Evidence of key role of Cdk2 overexpression in pemphigus vulgaris. - Wikidata - wikimedia - TriplyDB

Evidence of key role of Cdk2 overexpression in pemphigus vulgaris.

Evidence of key role of Cdk2 overexpression in pemphigus vulgaris.

http://www.wikidata.org/entity/Q53504783

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Histological and ultrastructural abnormalities in murine desmoglein 2-mutant hearts Pemphigus vulgaris IgG cause loss of desmoglein-mediated adhesion and keratinocyte dissociation independent of epidermal growth factor receptor Peptides Targeting the Desmoglein 3 Adhesive Interface Prevent Autoantibody-induced Acantholysis in Pemphigus.Reviewing putative industrial triggering in pemphigus: cluster of pemphigus in the area near the wastewater treatment plant.Keratinocytes synthesize and activate cortisol.Merging experimental data and in silico analysis: a systems-level approach to autoimmune disease and cancer.Desmoglein 2 mutant mice develop cardiac fibrosis and dilation.Cell-cell adhesions and cell contractility are upregulated upon desmosome disruption The extent of desmoglein 3 depletion in pemphigus vulgaris is dependent on Ca(2+)-induced differentiation: a role in suprabasal epidermal skin splitting?The desmosome and pemphigus.Advances in pemphigus and its endemic pemphigus foliaceus (Fogo Selvagem) phenotype: a paradigm of human autoimmunity.Involvement of the apoptotic mechanism in pemphigus foliaceus autoimmune injury of the skin.Apoptotic pathways in pemphigus.Pemphigus autoimmunity: hypotheses and realities.Urban legends: pemphigus vulgaris.Desmosomal adhesion and pemphigus vulgaris: the first half of the story.Desmosomal cadherins and signaling: lessons from autoimmune disease.150(th) anniversary series: Desmosomes and autoimmune disease, perspective of dynamic desmosome remodeling and its impairments in pemphigus.Characterisation of the cancer-associated glucocorticoid system: key role of 11β-hydroxysteroid dehydrogenase type 2.Genome-wide expression analysis suggests unique disease-promoting and disease-preventing signatures in Pemphigus vulgaris.Deregulation of PERK in the autoimmune disease pemphigus vulgaris occurs via IgG-independent mechanisms.Actin reorganization contributes to loss of cell adhesion in pemphigus vulgaris.Crosstalk between Signaling Pathways in Pemphigus: A Role for Endoplasmic Reticulum Stress in p38 Mitogen-Activated Protein Kinase Activation?Molecular insights into the effects of sodium hyaluronate preparations in keratinocytes.The Evolving Story of Autoantibodies in Pemphigus Vulgaris: Development of the "Super Compensation Hypothesis"

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P2860

Evidence of key role of Cdk2 overexpression in pemphigus vulgaris.

http://www.wikidata.org/entity/Q53504783

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

@zh

2008年學術文章

@zh-hant

name

Evidence of key role of Cdk2 overexpression in pemphigus vulgaris.

@en

Evidence of key role of Cdk2 overexpression in pemphigus vulgaris.

@nl

type

label

Evidence of key role of Cdk2 overexpression in pemphigus vulgaris.

@en

Evidence of key role of Cdk2 overexpression in pemphigus vulgaris.

@nl

prefLabel

Evidence of key role of Cdk2 overexpression in pemphigus vulgaris.

@en

Evidence of key role of Cdk2 overexpression in pemphigus vulgaris.

@nl

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Journal of Biological Chemistry

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Evidence of key role of Cdk2 overexpression in pemphigus vulgaris

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Amelia Casamassimi

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Fernando Gombos

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Luigi Rossiello

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Nicola Cirillo

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Raffaele Rossiello

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P2860

Novel human alpha9 acetylcholine receptor regulating keratinocyte adhesion is targeted by Pemphigus vulgaris autoimmunity

DAVID: Database for Annotation, Visualization, and Integrated Discovery

Structure and function of desmosomal proteins and their role in development and disease

'Hard' and 'soft' principles defining the structure, function and regulation of keratin intermediate filaments

Cell cycle sibling rivalry: Cdc2 vs. Cdk2

Autoantibodies against a novel epithelial cadherin in pemphigus vulgaris, a disease of cell adhesion

Normal keratinization in a spontaneously immortalized aneuploid human keratinocyte cell line

Explanations for the clinical and microscopic localization of lesions in pemphigus foliaceus and vulgaris.

Anti-epidermal-cell-surface pemphigus antibody detaches viable epidermal cells from culture plates by activation of proteinase.

Targeting c-Myc, Ras and IGF cascade to treat cancer and vascular disorders.

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8736-8745

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scholarly article

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10.1074/JBC.M702186200

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13

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283

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Alessandro Lanza

Concetta Schiano

Filomena de Nigris

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2008-01-16T00:00:00Z

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5651286

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18199752

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