Modifying the subunit composition of TASK channels alters the modulation of a leak conductance in cerebellar granule neurons.
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Raising cytosolic Cl- in cerebellar granule cells affects their excitability and vestibulo-ocular learning.Pungent agents from Szechuan peppers excite sensory neurons by inhibiting two-pore potassium channelsMouse Models Recapitulating Human Adrenocortical Tumors: What Is Lacking?Potentiation of Gamma Aminobutyric Acid Receptors (GABAAR) by Ethanol: How Are Inhibitory Receptors Affected?Two-pore domain potassium channels enable action potential generation in the absence of voltage-gated potassium channelsTASK channels contribute to neuroprotective action of inhalational anesthetics.Neurotensinergic Excitation of Dentate Gyrus Granule Cells via Gαq-Coupled Inhibition of TASK-3 Channels.The neuroprotective impact of the leak potassium channel TASK1 on stroke development in mice.TASK Channel Deletion Reduces Sensitivity to Local Anesthetic-induced Seizures.TASK1 modulates inflammation and neurodegeneration in autoimmune inflammation of the central nervous system.Motoneuronal TASK channels contribute to immobilizing effects of inhalational general anestheticsSilencing and un-silencing of tetracycline-controlled genes in neuronsActivation of neurotensin receptor 1 facilitates neuronal excitability and spatial learning and memory in the entorhinal cortex: beneficial actions in an Alzheimer's disease modelPacemaker neuron and network oscillations depend on a neuromodulator-regulated linear current.Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9).A new signalling pathway for parallel fibre presynaptic type 4 metabotropic glutamate receptors (mGluR4) in the rat cerebellar cortex.Strain differences in pH-sensitive K+ channel-expressing cells in chemosensory and nonchemosensory brain stem nuclei.Discrete change in volatile anesthetic sensitivity in mice with inactivated tandem pore potassium ion channel TRESK.A new TASK for Dipeptidyl Peptidase-like Protein 6.Targeting two-pore domain K(+) channels TREK-1 and TASK-3 for the treatment of depression: a new therapeutic concept.An unexpected role for TASK-3 potassium channels in network oscillations with implications for sleep mechanisms and anesthetic action.Physiological role of Kv1.3 channel in T lymphocyte cell investigated quantitatively by kinetic modeling.Differential sensitivity of TREK-1, TREK-2 and TRAAK background potassium channels to the polycationic dye ruthenium red.Two-pore domain potassium channels in the adrenal cortex.Avertin®, but Not Volatile Anesthetics Addressing the Two-Pore Domain K+ Channel, TASK-1, Slows Down Cilia-Driven Particle Transport in the Mouse Trachea.TASK-1 potassium channel is not critically involved in mediating hypoxic pulmonary vasoconstriction of murine intra-pulmonary arteries.Invalidation of TASK1 potassium channels disrupts adrenal gland zonation and mineralocorticoid homeostasisMouse models of adrenocortical tumors.Membrane potential-regulated Ca2+ signalling in development and maturation of mammalian cerebellar granule cells.Neuronal two-pore-domain potassium channels and their regulation by G protein-coupled receptors.Maternally inherited Birk Barel mental retardation dysmorphism syndrome caused by a mutation in the genomically imprinted potassium channel KCNK9GABA(A) receptors: structure and function in the basal gangliaGABA(B) receptor activation inhibits neuronal excitability and spatial learning in the entorhinal cortex by activating TREK-2 K+ channels.Developmental expression of a functional TASK-1 2P domain K+ channel in embryonic chick heartModulatory mechanisms and multiple functions of somatodendritic A-type K (+) channel auxiliary subunitsGating of two pore domain potassium channels.Molecular physiology of pH-sensitive background K(2P) channels.The role of K₂p channels in anaesthesia and sleep.The CNS under pathophysiologic attack--examining the role of K₂p channels.The family of K2P channels: salient structural and functional properties.
P2860
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P2860
Modifying the subunit composition of TASK channels alters the modulation of a leak conductance in cerebellar granule neurons.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Modifying the subunit composit ...... in cerebellar granule neurons.
@en
Modifying the subunit composit ...... in cerebellar granule neurons.
@nl
type
label
Modifying the subunit composit ...... in cerebellar granule neurons.
@en
Modifying the subunit composit ...... in cerebellar granule neurons.
@nl
prefLabel
Modifying the subunit composit ...... in cerebellar granule neurons.
@en
Modifying the subunit composit ...... in cerebellar granule neurons.
@nl
P50
P1476
Modifying the subunit composit ...... in cerebellar granule neurons
@en
P2093
Cristina Sandu
Louisa J Evans
P304
11455-11467
P356
10.1523/JNEUROSCI.3153-05.2005
P407
P50
P577
2005-12-01T00:00:00Z