Endogenous Aβ causes cell death via early tau hyperphosphorylation.
about
The Beta-amyloid protein of Alzheimer's disease: communication breakdown by modifying the neuronal cytoskeletonLocalization of axonal motor molecules machinery in neurodegenerative disordersDefects in mitochondrial dynamics and metabolomic signatures of evolving energetic stress in mouse models of familial Alzheimer's diseaseRotenone-dependent changes of anterograde motor protein expression and mitochondrial mobility in brain areas related to neurodegenerative diseasesMitochondrial superoxide contributes to blood flow and axonal transport deficits in the Tg2576 mouse model of Alzheimer's disease.Roles of tau protein in health and disease.Protein aggregation containing β-amyloid, α-synuclein and hyperphosphorylated τ in cultured cells of hippocampus, substantia nigra and locus coeruleus after rotenone exposure.The Intersection of NGF/TrkA Signaling and Amyloid Precursor Protein Processing in Alzheimer's Disease NeuropathologyTau Ser262 phosphorylation is critical for Abeta42-induced tau toxicity in a transgenic Drosophila model of Alzheimer's disease.GSK-3 in Neurodegenerative Diseases.Abnormal tau, mitochondrial dysfunction, impaired axonal transport of mitochondria, and synaptic deprivation in Alzheimer's diseaseIntraneuronal Aβ detection in 5xFAD mice by a new Aβ-specific antibody.Is the mitochondrial outermembrane protein VDAC1 therapeutic target for Alzheimer's disease?Increased tauopathy drives microglia-mediated clearance of beta-amyloid.Nec-1 alleviates cognitive impairment with reduction of Aβ and tau abnormalities in APP/PS1 mice.Tau protein modifications and interactions: their role in function and dysfunction.Multiple faces of dynamin-related protein 1 and its role in Alzheimer's disease pathogenesisImpaired NGF/TrkA Signaling Causes Early AD-Linked Presynaptic Dysfunction in Cholinergic Primary NeuronsNerve growth factor and Alzheimer's disease: new facts for an old hypothesis.MAPping out distribution routes for kinesin couriers.Tau acts as a mediator for Alzheimer's disease-related synaptic deficits.Abolishing Tau cleavage by caspases at Aspartate421 causes memory/synaptic plasticity deficits and pre-pathological Tau alterations.A disease with a sweet tooth: exploring the Warburg effect in Alzheimer's disease.Defective lysosomal proteolysis and axonal transport are early pathogenic events that worsen with age leading to increased APP metabolism and synaptic Abeta in transgenic APP/PS1 hippocampus.Increased tau phosphorylation and cleavage in mouse models of type 1 and type 2 diabetes.α-secretase cleaved amyloid precursor protein (APP) accumulates in cholinergic dystrophic neurites in normal, aged hippocampus.Cholinergic Basal Forebrain Lesion Decreases Neurotrophin Signaling without Affecting Tau Hyperphosphorylation in Genetically Susceptible Mice.Oxidant/Antioxidant Imbalance in Alzheimer's Disease: Therapeutic and Diagnostic Prospects.Phosphorylation of different tau sites during progression of Alzheimer's disease.
P2860
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P2860
Endogenous Aβ causes cell death via early tau hyperphosphorylation.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
Endogenous Aβ causes cell death via early tau hyperphosphorylation.
@en
Endogenous Aβ causes cell death via early tau hyperphosphorylation.
@nl
type
label
Endogenous Aβ causes cell death via early tau hyperphosphorylation.
@en
Endogenous Aβ causes cell death via early tau hyperphosphorylation.
@nl
prefLabel
Endogenous Aβ causes cell death via early tau hyperphosphorylation.
@en
Endogenous Aβ causes cell death via early tau hyperphosphorylation.
@nl
P2093
P1476
Endogenous Aβ causes cell death via early tau hyperphosphorylation
@en
P2093
F Florenzano
M T Ciotti
P Calissano
V Corsetti
P304
P356
10.1016/J.NEUROBIOLAGING.2009.06.005
P577
2009-07-22T00:00:00Z