Non-enzymatically glycated tau in Alzheimer's disease induces neuronal oxidant stress resulting in cytokine gene expression and release of amyloid beta-peptide.
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Oxidative Stress Signaling in Alzheimers DiseaseRAGE-mediated signaling contributes to intraneuronal transport of amyloid-beta and neuronal dysfunctionThe Rationale for Insulin Therapy in Alzheimer's DiseaseAdvanced Glycation End Products: Link between Diet and Ovulatory Dysfunction in PCOS?Neuronal failure in Alzheimer's disease: a view through the oxidative stress looking-glassOxidized cholesterol as the driving force behind the development of Alzheimer's diseaseNeuronal Gene Targets of NF-κB and Their Dysregulation in Alzheimer's DiseaseInflammation and Alzheimer's diseaseA multimodal RAGE-specific inhibitor reduces amyloid β-mediated brain disorder in a mouse model of Alzheimer diseaseElectromagnetic fields, oxidative stress, and neurodegenerationAGEs induce cell death via oxidative and endoplasmic reticulum stresses in both human SH-SY5Y neuroblastoma cells and rat cortical neuronsMicroglial receptor for advanced glycation end product-dependent signal pathway drives beta-amyloid-induced synaptic depression and long-term depression impairment in entorhinal cortex.The sour side of neurodegenerative disorders: the effects of protein glycation.Precursor of advanced glycation end products mediates ER-stress-induced caspase-3 activation of human dermal fibroblasts through NAD(P)H oxidase 4Intraneuronal amyloid beta accumulation and oxidative damage to nucleic acids in Alzheimer disease.Increased iron and free radical generation in preclinical Alzheimer disease and mild cognitive impairment.RAGE inhibition in microglia prevents ischemia-dependent synaptic dysfunction in an amyloid-enriched environmentPathological correlations between traumatic brain injury and chronic neurodegenerative diseases.Microglia and neurodegeneration.Staging of Alzheimer's pathology in triple transgenic mice: a light and electron microscopic analysis.Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.Pathogenesis of beta(2)-microglobulin amyloidosis: role of monocytes/macrophages.Triple negative tumors accumulate significantly less methylglyoxal specific adducts than other human breast cancer subtypesHeme deficiency may be a factor in the mitochondrial and neuronal decay of aging.Pinocembrin protects against β-amyloid-induced toxicity in neurons through inhibiting receptor for advanced glycation end products (RAGE)-independent signaling pathways and regulating mitochondrion-mediated apoptosis.Oxidative damage to RNA: mechanisms, consequences, and diseases.Adiponectin is protective against oxidative stress induced cytotoxicity in amyloid-beta neurotoxicityMacrophage Migration Inhibitory Factor is subjected to glucose modification and oxidation in Alzheimer's Disease.Central role of RAGE-dependent neointimal expansion in arterial restenosis.Oxidative stress-mediated activation of extracellular signal-regulated kinase contributes to mild cognitive impairment-related mitochondrial dysfunctionOxidative damage and cognitive dysfunction: antioxidant treatments to promote healthy brain agingNeurovascular defects and faulty amyloid-β vascular clearance in Alzheimer's diseaseInhibition of NF-kappaB potentiates amyloid beta-mediated neuronal apoptosis.Advanced glycation end products in Alzheimer's disease and other neurodegenerative diseases.Evidence of oxidative stress and in vivo neurotoxicity of beta-amyloid in a transgenic mouse model of Alzheimer's disease: a chronic oxidative paradigm for testing antioxidant therapies in vivo.Evidence of neuronal oxidative damage in Alzheimer's disease.Human Microbiome and its Association With Health and Diseases.Staying connected: synapses in Alzheimer disease.Glyceraldehyde caused Alzheimer's disease-like alterations in diagnostic marker levels in SH-SY5Y human neuroblastoma cellsPathological significance of mitochondrial glycation
P2860
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P2860
Non-enzymatically glycated tau in Alzheimer's disease induces neuronal oxidant stress resulting in cytokine gene expression and release of amyloid beta-peptide.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh-hant
name
Non-enzymatically glycated tau ...... lease of amyloid beta-peptide.
@en
Non-enzymatically glycated tau ...... lease of amyloid beta-peptide.
@nl
type
label
Non-enzymatically glycated tau ...... lease of amyloid beta-peptide.
@en
Non-enzymatically glycated tau ...... lease of amyloid beta-peptide.
@nl
prefLabel
Non-enzymatically glycated tau ...... lease of amyloid beta-peptide.
@en
Non-enzymatically glycated tau ...... lease of amyloid beta-peptide.
@nl
P2093
P2860
P356
P1433
P1476
Non-enzymatically glycated tau ...... lease of amyloid beta-peptide.
@en
P2093
Kuppusamy P
P2860
P2888
P304
P356
10.1038/NM0795-693
P407
P50
P577
1995-07-01T00:00:00Z