Wild type but not deltaF508 CFTR inhibits Na+ conductance when coexpressed in Xenopus oocytes.
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Airway surface liquid volume regulation determines different airway phenotypes in liddle compared with betaENaC-overexpressing miceNeutrophil elastase and matrix metalloproteinase 12 in cystic fibrosis lung diseaseToll-like receptors in the host defense against Pseudomonas aeruginosa respiratory infection and cystic fibrosisUp-regulation of acid-gated Na(+) channels (ASICs) by cystic fibrosis transmembrane conductance regulator co-expression in Xenopus oocytesChloride channels: an emerging molecular pictureCl- interference with the epithelial Na+ channel ENaCThe cystic fibrosis transmembrane conductance regulator activates aquaporin 3 in airway epithelial cells.Structure and function of the cystic fibrosis transmembrane conductance regulator.Non-specific activation of the epithelial sodium channel by the CFTR chloride channelThe cystic fibrosis transmembrane conductance regulator (CFTR) inhibits ENaC through an increase in the intracellular Cl- concentrationGating of amiloride-sensitive Na(+) channels: subunit-subunit interactions and inhibition by the cystic fibrosis transmembrane conductance regulator.The cystic fibrosis transmembrane conductance regulator impedes proteolytic stimulation of the epithelial Na+ channel.Effects of cystic fibrosis and congenital bilateral absence of the vas deferens-associated mutations on cystic fibrosis transmembrane conductance regulator-mediated regulation of separate channelsCFTR regulates early pathogenesis of chronic obstructive lung disease in βENaC-overexpressing miceElectrolyte transport in the mammalian colon: mechanisms and implications for disease.Regulation of endogenous ENaC functional expression by CFTR and ΔF508-CFTR in airway epithelial cells.Mucus clearance as a primary innate defense mechanism for mammalian airways.Novel molecular approaches to cystic fibrosis gene therapy.The first-nucleotide binding domain of the cystic-fibrosis transmembrane conductance regulator is important for inhibition of the epithelial Na+ channel.Targeted therapy for cystic fibrosis: cystic fibrosis transmembrane conductance regulator mutation-specific pharmacologic strategies.Liddle's syndrome mutations disrupt cAMP-mediated translocation of the epithelial Na(+) channel to the cell surfaceSubcellular carrier-based optical ion-selective nanosensors.Identification of the cystic fibrosis transmembrane conductance regulator domains that are important for interactions with ROMK2.Lentiviral vectors and cystic fibrosis gene therapyInhibition of protein kinase CK2 closes the CFTR Cl channel, but has no effect on the cystic fibrosis mutant deltaF508-CFTR.The amiloride-inhibitable Na+ conductance is reduced by the cystic fibrosis transmembrane conductance regulator in normal but not in cystic fibrosis airways.Protein kinase CK2, cystic fibrosis transmembrane conductance regulator, and the deltaF508 mutation: F508 deletion disrupts a kinase-binding site.The membrane transporters regulating epithelial NaCl secretion.Organelle redox of CF and CFTR-corrected airway epithelia.CFTR channel in oocytes from Xenopus laevis and its regulation by xShroom1 protein.pH-sensitive capsules as intracellular optical reporters for monitoring lysosomal pH changes upon stimulation.Inhibition of amiloride-sensitive epithelial Na(+) absorption by extracellular nucleotides in human normal and cystic fibrosis airways.Cystic fibrosis transmembrane conductance regulator differentially regulates human and mouse epithelial sodium channels in Xenopus oocytes.Genistein restores functional interactions between Delta F508-CFTR and ENaC in Xenopus oocytes.Genistein improves regulatory interactions between G551D-cystic fibrosis transmembrane conductance regulator and the epithelial sodium channel in Xenopus oocytes.Regulation of the epithelial Na+ channel by cytosolic ATP.Cystic fibrosis transmembrane conductance regulator inverts protein kinase A-mediated regulation of epithelial sodium channel single channel kinetics.Development of chronic bronchitis and emphysema in beta-epithelial Na+ channel-overexpressing mice.Ion and pH sensing with colloidal nanoparticles: influence of surface charge on sensing and colloidal properties.Role of the amiloride-sensitive epithelial Na+ channel in the pathogenesis and as a therapeutic target for cystic fibrosis lung disease.
P2860
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P2860
Wild type but not deltaF508 CFTR inhibits Na+ conductance when coexpressed in Xenopus oocytes.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh-hant
name
Wild type but not deltaF508 CF ...... oexpressed in Xenopus oocytes.
@en
Wild type but not deltaF508 CF ...... oexpressed in Xenopus oocytes.
@nl
type
label
Wild type but not deltaF508 CF ...... oexpressed in Xenopus oocytes.
@en
Wild type but not deltaF508 CF ...... oexpressed in Xenopus oocytes.
@nl
prefLabel
Wild type but not deltaF508 CF ...... oexpressed in Xenopus oocytes.
@en
Wild type but not deltaF508 CF ...... oexpressed in Xenopus oocytes.
@nl
P2093
P2860
P1433
P1476
Wild type but not deltaF508 CF ...... oexpressed in Xenopus oocytes.
@en
P2093
P2860
P356
10.1016/0014-5793(96)00079-8
P407
P577
1996-02-01T00:00:00Z