Three-dimensional reconstruction of fracture callus morphogenesis. - Wikidata - wikimedia - TriplyDB

Three-dimensional reconstruction of fracture callus morphogenesis.

Three-dimensional reconstruction of fracture callus morphogenesis.

http://www.wikidata.org/entity/Q52011688

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Overview of biological mechanisms and applications of three murine models of bone repair: closed fracture with intramedullary fixation, distraction osteogenesis, and marrow ablation by reaming A biomechanical, micro-computertomographic and histological analysis of the influence of diclofenac and prednisolone on fracture healing in vivo.Mechanical stimulation alters tissue differentiation and molecular expression during bone healing Lysyl oxidase-like-2 (LOXL2) is a major isoform in chondrocytes and is critically required for differentiation Micro-computed tomography assessment of fracture healing: relationships among callus structure, composition, and mechanical function.Fully automated segmentation of callus by micro-CT compared to biomechanics.Three-dimensional reconstructions of intrahepatic bile duct tubulogenesis in human liver Decreased autocrine EGFR signaling in metastatic breast cancer cells inhibits tumor growth in bone and mammary fat pad Recent advances in the use of serological bone formation markers to monitor callus development and fracture healing Role of Fas and Treg cells in fracture healing as characterized in the fas-deficient (lpr) mouse model of lupus.The biology of fracture healing.Molecular mechanisms controlling bone formation during fracture healing and distraction osteogenesis Diabetes reduces mesenchymal stem cells in fracture healing through a TNFα-mediated mechanism Urine matrix metalloproteinases (MMPs) as biomarkers for the progression of fracture healing Muscle-bone interactions during fracture healing.Generation of closed transverse fractures in small animals.MC3T3-E1 osteoprogenitor cells systemically migrate to a bone defect and enhance bone healing Deferoxamine restores callus size, mineralization, and mechanical strength in fracture healing after radiotherapy.Vascular tissues are a primary source of BMP2 expression during bone formation induced by distraction osteogenesis Genetic variation in the patterns of skeletal progenitor cell differentiation and progression during endochondral bone formation affects the rate of fracture healing.Bone formation during distraction osteogenesis is dependent on both VEGFR1 and VEGFR2 signaling.Fibroblast growth factor expression during skeletal fracture healing in mice Diabetes causes the accelerated loss of cartilage during fracture repair which is reversed by insulin treatment.High levels of tumor necrosis factor-alpha contribute to accelerated loss of cartilage in diabetic fracture healing.Effect of perioperative buccal fracture of the proximal segment on postoperative stability after sagittal split ramus osteotomy.Modulation of fixation stiffness from flexible to stiff in a rat model of bone healing.Bone fracture and bone fracture repair.Nanofiber-based delivery of bioactive agents and stem cells to bone sites.Drugs for bone healing.Initiation and early control of tissue regeneration - bone healing as a model system for tissue regeneration.Overview of skeletal repair (fracture healing and its assessment).The transcriptome of fracture healing defines mechanisms of coordination of skeletal and vascular development during endochondral bone formation.Does cefuroxime alter fracture healing in vivo? A micro-computertomographic, biomechanical, and histomorphometric evaluation using a rat fracture model.Bone fracture healing in mechanobiological modeling: A review of principles and methods.Assessment of a mechano-regulation theory of skeletal tissue differentiation in an in vivo model of mechanically induced cartilage formation.Mechanical microenvironments and protein expression associated with formation of different skeletal tissues during bone healing.Chemokine expression is upregulated in chondrocytes in diabetic fracture healing.Mouse models of bone healing: fracture, marrow ablation, and distraction osteogenesis.MRT letter: Contrast-enhanced computed tomographic imaging of soft callus formation in fracture healing.TNF-alpha mediates diabetes-enhanced chondrocyte apoptosis during fracture healing and stimulates chondrocyte apoptosis through FOXO1.

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Three-dimensional reconstruction of fracture callus morphogenesis.

http://www.wikidata.org/entity/Q52011688

description

2006 nî lūn-bûn

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2006年の論文

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年學術文章

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2006年學術文章

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name

Three-dimensional reconstruction of fracture callus morphogenesis.

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Three-dimensional reconstruction of fracture callus morphogenesis.

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type

label

Three-dimensional reconstruction of fracture callus morphogenesis.

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Three-dimensional reconstruction of fracture callus morphogenesis.

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prefLabel

Three-dimensional reconstruction of fracture callus morphogenesis.

@en

Three-dimensional reconstruction of fracture callus morphogenesis.

@nl

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JHC.6A6959.2006

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Journal of Histochemistry & Cytochemistry

P1476

Three-dimensional reconstruction of fracture callus morphogenesis

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P2093

Dana T Graves

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Elizabeth A Krall

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Fred H Nicholls

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Jennifer L Fitch

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Karl J Jepsen

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Megan Bauer

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Rayyan Kayal

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Stephanie N Stapleton

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P2860

Bone histomorphometry: standardization of nomenclature, symbols, and units. Report of the ASBMR Histomorphometry Nomenclature Committee

Tumor necrosis factor alpha (TNF-alpha) coordinately regulates the expression of specific matrix metalloproteinases (MMPS) and angiogenic factors during fracture healing.

Vascular endothelial growth factor stimulates bone repair by promoting angiogenesis and bone turnover.

Angiogenesis and bone growth.

Cyclooxygenase-2 regulates mesenchymal cell differentiation into the osteoblast lineage and is critically involved in bone repair.

Fracture healing as a post-natal developmental process: molecular, spatial, and temporal aspects of its regulation.

Application of histomorphometric methods to the study of bone repair.

Musculoskeletal ontogeny, phylogeny, and functional adaptation.

Differential inhibition of fracture healing by non-selective and cyclooxygenase-2 selective non-steroidal anti-inflammatory drugs.

Normal and pathological remodeling of human trabecular bone: three dimensional reconstruction of the remodeling sequence in normals and in metabolic bone disease.

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1215-1228

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10.1369/JHC.6A6959.2006

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11

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54

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Thomas A Einhorn

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2006-07-24T00:00:00Z

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16864894

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