SOX2 silencing in glioblastoma tumor-initiating cells causes stop of proliferation and loss of tumorigenicity. - Wikidata - wikimedia - TriplyDB

SOX2 silencing in glioblastoma tumor-initiating cells causes stop of proliferation and loss of tumorigenicity.

SOX2 silencing in glioblastoma tumor-initiating cells causes stop of proliferation and loss of tumorigenicity.

http://www.wikidata.org/entity/Q55465654

about

Identification of CBX3 and ABCA5 as putative biomarkers for tumor stem cells in osteosarcoma SOX2 is an oncogene activated by recurrent 3q26.3 amplifications in human lung squamous cell carcinomas Yin Yang 1 is associated with cancer stem cell transcription factors (SOX2, OCT4, BMI1) and clinical implication Paradoxical role of SOX2 in gastric cancer Cancer Stem Cell Hierarchy in Glioblastoma Multiforme Glioma Stem Cells and Their Microenvironments: Providers of Challenging Therapeutic Targets Targeting Aggressive Cancer Stem Cells in Glioblastoma The role of SOX2 in small cell lung cancer, lung adenocarcinoma and squamous cell carcinoma of the lung Glioblastoma Circulating Cells: Reality, Trap or Illusion?Pluripotency Genes and Their Functions in the Normal and Aberrant Breast and Brain Role of SOX family of transcription factors in central nervous system tumors Brain tumor stem cells: Molecular characteristics and their impact on therapy Targeting SOX2 as a Therapeutic Strategy in Glioblastoma Accelerating glioblastoma drug discovery: Convergence of patient-derived models, genome editing and phenotypic screening Concise Review: NANOG in Cancer Stem Cells and Tumor Development: An Update and Outstanding Questions CD44posCD49fhiCD133/2hi defines xenograft-initiating cells in estrogen receptor-negative breast cancer GSK3beta regulates differentiation and growth arrest in glioblastoma β-Catenin signaling initiates the activation of astrocytes and its dysregulation contributes to the pathogenesis of astrocytomas.PPARγ agonists regulate the expression of stemness and differentiation genes in brain tumour stem cells Functional analysis of HOXD9 in human gliomas and glioma cancer stem cells NANOG promotes cancer stem cell characteristics and prostate cancer resistance to androgen deprivation.A cell-penetrating peptide based on the interaction between c-Src and connexin43 reverses glioma stem cell phenotype.Elevated FOXG1 and SOX2 in glioblastoma enforces neural stem cell identity through transcriptional control of cell cycle and epigenetic regulators.Stem Cell Factor-Based Identification and Functional Properties of In Vitro-Selected Subpopulations of Malignant Mesothelioma Cells.Expression of the embryonic stem cell transcription factor SOX2 in human skin: relevance to melanocyte and merkel cell biology Molecular culprits generating brain tumor stem cells NOS2 expression in glioma cell lines and glioma primary cell cultures: correlation with neurosphere generation and SOX-2 expression.The Transcription Regulator Krüppel-Like Factor 4 and Its Dual Roles of Oncogene in Glioblastoma and Tumor Suppressor in Neuroblastoma.The hypoxic microenvironment maintains glioblastoma stem cells and promotes reprogramming towards a cancer stem cell phenotype.Inactivation of p38 MAPK contributes to stem cell-like properties of non-small cell lung cancer Primary brain tumors, neural stem cell, and brain tumor cancer cells: where is the link?HMMR maintains the stemness and tumorigenicity of glioblastoma stem-like cells.Cancer stem cell-specific scavenger receptor CD36 drives glioblastoma progression.The SOX2 response program in glioblastoma multiforme: an integrated ChIP-seq, expression microarray, and microRNA analysis Small molecules targeting histone demethylase genes (KDMs) inhibit growth of temozolomide-resistant glioblastoma cells Activation of neural and pluripotent stem cell signatures correlates with increased malignancy in human glioma Sox2 promotes malignancy in glioblastoma by regulating plasticity and astrocytic differentiation Activation of SOX2 expression by BRD4-NUT oncogenic fusion drives neoplastic transformation in NUT midline carcinoma.Characterization of cancer stem-like cells derived from mouse induced pluripotent stem cells transformed by tumor-derived extracellular vesicles.EphA2 promotes infiltrative invasion of glioma stem cells in vivo through cross-talk with Akt and regulates stem cell properties.

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P2860

SOX2 silencing in glioblastoma tumor-initiating cells causes stop of proliferation and loss of tumorigenicity.

http://www.wikidata.org/entity/Q55465654

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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2009年學術文章

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name

SOX2 silencing in glioblastoma ...... on and loss of tumorigenicity.

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SOX2 silencing in glioblastoma ...... on and loss of tumorigenicity.

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type

label

SOX2 silencing in glioblastoma ...... on and loss of tumorigenicity.

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SOX2 silencing in glioblastoma ...... on and loss of tumorigenicity.

@nl

prefLabel

SOX2 silencing in glioblastoma ...... on and loss of tumorigenicity.

@en

SOX2 silencing in glioblastoma ...... on and loss of tumorigenicity.

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STEMCELLS.2008-0493

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SOX2 silencing in glioblastoma ...... on and loss of tumorigenicity.

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Daniela Marubbi

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Fabrizio Griffero

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Gian Luigi Ravetti

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Gian Luigi Zona

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Giorgio Corte

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Maria Cristina Capra

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Marzia Perera

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Paolo Malatesta

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Rosaria Maria Rita Gangemi

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40-48

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scholarly article

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10.1634/STEMCELLS.2008-0493

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1

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18948646

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