Genetic upregulation of BK channel activity normalizes multiple synaptic and circuit defects in a mouse model of fragile X syndrome.
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BK Channels in the Central Nervous System.MaxiK channel interactome reveals its interaction with GABA transporter 3 and heat shock protein 60 in the mammalian brainPresynaptic BK channels control transmitter release: physiological relevance and potential therapeutic implications.Dynamic balance of excitation and inhibition rapidly modulates spike probability and precision in feed-forward hippocampal circuits.HRPU-2, a homologue of mammalian hnRNP U, regulates synaptic transmission by controlling the expression of SLO-2 potassium channel in C. elegans.Imbalance of synaptic actin dynamics as a key to the Fragile X syndrome?Potential Involvement of Impaired BKCa Channel Function in Sensory Defensiveness and Some Behavioral Disturbances Induced by Unfamiliar Environment in a Mouse Model of Fragile X Syndrome.Fragile X mental retardation protein controls ion channel expression and activity.Of Men and Mice: Modeling the Fragile X Syndrome.Sensory Processing Phenotypes in Fragile X Syndrome
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P2860
Genetic upregulation of BK channel activity normalizes multiple synaptic and circuit defects in a mouse model of fragile X syndrome.
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2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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name
Genetic upregulation of BK cha ...... e model of fragile X syndrome.
@en
Genetic upregulation of BK cha ...... e model of fragile X syndrome.
@nl
type
label
Genetic upregulation of BK cha ...... e model of fragile X syndrome.
@en
Genetic upregulation of BK cha ...... e model of fragile X syndrome.
@nl
prefLabel
Genetic upregulation of BK cha ...... e model of fragile X syndrome.
@en
Genetic upregulation of BK cha ...... e model of fragile X syndrome.
@nl
P2860
P356
P1476
Genetic upregulation of BK cha ...... e model of fragile X syndrome.
@en
P2093
Pan-Yue Deng
Vitaly A Klyachko
P2860
P356
10.1113/JP271031
P407
P577
2015-10-02T00:00:00Z