about
JAK2 activation by growth hormone and other cytokinesThe growth hormone receptor: mechanism of activation and clinical implicationsThe extracellular domain of the growth hormone receptor interacts with coactivator activator to promote cell proliferationCharacterisation of inter- and intra-molecular interactions of the dengue virus RNA dependent RNA polymerase as potential drug targets.Mechanism of activation of protein kinase JAK2 by the growth hormone receptor.Nuclear targeting of the growth hormone receptor results in dysregulation of cell proliferation and tumorigenesis.Growth hormone receptor; mechanism of action.Growth hormone and cell growth.A new mechanism for growth hormone receptor activation of JAK2, and implications for related cytokine receptors.Janus kinases and Src family kinases in the regulation of EGF-induced vimentin expression in MDA-MB-468 breast cancer cells.STAT5 Activation in the Dermal Papilla Is Important for Hair Follicle Growth Phase Induction.Intrinsically disordered cytoplasmic domains of two cytokine receptors mediate conserved interactions with membranes.Tmem2 Regulates Embryonic Vegf Signaling by Controlling Hyaluronic Acid Turnover.Epstein-Barr virus EBNA-3C is targeted to and regulates expression from the bidirectional LMP-1/2B promoter.The interdomain region of dengue NS5 protein that binds to the viral helicase NS3 contains independently functional importin beta 1 and importin alpha/beta-recognized nuclear localization signals.Exploiting hydrophobicity for efficient production of transmembrane helices for structure determination by NMR spectroscopy.The 37-amino-acid interdomain of dengue virus NS5 protein contains a functional NLS and inhibitory CK2 site.Uropathogenic Escherichia coli Engages CD14-Dependent Signaling to Enable Bladder-Macrophage-Dependent Control of Acute Urinary Tract Infection.Rewriting the mechanism of JAK2 activation.An agonist-induced conformational change in the growth hormone receptor determines the choice of signalling pathway.A growth hormone receptor SNP promotes lung cancer by impairment of SOCS2-mediated degradation.Caveolin-1 is necessary for hepatic oxidative lipid metabolism: evidence for crosstalk between caveolin-1 and bile acid signaling.Innate transcriptional networks activated in bladder in response to uropathogenic Escherichia coli drive diverse biological pathways and rapid synthesis of IL-10 for defense against bacterial urinary tract infection.The Growth Hormone Receptor: Mechanism of Receptor Activation, Cell Signaling, and Physiological Aspects.Growth hormone receptor: structure function relationships.Role of the growth hormone–IGF-1 axis in cancerNovel drivers and modifiers of MPL-dependent oncogenic transformation identified by deep mutational scanningOncogenic basic amino acid insertions at the extracellular juxtamembrane region of IL7RA cause receptor hypersensitivity
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Andrew J. Brooks
@ast
Andrew J. Brooks
@en
Andrew J. Brooks
@es
Andrew J. Brooks
@nl
Andrew J. Brooks
@sl
type
label
Andrew J. Brooks
@ast
Andrew J. Brooks
@en
Andrew J. Brooks
@es
Andrew J. Brooks
@nl
Andrew J. Brooks
@sl
prefLabel
Andrew J. Brooks
@ast
Andrew J. Brooks
@en
Andrew J. Brooks
@es
Andrew J. Brooks
@nl
Andrew J. Brooks
@sl
P1053
A-9969-2011
P106
P1153
36440975300
P21
P2798
P31
P3829
P496
0000-0002-2691-1668