ERK1 is dispensable for mouse pancreatic beta cell function but is necessary for glucose-induced full activation of MSK1 and CREB.

about

ERK1 is dispensable for mouse pancreatic beta cell function but is necessary for glucose-induced full activation of MSK1 and CREB.

Item

http://www.wikidata.org/entity/Q50882981

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

ERK1 is dispensable for mouse ...... l activation of MSK1 and CREB.

@en

ERK1 is dispensable for mouse ...... l activation of MSK1 and CREB.

@nl

type

Item

label

ERK1 is dispensable for mouse ...... l activation of MSK1 and CREB.

@en

ERK1 is dispensable for mouse ...... l activation of MSK1 and CREB.

@nl

prefLabel

ERK1 is dispensable for mouse ...... l activation of MSK1 and CREB.

@en

ERK1 is dispensable for mouse ...... l activation of MSK1 and CREB.

@nl

P1476

ERK1 is dispensable for mouse ...... l activation of MSK1 and CREB.

@en

P2093

Annie Varrault

http://www.w3.org/2001/XMLSchema#string

Dany Muller

http://www.w3.org/2001/XMLSchema#string

Gilles Pagès

http://www.w3.org/2001/XMLSchema#string

Gyslaine Bertrand

http://www.w3.org/2001/XMLSchema#string

Jacques Pouyssegur

http://www.w3.org/2001/XMLSchema#string

Joy Richard

http://www.w3.org/2001/XMLSchema#string

Julia Mathieu

http://www.w3.org/2001/XMLSchema#string

Michele Leduc

http://www.w3.org/2001/XMLSchema#string

P2860

ERKs: a family of protein-serine/threonine kinases that are activated and tyrosine phosphorylated in response to insulin and NGF

Activation and function of the MAPKs and their substrates, the MAPK-activated protein kinases

The selectivity of protein kinase inhibitors: a further update

Defective thymocyte maturation in p44 MAP kinase (Erk 1) knockout mice

MSK1 is required for CREB phosphorylation in response to mitogens in mouse embryonic stem cells

Imaging a target of Ca2+ signalling: dense core granule exocytosis viewed by total internal reflection fluorescence microscopy.

Molecular analysis of db gene-related pancreatic beta cell dysfunction; evidence for a compensatory mechanism inhibiting development of diabetes in the db gene heterozygote.

Protein farnesylation-dependent Raf/extracellular signal-related kinase signaling links to cytoskeletal remodeling to facilitate glucose-induced insulin secretion in pancreatic beta-cells.

The NOD mouse model of type 1 diabetes: as good as it gets?

The transcriptional landscape of mouse beta cells compared to human beta cells reveals notable species differences in long non-coding RNA and protein-coding gene expression.

P2888

s00125-017-4356-6

P304

1999-2010

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1007/S00125-017-4356-6

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

P577

2017-07-18T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28721437

http://www.w3.org/2001/XMLSchema#string