Growth Inhibition by Testosterone in an Androgen Receptor Splice Variant-Driven Prostate Cancer Model.

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Glycogen synthase kinase-3 inhibitors suppress the AR-V7-mediated transcription and selectively inhibit cell growth in AR-V7-positive prostate cancer cells.Supraphysiologic Testosterone Therapy in the Treatment of Prostate Cancer: Models, Mechanisms and Questions.

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Growth Inhibition by Testosterone in an Androgen Receptor Splice Variant-Driven Prostate Cancer Model.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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Growth Inhibition by Testoster ...... -Driven Prostate Cancer Model.

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Growth Inhibition by Testoster ...... -Driven Prostate Cancer Model.

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Growth Inhibition by Testoster ...... -Driven Prostate Cancer Model.

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Growth Inhibition by Testoster ...... -Driven Prostate Cancer Model.

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Growth Inhibition by Testoster ...... -Driven Prostate Cancer Model.

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Growth Inhibition by Testoster ...... -Driven Prostate Cancer Model.

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P1476

Growth Inhibition by Testoster ...... t-Driven Prostate Cancer Model

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P2093

Akira Tanaka

http://www.w3.org/2001/XMLSchema#string

Kazuhide Nakayama

http://www.w3.org/2001/XMLSchema#string

Masami Kusaka

http://www.w3.org/2001/XMLSchema#string

Tatsuya Watanabe

http://www.w3.org/2001/XMLSchema#string

Tsuneo Masaki

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P2860

Expression of androgen receptor splice variants in prostate cancer bone metastases is associated with castration-resistance and short survival

Androgen receptor variants occur frequently in castration resistant prostate cancer metastases

Human prostate tumor growth in athymic mice: inhibition by androgens and stimulation by finasteride

Structure-activity relationship for thiohydantoin androgen receptor antagonists for castration-resistant prostate cancer (CRPC)

Recurrent fusion of TMPRSS2 and ETS transcription factor genes in prostate cancer.

AR-V7 and resistance to enzalutamide and abiraterone in prostate cancer

A novel androgen receptor splice variant is up-regulated during prostate cancer progression and promotes androgen depletion-resistant growth

The development of androgen-independent prostate cancer

The RNA-binding protein Sam68 regulates expression and transcription function of the androgen receptor splice variant AR-V7

Ligand-independent androgen receptor variants derived from splicing of cryptic exons signify hormone-refractory prostate cancer

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1536-1545

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scholarly article

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10.1002/PROS.23238

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P433

http://www.w3.org/2001/XMLSchema#string

P478

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Daisuke Nakata

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2016-07-30T00:00:00Z

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27473672

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Growth Inhibition by Testosterone in an Androgen Receptor Splice Variant-Driven Prostate Cancer Model.

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P2860

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Growth Inhibition by Testosterone in an Androgen Receptor Splice Variant-Driven Prostate Cancer Model.

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P2860

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