CD28 interactions with either CD80 or CD86 are sufficient to induce allergic airway inflammation in mice
about
The landscape of human genes involved in the immune response to parasitic wormsAn enigmatic tail of CD28 signaling.The expanding B7 superfamily: increasing complexity in costimulatory signals regulating T cell function.Role of dendritic cells and Th2 lymphocytes in asthma: lessons from eosinophilic airway inflammation in the mouse.Role of T cell co-stimulation in murine models of allergic asthma.Dendritic cells in asthma: a function beyond sensitization.Co-stimulatory molecules as potential targets for therapeutic intervention in allergic airway disease.In vivo depletion of lung CD11c+ dendritic cells during allergen challenge abrogates the characteristic features of asthmaThe costimulatory molecule SLAM is critical for pulmonary allergic responses.CD28 and ICOS play complementary non-overlapping roles in the development of Th2 immunity in vivo.Galiximab: a review.Immunotherapy for allergies and asthma: present and futureGenetic regulation of immunoglobulin E level in different pathological states: integration of mouse and human genetics.Blockade of CD28/B7-2 costimulation inhibits experimental obliterative bronchiolitis in rat tracheal allografts: suppression of helper T cell type1-dominated immune response.CD28-mediated T cell response is upregulated by exogenous application of autologous Hsp70-peptide complex in a tumor-bearing host.Langerhans' cells in vernal keratoconjunctivitis express the costimulatory molecule B7-2 (CD86), but not B7-1 (CD80).Myeloid dendritic cells induce Th2 responses to inhaled antigen, leading to eosinophilic airway inflammation.CD28/CTLA4 double deficient mice demonstrate crucial role for B7 co-stimulation in the induction of allergic lower airways disease.The p38 mitogen-activated protein kinase regulates effector functions of primary human CD4 T cells.The costimulatory molecules CD80, CD86 and OX40L are up-regulated in Aspergillus fumigatus sensitized mice.Association and interaction analyses of eight genes under asthma linkage peaksIncreased expression of plasma and cell surface co-stimulatory molecules CTLA-4, CD28 and CD86 in adult patients with allergic asthmaBlockade of CTLA-4 enhances allergic sensitization and eosinophilic airway inflammation in genetically predisposed miceCD28 and CTLA4 coordinately regulate airway inflammatory cell recruitment and T-helper cell differentiation after inhaled allergen
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P2860
CD28 interactions with either CD80 or CD86 are sufficient to induce allergic airway inflammation in mice
description
im Oktober 1999 veröffentlichter wissenschaftlicher Artikel
@de
wetenschappelijk artikel
@nl
наукова стаття, опублікована в жовтні 1999
@uk
name
CD28 interactions with either ...... ic airway inflammation in mice
@en
CD28 interactions with either ...... ic airway inflammation in mice
@nl
type
label
CD28 interactions with either ...... ic airway inflammation in mice
@en
CD28 interactions with either ...... ic airway inflammation in mice
@nl
prefLabel
CD28 interactions with either ...... ic airway inflammation in mice
@en
CD28 interactions with either ...... ic airway inflammation in mice
@nl
P2093
P2860
P356
P1476
CD28 interactions with either ...... ic airway inflammation in mice
@en
P2093
J A Bluestone
J Weinstock
K Herrmann
P2860
P304
P356
10.1165/AJRCMB.21.4.3714
P407
P577
1999-10-01T00:00:00Z