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Intratumor Heterogeneity and Branched Evolution Revealed by Multiregion SequencingPyk2 and FAK regulate neurite outgrowth induced by growth factors and integrinsCyclin D mediates tolerance of genome-doubling in cancers with functional p53.Tolerance of whole-genome doubling propagates chromosomal instability and accelerates cancer genome evolution.SETD2 loss-of-function promotes renal cancer branched evolution through replication stress and impaired DNA repair.Requirement for interaction of PI3-kinase p110α with RAS in lung tumor maintenance.YY1 inhibits the activation of the p53 tumor suppressor in response to genotoxic stress.Replication stress links structural and numerical cancer chromosomal instabilityCoactivator-dependent acetylation stabilizes members of the SREBP family of transcription factors.The DNA binding activities of Smad2 and Smad3 are regulated by coactivator-mediated acetylation.The balance between acetylation and deacetylation controls Smad7 stability.RAD18, WRNIP1 and ATMIN promote ATM signalling in response to replication stress.Leukotriene D4-induced signal transduction.Leukotriene D4-induced mobilization of intracellular Ca2+ in epithelial cells is critically dependent on activation of the small GTP-binding protein Rho.The regulation of leukotriene D4-induced calcium influx in human epithelial cells involves protein tyrosine phosphorylation.Induction of aggregation of Raji human B-lymphoblastic cells by vasoactive intestinal peptide.Extreme chromosomal instability forecasts improved outcome in ER-negative breast cancer: a prospective validation cohort study from the TACT trial.Deterministic Evolutionary Trajectories Influence Primary Tumor Growth: TRACERx Renal.Corrigendum: Phylogenetic ctDNA analysis depicts early-stage lung cancer evolutionErratum: RAD18, WRNIP1 and ATMIN promote ATM signalling in response to replication stressSelective inhibition of cancer cell self-renewal through a Quisinostat-histone H1.0 axisTolerance of Chromosomal Instability in Cancer: Mechanisms and Therapeutic Opportunities
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description
researcher ORCID ID = 0000-0001-8303-5409
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wetenschapper
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name
Eva Gronroos
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Eva Gronroos
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Eva Gronroos
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Eva Gronroos
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type
label
Eva Gronroos
@ast
Eva Gronroos
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Eva Gronroos
@es
Eva Gronroos
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prefLabel
Eva Gronroos
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Eva Gronroos
@en
Eva Gronroos
@es
Eva Gronroos
@nl
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0000-0001-8303-5409