about
Mitochondria-associated endoplasmic reticulum membrane (MAM) integrity is required for insulin signaling and is implicated in hepatic insulin resistanceEffect of transient and permanent permeability transition pore opening on NAD(P)H localization in intact cells.Exosomes participate in the alteration of muscle homeostasis during lipid-induced insulin resistance in mice.Insulin resistance is associated with MCP1-mediated macrophage accumulation in skeletal muscle in mice and humansShort-term and long-term effects of submaximal maternal exercise on offspring glucose homeostasis and pancreatic function.Preserved endothelium-dependent dilatation of the coronary microvasculature at the early phase of diabetes mellitus despite the increased oxidative stress and depressed cardiac mechanical function ex vivo.Adipocytes, like their progenitors, contribute to inflammation of adipose tissues through promotion of Th-17 cells and activation of monocytes, in obese subjects.Body adiposity dictates different mechanisms of increased coronary reactivity related to improved in vivo cardiac function.Liver mitochondria and insulin resistance.Mitochondria-associated endoplasmic reticulum membranes allow adaptation of mitochondrial metabolism to glucose availability in the liver.Liver mitochondrial function in ZDF rats during the early stages of diabetes disease.Exosome-like vesicles released from lipid-induced insulin-resistant muscles modulate gene expression and proliferation of beta recipient cells in mice.Disruption of calcium transfer from ER to mitochondria links alterations of mitochondria-associated ER membrane integrity to hepatic insulin resistance.FTO contributes to hepatic metabolism regulation through regulation of leptin action and STAT3 signalling in liver.Synergistic cellular effects including mitochondrial destabilization, autophagy and apoptosis following low-level exposure to a mixture of lipophilic persistent organic pollutants.Imeglimin prevents human endothelial cell death by inhibiting mitochondrial permeability transition without inhibiting mitochondrial respiration.Reduction of endoplasmic reticulum- mitochondria interactions in beta cells from patients with type 2 diabetes.Sodium excretion is higher in patients with rheumatoid arthritis than in matched controls.Preventing cell death with a 'check valve' in mitochondrial complex I?Imeglimin normalizes glucose tolerance and insulin sensitivity and improves mitochondrial function in liver of a high-fat, high-sucrose diet mice model.Na+/H+ exchange inhibition with cariporide prevents alterations of coronary endothelial function in streptozotocin-induced diabetes.Losartan, an angiotensin II type 1 receptor blocker, protects human islets from glucotoxicity through the phospholipase C pathway.Maternal exercise modifies body composition and energy substrates handling in male offspring fed a high-fat/high-sucrose diet.Sulforaphane improves disrupted ER-mitochondria interactions and suppresses exaggerated hepatic glucose production.AMPKα1-LDH pathway regulates muscle stem cell self-renewal by controlling metabolic homeostasis.Inhibition of complex I regulates the mitochondrial permeability transition through a phosphate-sensitive inhibitory site masked by cyclophilin D.Adipose Tissue-Derived Stem Cells From Obese Subjects Contribute to Inflammation and Reduced Insulin Response in Adipocytes Through Differential Regulation of the Th1/Th17 Balance and Monocyte Activation.Role of Mitochondria in the Mechanism(s) of Action of MetforminEffects of a high-fat diet on energy metabolism and ROS production in rat liverAcute mitochondrial actions of glitazones on the liver: a crucial parameter for their antidiabetic propertiesAssessing Mitochondrial Bioenergetics by Respirometry in Cells or Isolated OrganellesMaternal exercise before and during gestation modifies liver and muscle mitochondria in rat offspringRegulation of Mitochondria-Associated Membranes (MAMs) by NO/sGC/PKG Participates in the Control of Hepatic Insulin Response
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Guillaume Vial
@ast
Guillaume Vial
@en
Guillaume Vial
@es
Guillaume Vial
@nl
type
label
Guillaume Vial
@ast
Guillaume Vial
@en
Guillaume Vial
@es
Guillaume Vial
@nl
prefLabel
Guillaume Vial
@ast
Guillaume Vial
@en
Guillaume Vial
@es
Guillaume Vial
@nl
P106
P31
P496
0000-0002-7333-8526