Mouse anti-mouse IgD monoclonal antibodies generated in IgD-deficient mice
about
Receptor editing is the main mechanism of B cell tolerance toward membrane antigensMice reconstituted with DNA polymerase beta-deficient fetal liver cells are able to mount a T cell-dependent immune response and mutate their Ig genes normallyRas activation of Erk restores impaired tonic BCR signaling and rescues immature B cell differentiation.Different sensitivity to receptor editing of B cells from mice hemizygous or homozygous for targeted Ig transgenesNeonatally induced inactivation of the vascular cell adhesion molecule 1 gene impairs B cell localization and T cell-dependent humoral immune responseThe mouse mismatch repair protein, MSH3, is a nucleoplasmic protein that aggregates into denser nuclear bodies under conditions of stress.
P2860
Mouse anti-mouse IgD monoclonal antibodies generated in IgD-deficient mice
description
scientific article published on 01 June 1995
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована в червні 1995
@uk
name
Mouse anti-mouse IgD monoclonal antibodies generated in IgD-deficient mice
@en
Mouse anti-mouse IgD monoclonal antibodies generated in IgD-deficient mice
@nl
type
label
Mouse anti-mouse IgD monoclonal antibodies generated in IgD-deficient mice
@en
Mouse anti-mouse IgD monoclonal antibodies generated in IgD-deficient mice
@nl
prefLabel
Mouse anti-mouse IgD monoclonal antibodies generated in IgD-deficient mice
@en
Mouse anti-mouse IgD monoclonal antibodies generated in IgD-deficient mice
@nl
P1476
Mouse anti-mouse IgD monoclonal antibodies generated in IgD-deficient mice
@en
P2093
P304
P356
10.1016/0022-1759(95)00059-J
P577
1995-06-01T00:00:00Z