B, GFAP, NF-L, tTG, AbetaPP, and tau were concomitantly enhanced and the UPS was impaired during acute brain injury caused by Toxocara canis in mice.MicroRNA 146a (miR-146a) is over-expressed during prion disease and modulates the innate immune response and the microglial activation state.Hepatic stellate cells and astrocytes: Stars of scar formation and tissue repair.Smad proteins differentially regulate transforming growth factor-β-mediated induction of chondroitin sulfate proteoglycansOsthole confers neuroprotection against cortical stab wound injury and attenuates secondary brain injuryRoles of chondroitin sulfate and dermatan sulfate in the formation of a lesion scar and axonal regeneration after traumatic injury of the mouse brain.Arctigenin Treatment Protects against Brain Damage through an Anti-Inflammatory and Anti-Apoptotic Mechanism after Needle InsertionThe role of the innate immune system in Alzheimer's disease and frontotemporal lobar degeneration: an eye on microglia.Alterations in sulfated chondroitin glycosaminoglycans following controlled cortical impact injury in mice.Blood-brain barrier breakdown-inducing astrocytic transformation: novel targets for the prevention of epilepsy.Smad3 knockout mice exhibit impaired intestinal mucosal healing.Bridging the Divide between Neuroprosthetic Design, Tissue Engineering and Neurobiology.Reactive astrocytes as therapeutic targets for CNS disorders.The stem cell potential of glia: lessons from reactive gliosis.Vascular damage in the central nervous system: a multifaceted role for vascular-derived TGF-β.Facilitating axon regeneration in the injured CNS by microtubules stabilization.Should losartan be administered following brain injury?"Targeting astrocytes in CNS injury and disease: A translational research approach".Astrocytes: Integrative Regulators of Neuroinflammation in Stroke and Other Neurological Diseases.Culturing layer-specific neocortical neurons as a cell replacement therapy following traumatic brain injuryCD36 is involved in astrocyte activation and astroglial scar formationThe Regulatory Effects of Transforming Growth Factor-β on Nerve Regeneration.Accumulation of a repulsive axonal guidance molecule RGMa in amyloid plaques: a possible hallmark of regenerative failure in Alzheimer's disease brains.Small molecule inhibitor of type I transforming growth factor-β receptor kinase ameliorates the inhibitory milieu in injured brain and promotes regeneration of nigrostriatal dopaminergic axons.The Biology of Regeneration Failure and Success After Spinal Cord Injury.Neuronal and astroglial TGFβ-Smad3 signaling pathways differentially regulate dendrite growth and synaptogenesis.2-carba cyclic phosphatidic acid suppresses inflammation via regulation of microglial polarisation in the stab-wounded mouse cerebral cortex.
P2860
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P2860
Smad3 null mice display more rapid wound closure and reduced scar formation after a stab wound to the cerebral cortex.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh-hant
name
Smad3 null mice display more r ...... wound to the cerebral cortex.
@en
Smad3 null mice display more r ...... wound to the cerebral cortex.
@nl
type
label
Smad3 null mice display more r ...... wound to the cerebral cortex.
@en
Smad3 null mice display more r ...... wound to the cerebral cortex.
@nl
prefLabel
Smad3 null mice display more r ...... wound to the cerebral cortex.
@en
Smad3 null mice display more r ...... wound to the cerebral cortex.
@nl
P2093
P1476
Smad3 null mice display more r ...... wound to the cerebral cortex.
@en
P2093
Helina Moges
Yasmin Bharucha
P304
P356
10.1016/J.EXPNEUROL.2006.08.006
P407
P577
2006-09-22T00:00:00Z