Cell Type-Specific Contributions of the Angiotensin II Type 1a Receptor to Aorta Homeostasis and Aneurysmal Disease.

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ClC-2 knockdown prevents cerebrovascular remodeling via inhibition of the Wnt/β-catenin signaling pathway.

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Q55455138-F7EE05C0-C33F-4977-8AA7-336E88A68CFD

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Cell Type-Specific Contributions of the Angiotensin II Type 1a Receptor to Aorta Homeostasis and Aneurysmal Disease.

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2018 nî lūn-bûn

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2018年学术文章

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Cell Type-Specific Contributio ...... stasis and Aneurysmal Disease.

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Cell Type-Specific Contributio ...... stasis and Aneurysmal Disease.

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Cell Type-Specific Contributio ...... stasis and Aneurysmal Disease.

@en

Cell Type-Specific Contributio ...... stasis and Aneurysmal Disease.

@nl

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Cell Type-Specific Contributio ...... stasis and Aneurysmal Disease.

@en

Cell Type-Specific Contributio ...... stasis and Aneurysmal Disease.

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Cell Type-Specific Contributio ...... stasis and Aneurysmal Disease.

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Cristina I Caescu

http://www.w3.org/2001/XMLSchema#string

Francesco Ramirez

http://www.w3.org/2001/XMLSchema#string

Irving Miramontes

http://www.w3.org/2001/XMLSchema#string

Jason Cook

http://www.w3.org/2001/XMLSchema#string

Jens Hansen

http://www.w3.org/2001/XMLSchema#string

Josephine Galatioto

http://www.w3.org/2001/XMLSchema#string

Ravi Iyengar

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P2860

Losartan, an AT1 antagonist, prevents aortic aneurysm in a mouse model of Marfan syndrome

Regional variation in aortic AT1b receptor mRNA abundance is associated with contractility but unrelated to atherosclerosis and aortic aneurysms

Endothelial cell-specific deficiency of Ang II type 1a receptors attenuates Ang II-induced ascending aortic aneurysms in LDL receptor-/- mice.

Noncanonical TGFβ signaling contributes to aortic aneurysm progression in Marfan syndrome mice.

Dimorphic effects of transforming growth factor-β signaling during aortic aneurysm progression in mice suggest a combinatorial therapy for Marfan syndrome

Fibroblast Angiotensin II Type 1a Receptors Contribute to Angiotensin II-Induced Medial Hyperplasia in the Ascending Aorta

Conundrum of angiotensin II and TGF-β interactions in aortic aneurysms

Tgfbr2 disruption in postnatal smooth muscle impairs aortic wall homeostasis.

Abnormal muscle mechanosignaling triggers cardiomyopathy in mice with Marfan syndrome.

Aortopathy in a Mouse Model of Marfan Syndrome Is Not Mediated by Altered Transforming Growth Factor β Signaling

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scholarly article

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10.1161/ATVBAHA.117.310609

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English

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2018-01-25T00:00:00Z

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29371244

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P932

5823778

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Cell Type-Specific Contributions of the Angiotensin II Type 1a Receptor to Aorta Homeostasis and Aneurysmal Disease.

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Cell Type-Specific Contributions of the Angiotensin II Type 1a Receptor to Aorta Homeostasis and Aneurysmal Disease.

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P1476

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P577

P698

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